Transforming Growth Factor Beta 2 and Heme Oxygenase 1 Genes Are Risk Factors for the Cerebral Malaria Syndrome in Angolan Children
暂无分享,去创建一个
T. Clark | A. Coutinho | O. Wagner | N. Sepúlveda | L. Gonçalves | C. Penha-Gonçalves | M. Sambo | S. Mustafa | M. J. Trovoada | Carla Benchimol | M. I. Marques | Vatúsia Quinhentos | R. Velosa | T. Clark
[1] Martin Olivier,et al. Cerebral malaria: human versus mouse studies. , 2010, Trends in parasitology.
[2] Pardis C Sabeti,et al. Positive selection of a CD36 nonsense variant in sub-Saharan Africa, but no association with severe malaria phenotypes , 2009, Human molecular genetics.
[3] Hong-Wen Deng,et al. Is Replication the Gold Standard for Validating Genome-Wide Association Findings? , 2008, PloS one.
[4] Taane G. Clark,et al. A global network for investigating the genomic epidemiology of malaria , 2008, Nature.
[5] D. Kwiatkowski,et al. Variation in the ICAM1 gene is not associated with severe malaria phenotypes , 2008, Genes and Immunity.
[6] J. Alves,et al. The causes of death of hospitalized children in Angola , 2008, Tropical doctor.
[7] J. Anrather,et al. Heme Oxygenase-1 Inhibits the Expression of Adhesion Molecules Associated with Endothelial Cell Activation via Inhibition of NF-κB RelA Phosphorylation at Serine 2761 , 2007, The Journal of Immunology.
[8] S. Issifou,et al. Differences in presentation of severe malaria in urban and rural Gabon. , 2007, The American journal of tropical medicine and hygiene.
[9] Manuel A. R. Ferreira,et al. PLINK: a tool set for whole-genome association and population-based linkage analyses. , 2007, American journal of human genetics.
[10] P. Donnelly,et al. Replicating genotype–phenotype associations , 2007, Nature.
[11] M. Mota,et al. Heme oxygenase-1 and carbon monoxide suppress the pathogenesis of experimental cerebral malaria , 2007, Nature Medicine.
[12] M. Mota,et al. Bone marrow chimeric mice reveal a dual role for CD36 in Plasmodium berghei ANKA infection , 2007, Malaria Journal.
[13] O. Doumbo,et al. X-Linked G6PD Deficiency Protects Hemizygous Males but Not Heterozygous Females against Severe Malaria , 2007, PLoS medicine.
[14] Augustin Luna,et al. snp.plotter: an R-based SNP/haplotype association and linkage disequilibrium plotting package , 2007, Bioinform..
[15] Xavier Estivill,et al. SNPassoc: an R package to perform whole genome association studies , 2007, Bioinform..
[16] R. Snow,et al. The Effect of α +-Thalassaemia on the Incidence of Malaria and Other Diseases in Children Living on the Coast of Kenya , 2006, PLoS medicine.
[17] C. Newton,et al. Pathogenesis, clinical features, and neurological outcome of cerebral malaria , 2005, The Lancet Neurology.
[18] R. Snow,et al. Heritability of Malaria in Africa , 2005, PLoS medicine.
[19] S. Shibahara,et al. Microsatellite polymorphism in the heme oxygenase-1 gene promoter is associated with susceptibility to cerebral malaria in Myanmar. , 2005, Japanese journal of infectious diseases.
[20] R. Sinden,et al. Upregulation of TGF-beta, FOXP3, and CD4+CD25+ regulatory T cells correlates with more rapid parasite growth in human malaria infection. , 2005, Immunity.
[21] D. Kwiatkowski. How malaria has affected the human genome and what human genetics can teach us about malaria. , 2005, American journal of human genetics.
[22] R. Snow,et al. Sickle cell trait and the risk of Plasmodium falciparum malaria and other childhood diseases. , 2005, The Journal of infectious diseases.
[23] E. Dietz,et al. Short Communication: Limited influence of haptoglobin genotypes on severe malaria in Ghanaian children , 2005, Tropical medicine & international health : TM & IH.
[24] K. Maitland,et al. Both heterozygous and homozygous alpha+ thalassemias protect against severe and fatal Plasmodium falciparum malaria on the coast of Kenya. , 2005, Blood.
[25] C. Rogier,et al. Genetic study of ICAM1 in clinical malaria in Senegal. , 2005, Tissue antigens.
[26] Sammy Wambua,et al. An Immune Basis for Malaria Protection by the Sickle Cell Trait , 2005, PLoS medicine.
[27] K. Marsh,et al. Clinical features and pathogenesis of severe malaria. , 2004, Trends in parasitology.
[28] V. Adabayeri,et al. Allelic polymorphisms in the repeat and promoter regions of the interleukin‐4 gene and malaria severity in Ghanaian children , 2004, Clinical and experimental immunology.
[29] E. Dietz,et al. α+-thalassemia protects African children from severe malaria , 2004 .
