The Immune Response to Skin Trauma Is Dependent on the Etiology of Injury in a Mouse Model of Burn and Excision.
暂无分享,去创建一个
M. Fear | F. Wood | J. Waithman | V. Fear | S. Valvis | Vanessa S. Fear
[1] J. Duke,et al. Developing a burn injury severity score (BISS): adding age and total body surface area burned to the injury severity score (ISS) improves mortality concordance. , 2014, Burns : journal of the International Society for Burn Injuries.
[2] B. Malissen,et al. The origins and functions of dendritic cells and macrophages in the skin , 2014, Nature Reviews Immunology.
[3] J. Duke,et al. Burn injury, gender and cancer risk: population-based cohort study using data from Scotland and Western Australia , 2014, BMJ Open.
[4] M. Norval,et al. Altered Immunity and Dendritic Cell Activity in the Periphery of Mice after Long-Term Engraftment with Bone Marrow from Ultraviolet-Irradiated Mice , 2013, The Journal of Immunology.
[5] P. D. de Almeida,et al. Burn Injury Triggered Dysfunction in Dendritic Cell Response to TLR9 Activation and Resulted in Skewed T Cell Functions , 2012, PloS one.
[6] J. Duke,et al. Burn and cancer risk: a state-wide longitudinal analysis. , 2012, Burns : journal of the International Society for Burn Injuries.
[7] D. Greenhalgh,et al. Multiplex polymerase chain reaction pathogen detection in patients with suspected septicemia after trauma, emergency, and burn surgery. , 2012, Surgery.
[8] B. Lambrecht,et al. Directed antigen targeting in vivo identifies a role for CD103+ dendritic cells in both tolerogenic and immunogenic T-cell responses , 2011, Mucosal Immunology.
[9] P. Sly,et al. Restricted Aeroallergen Access to Airway Mucosal Dendritic Cells In Vivo Limits Allergen-Specific CD4+ T Cell Proliferation during the Induction of Inhalation Tolerance , 2011, The Journal of Immunology.
[10] N. Zmora,et al. Is Prophylactic Acyclovir Treatment Warranted for Prevention of Herpes Simplex Virus Infections in Facial Burns? A Review of the Literature , 2011, Journal of burn care & research : official publication of the American Burn Association.
[11] J. Lederer,et al. Injury Induces Early Activation of T-Cell Receptor Signaling Pathways in CD4+ Regulatory T Cells , 2011, Shock.
[12] J. Lederer,et al. Regulatory T cells suppress antigen‐driven CD4 T cell reactivity following injury , 2011, Journal of leukocyte biology.
[13] Bali Pulendran,et al. Programming dendritic cells to induce TH2 and tolerogenic responses , 2010, Nature Immunology.
[14] M. Alexander-Miller,et al. Functional Divergence among CD103+ Dendritic Cell Subpopulations following Pulmonary Poxvirus Infection , 2010, Journal of Virology.
[15] J. Bernier,et al. Selective effect of burn injury on splenic CD11c+ dendritic cells and CD8α+CD4−CD11c+ dendritic cell subsets , 2010, Cellular and Molecular Life Sciences.
[16] A. Accardo-Palumbo,et al. Circulating dendritic cells following burn. , 2009, Burns : journal of the International Society for Burn Injuries.
[17] R. Zbinden,et al. Changes in bacterial isolates from burn wounds and their antibiograms: a 20-year study (1986-2005). , 2009, Burns : journal of the International Society for Burn Injuries.
[18] O. Ohara,et al. Novel Subset of CD8α+ Dendritic Cells Localized in the Marginal Zone Is Responsible for Tolerance to Cell-Associated Antigens1 , 2009, The Journal of Immunology.
[19] G. Randolph,et al. Optimization of methods to study pulmonary dendritic cell migration reveals distinct capacities of DC subsets to acquire soluble versus particulate antigen. , 2008, Journal of immunological methods.
[20] B. R. Sharma,et al. Infection in patients with severe burns: causes and prevention thereof. , 2007, Infectious disease clinics of North America.
[21] S. Sung,et al. Diverse and Potent Chemokine Production by Lung CD11bhigh Dendritic Cells in Homeostasis and in Allergic Lung Inflammation1 , 2007, The Journal of Immunology.
[22] B. Beilin,et al. Anesthesiologists at work: an increase in pro‐inflammatory and Th2 cytokine production, and alterations in proliferative immune responses , 2006, Acta anaesthesiologica Scandinavica.
[23] B. Cairns,et al. The effect of burn injury on CD8+ and CD4+ T cells in an irradiation model of homeostatic proliferation. , 2006, The Journal of trauma.
[24] H. Redmond,et al. Cell response to surgery. , 2006, Archives of surgery.
