Involvement of tubulin in MPP+ neurotoxicity on NGF‐differentiated PC12 cells.
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[1] Reaction of Lewy bodies with antibodies to phosphorylated and non-phosphorylated neurofilaments , 1986, Neuroscience Letters.
[2] S. Orrenius,et al. The mechanism of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine toxicity: role of intracellular calcium. , 1988, Archives of biochemistry and biophysics.
[3] M. Kirschner,et al. Dynamic instability of microtubule growth , 1984, Nature.
[4] D. Jacobowitz,et al. A primate model of parkinsonism: selective destruction of dopaminergic neurons in the pars compacta of the substantia nigra by N-methyl-4-phenyl-1,2,3,6-tetrahydropyridine. , 1983, Proceedings of the National Academy of Sciences of the United States of America.
[5] L. Greene,et al. Release, storage and uptake of catecholamines by a clonal cell line of nerve growth factor (NGF) responsive pheochromocytoma cells , 1977, Brain Research.
[6] L. Hartwell,et al. Dominant effects of tubulin overexpression in Saccharomyces cerevisiae , 1989, Molecular and cellular biology.
[7] M. Mattson. Cellular signaling mechanisms common to the development and degeneration of neuroarchitecture. A review , 1989, Mechanisms of Ageing and Development.
[8] J. Poirier,et al. The specific vulnerability of the substantia nigra to MPTP is related to the presence of transition metals. , 1985, Biochemical and biophysical research communications.
[9] B. Howard,et al. A Dopaminergic Cell Line Variant Resistant to the Neurotoxin 1‐Methyl‐4‐Phenyl‐1,2,3,6‐Tetrahydropyridine , 1987, Journal of neurochemistry.
[10] R. S. Burns,et al. Intraneuronal generation of a pyridinium metabolite may cause drug-induced parkinsonism , 1984, Nature.
[11] C. Mytilineou,et al. 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine destroys dopamine neurons in explants of rat embryo mesencephalon. , 1984, Science.
[12] U. K. Laemmli,et al. Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4 , 1970, Nature.
[13] W. Nicklas,et al. Inhibition of NADH-linked oxidation in brain mitochondria by 1-methyl-4-phenyl-pyridine, a metabolite of the neurotoxin, 1-methyl-4-phenyl-1,2,5,6-tetrahydropyridine. , 1985, Life sciences.
[14] R. Ramsay,et al. Uptake of the neurotoxin 1-methyl-4-phenylpyridine (MPP+) by mitochondria and its relation to the inhibition of the mitochondrial oxidation of NAD+-linked substrates by MPP+. , 1986, Biochemical and biophysical research communications.
[15] M. Bunge. The axonal cytoskeleton: its role in generating and maintaining cell form , 1986, Trends in Neurosciences.
[16] R. Duvoisin,et al. Dopaminergic neurotoxicity of 1-methyl-4-phenyl-1,2,5,6-tetrahydropyridine in mice. , 1984, Science.
[17] H. Towbin,et al. Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications. , 1979, Proceedings of the National Academy of Sciences of the United States of America.
[18] D. Cleveland. Autoregulated control of tubulin synthesis in animal cells. , 1989, Current opinion in cell biology.
[19] I. Grundke‐Iqbal,et al. Lewy Bodies Contain Epitopes Both Shared and Distinct from Alzheimer Neurofibrillary Tangles , 1988, Journal of neuropathology and experimental neurology.
[20] M. Camatini,et al. N-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) induces cytoskeletal alterations on ‘Swiss 3T3’ mouse fibroblasts , 1991, Neuroscience Letters.
[21] L. Greene,et al. Establishment of a noradrenergic clonal line of rat adrenal pheochromocytoma cells which respond to nerve growth factor. , 1976, Proceedings of the National Academy of Sciences of the United States of America.
[22] M. Swash,et al. Antibody to an abnormal protein in amyotrophic lateral sclerosis identifies Lewy body-like inclusions in ALS and Lewy bodies in Parkinson's disease , 1993, Neuroscience Letters.
[23] D. Schubert,et al. Storage and release of acetylcholine by a clonal cell line. , 1977, Proceedings of the National Academy of Sciences of the United States of America.
[24] S. Snyder,et al. Parkinsonism-inducing neurotoxin, N-methyl-4-phenyl-1,2,3,6 -tetrahydropyridine: uptake of the metabolite N-methyl-4-phenylpyridine by dopamine neurons explains selective toxicity. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[25] M. Zompo,et al. Evaluation of the toxicity of the dopaminergic neurotoxins MPTP and MPP+ in PC12 pheochromocytoma cells: Binding and biological studies , 1988, Neuroscience Letters.