[30] D. Kwiatkowski,et al. Interferon-alpha receptor-1 (IFNAR1) variants are associated with protection against cerebral malaria in The Gambia , 2003, Genes and Immunity.
[31] K. Tokunaga,et al. CD36 polymorphism is associated with protection from cerebral malaria. , 2003, American journal of human genetics.
[32] D. Granger,et al. Regulation of Endothelial Cell Adhesion Molecule Expression in an Experimental Model of Cerebral Malaria , 2002, Microcirculation.
[33] Pak Chung Sham,et al. GENECOUNTING: haplotype analysis with missing genotypes , 2002, Bioinform..
[34] D. Holmberg,et al. Identification of two cerebral malaria resistance loci using an inbred wild-derived mouse strain , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[35] D. Kwiatkowski,et al. IFNGR1 gene promoter polymorphisms and susceptibility to cerebral malaria. , 2002, The Journal of infectious diseases.
[36] E. Riley,et al. Cerebral malaria: the contribution of studies in animal models to our understanding of immunopathogenesis. , 2002, Microbes and infection.
[37] D. Schaid,et al. Score tests for association between traits and haplotypes when linkage phase is ambiguous. , 2002, American journal of human genetics.
[38] I. Naka,et al. Absence of association between the allele coding methionine at position 29 in the N-terminal domain of ICAM-1 (ICAM-1(Kilifi)) and severe malaria in the northwest of Thailand. , 2001, Japanese journal of infectious diseases.
[39] A. Pain,et al. A non-sense mutation in Cd36 gene is associated with protection from severe malaria , 2001, The Lancet.
[40] P. Kremsner,et al. Differential Cellular Accumulation of Transforming Growth Factor-β1, -β2, and -β3 in Brains of Patients Who Died with Cerebral Malaria , 2000 .
[41] M. Alpers,et al. Association of the ICAM-1Kilifi mutation with protection against severe malaria in Lambaréné, Gabon. , 1999, The American journal of tropical medicine and hygiene.
[42] D. Clayton,et al. A generalization of the transmission/disequilibrium test for uncertain-haplotype transmission. , 1999, American journal of human genetics.
[43] L. Abel,et al. Malaria in humans: Plasmodium falciparum blood infection levels are linked to chromosome 5q31-q33. , 1998, American journal of human genetics.
[44] Charles R. Newton,et al. Severe falciparum malaria in children: current understanding of pathophysiology and supportive treatment. , 1998, Pharmacology & therapeutics.
[45] A. Hill,et al. Glucose-6-phosphate dehydrogenase deficiency and malaria , 1998, Journal of Molecular Medicine.
[46] D. Kwiatkowski,et al. Absence of an association between intercellular adhesion molecule 1, complement receptor 1 and interleukin 1 receptor antagonist gene polymorphisms and severe malaria in a West African population. , 1998, Transactions of the Royal Society of Tropical Medicine and Hygiene.
[47] R. Snow,et al. A high frequency African coding polymorphism in the N-terminal domain of ICAM-1 predisposing to cerebral malaria in Kenya. , 1997, Human molecular genetics.
[48] B. M. Greenwood,et al. Natural selection of hemi- and heterozygotes for G6PD deficiency in Africa by resistance to severe malaria , 1995, Nature.
[49] M. Soares,et al. Heme Oxygenase-1 Inhibits the Expression of Adhesion Molecules Associated with Endothelial Cell Activation via Inhibition of NF- B RelA Phosphorylation at Serine 276 , 2007 .
[50] L. Rénia,et al. Experimental models of cerebral malaria. , 2005, Current topics in microbiology and immunology.
[51] E. Dietz,et al. Alpha(+)-thalassemia protects African children from severe malaria. , 2004, Blood.
[52] D. Kwiatkowski,et al. Nucleotide and haplotypic diversity of the NOS2A promoter region and its relationship to cerebral malaria , 2004, Human Genetics.
[53] K. Tokunaga,et al. Polymorphisms of CD36 in Thai malaria patients. , 2002, The Southeast Asian journal of tropical medicine and public health.
[54] P. Kremsner,et al. Differential cellular accumulation of transforming growth factor-beta1, -beta2, and -beta3 in brains of patients who died with cerebral malaria. , 2000, The Journal of infectious diseases.
[55] E. Riley,et al. Maintaining the immunological balance in parasitic infections: a role for TGF-beta? , 2000, Parasitology today.
[56] E. Riley,et al. Transforming Growth Factor (cid:98) Production Is Inversely Correlated with Severity of Murine Malaria Infection , 1998 .
[57] G. Snounou. Detection and identification of the four malaria parasite species infecting humans by PCR amplification. , 1996, Methods in molecular biology.
[58] B. Greenwood,et al. Comparison of two simple methods for determining malaria parasite density. , 1991, Transactions of the Royal Society of Tropical Medicine and Hygiene.
[59] Peter Donnelly,et al. Genome-wide and fine-resolution association analysis of malaria in West Africa , 2009, Nature Genetics.