[25] S. Ju,et al. A Major Lung CD103 (αE)-β7 Integrin-Positive Epithelial Dendritic Cell Population Expressing Langerin and Tight Junction Proteins1 , 2006, The Journal of Immunology.
[26] R. Gamelli,et al. Exacerbation of intestinal permeability in rats after a two-hit injury: Burn and Enterococcus faecalis infection* , 2004, Critical care medicine.
[27] M. Akçay,et al. Changes of microbial flora and wound colonization in burned patients. , 2004, Burns : journal of the International Society for Burn Injuries.
[28] J. Lederer,et al. Injury, sepsis, and the regulation of Toll‐like receptor responses , 2004, Journal of leukocyte biology.
[29] J. Lederer,et al. Burn Injury Promotes Antigen-Driven Th2-Type Responses In Vivo 1 , 2003, The Journal of Immunology.
[30] C. Miller‐Graziano,et al. Failure of Monocytes of Trauma Patients to Convert to Immature Dendritic Cells is Related to Preferential Macrophage-Colony-Stimulating Factor-Driven Macrophage Differentiation1 , 2003, The Journal of Immunology.
[31] E. Kovacs,et al. Elevated monocyte chemoattractant protein‐1 levels following thermal injury precede monocyte recruitment to the wound site and are controlled, in part, by tumor necrosis factor‐α , 2003, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[32] D. Herndon,et al. Effects of Burn Wound Excision on Bacterial Colonization and Invasion , 2003, Plastic and reconstructive surgery.
[33] A. Mehrhof. Effects of Burn Wound Excision on Bacterial Colonization and Invasion; Juan P. Barret, M.D., Ph.D., David N. Herndon, M.D., , 2003 .
[34] A. Singer,et al. Persistent wound infection delays epidermal maturation and increases scarring in thermal burns , 2002, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[35] J. Blache,et al. Early postoperative compensatory anti‐inflammatory response syndrome is associated with septic complications after major surgical trauma in patients with cancer , 2002, The British journal of surgery.
[36] Richard L. George,et al. GENDER DIFFERENCES IN MORTALITY FOLLOWING BURN INJURY , 2002, Shock.
[37] G. Trinchieri,et al. Reversal of Tumor-induced Dendritic Cell Paralysis by CpG Immunostimulatory Oligonucleotide and Anti–Interleukin 10 Receptor Antibody , 2002, The Journal of experimental medicine.
[38] A. Wysocki. Evaluating and managing open skin wounds: colonization versus infection. , 2002, AACN clinical issues.
[39] R. Sheridan. A great constitutional disturbance. , 2001, The New England journal of medicine.
[40] B. Rollins,et al. Wound Healing in MIP-1α−/− and MCP-1−/− Mice , 2001 .
[41] G. O’Keefe,et al. An evaluation of risk factors for mortality after burn trauma and the identification of gender-dependent differences in outcomes. , 2001, Journal of the American College of Surgeons.
[42] J. Banchereau,et al. IL-6 switches the differentiation of monocytes from dendritic cells to macrophages , 2000, Nature Immunology.
[43] J. Lederer,et al. Severe injury triggers antigen-specific T-helper cell dysfunction. , 1999, Shock.
[44] J. Lederer,et al. The effects of injury on the adaptive immune response. , 1999, Shock.
[45] A. Meyer,et al. The effector component of the cytotoxic T-lymphocyte response has a biphasic pattern after burn injury. , 1998, The Journal of surgical research.
[46] J. Lederer,et al. Anti-interleukin-10 antibody restores burn-induced defects in T-cell function. , 1997, Surgery.
[47] B. Cairns,et al. Early, complete burn wound excision partially restores cytotoxic T lymphocyte function. , 1995, Surgery.
[48] D. Kwiatkowski,et al. Systemic cytokine response after major surgery , 1992, The British journal of surgery.
[49] W. Fraser,et al. Response of serum interleukin-6 in patients undergoing elective surgery of varying severity. , 1990, Clinical science.
[50] T. Geijtenbeek,et al. Burn injury suppresses human dermal dendritic cell and Langerhans cell function. , 2011, Cellular immunology.
[51] M. Fear,et al. A peptide inhibitor of c‐Jun promotes wound healing in a mouse full‐thickness burn model , 2008, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[52] S. Tschoeke,et al. Human leukocyte antigen expression in sepsis: what have we learned? , 2005, Critical care medicine.
[53] W. Fraser,et al. The serum interleukin 6 response to elective surgery. , 1989, Lymphokine research.
[54] G. Mills,et al. Interleukin-2 secretion and transmembrane signalling in burned patients. , 1988, The Journal of trauma.