Discovery of somatic STAT5b mutations in large granular lymphocytic leukemia.
暂无分享,去创建一个
S. Mustjoki | O. Kallioniemi | K. Wennerberg | J. Maciejewski | P. Ellonen | K. Porkka | S. Eldfors | A. Jerez | C. Heckman | E. Andersson | T. Loughran | H. Rajala | T. Olson | M. Clemente | H. Kuusanmäki | S. Lagström | A. V. van Adrichem | Yiyi Yan | Dan Zhang | A. Awwad
[1] Keunsoo Kang,et al. Comprehensive meta-analysis of Signal Transducers and Activators of Transcription (STAT) genomic binding patterns discerns cell-specific cis-regulatory modules , 2012, BMC Genomics.
[2] Laura A. Sullivan,et al. Aberrant overexpression of IL-15 initiates large granular lymphocyte leukemia through chromosomal instability and DNA hypermethylation. , 2012, Cancer cell.
[3] S. Mustjoki,et al. STAT3 mutations unify the pathogenesis of chronic lymphoproliferative disorders of NK cells and T-cell large granular lymphocyte leukemia. , 2012, Blood.
[4] T. Loughran,et al. Large granular lymphocyte leukemia: from dysregulated pathways to therapeutic targets. , 2012, Future oncology.
[5] Kari Christine Nadeau,et al. The STAT5b Pathway Defect and Autoimmunity , 2012, Front. Immun..
[6] S. Mustjoki,et al. Somatic STAT3 mutations in large granular lymphocytic leukemia. , 2012, The New England journal of medicine.
[7] Mikko Koski,et al. Chipster: user-friendly analysis software for microarray and other high-throughput data , 2011, BMC Genomics.
[8] B. Ebert,et al. The STAT5 inhibitor pimozide decreases survival of chronic myelogenous leukemia cells resistant to kinase inhibitors. , 2011, Blood.
[9] S. Mustjoki,et al. Mono/oligoclonal T and NK cells are common in chronic myeloid leukemia patients at diagnosis and expand during dasatinib therapy. , 2010, Blood.
[10] Liisa Holm,et al. Advances and pitfalls of protein structural alignment. , 2009, Current opinion in structural biology.
[11] S. Mustjoki,et al. Clonal expansion of T/NK-cells during tyrosine kinase inhibitor dasatinib therapy , 2008, Leukemia.
[12] Torsten Schwede,et al. The SWISS-MODEL Repository and associated resources , 2008, Nucleic Acids Res..
[13] R. Albert,et al. Network model of survival signaling in large granular lymphocyte leukemia , 2008, Proceedings of the National Academy of Sciences.
[14] Gordon K. Smyth,et al. A comparison of background correction methods for two-colour microarrays , 2007, Bioinform..
[15] B. Little,et al. Growth Hormone Insensitivity and Severe Short Stature in Siblings: A Novel Mutation at the Exon 13-Intron 13 Junction of the STAT5b Gene , 2007, Hormone Research in Paediatrics.
[16] E. Burks,et al. Pathogenesis of neutropenia in large granular lymphocyte leukemia and Felty syndrome. , 2006, Blood reviews.
[17] L. Hennighausen,et al. Stat5a/b are essential for normal lymphoid development and differentiation. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[18] Mario Cazzola,et al. A gain-of-function mutation of JAK2 in myeloproliferative disorders. , 2005, The New England journal of medicine.
[19] Gordon K Smyth,et al. Statistical Applications in Genetics and Molecular Biology Linear Models and Empirical Bayes Methods for Assessing Differential Expression in Microarray Experiments , 2011 .
[20] Terry Speed,et al. Normalization of cDNA microarray data. , 2003, Methods.
[21] M. D'urso,et al. The structure of human STAT5A and B genes reveals two regions of nearly identical sequence and an alternative tissue specific STAT5B promoter. , 2002, Gene.
[22] J. Turkson,et al. Inhibition of STAT3 signaling leads to apoptosis of leukemic large granular lymphocytes and decreased Mcl-1 expression. , 2001, The Journal of clinical investigation.
[23] A. Miyajima,et al. Constitutive Activation of STAT5 by a Point Mutation in the SH2 Domain* , 2000, The Journal of Biological Chemistry.
[24] S. Asano,et al. Constitutively active STAT5A and STAT5B in vitro and in vivo: mutation of STAT5 is not a frequent cause of leukemogenesis. , 2000, International journal of hematology.
[25] Bernd Groner,et al. TRANSCRIPTION FACTOR STAT3B/DNA COMPLEX , 1999 .
[26] T. Loughran,et al. Large granular lymphocyte leukemia , 2007, Current hematologic malignancy reports.
[27] W. Leonard,et al. Stat5b Is Essential for Natural Killer Cell–mediated Proliferation and Cytolytic Activity , 1998, The Journal of experimental medicine.
[28] S. Becker,et al. Three-dimensional structure of the Stat3β homodimer bound to DNA , 1998, Nature.
[29] W. Leonard,et al. Jaks and STATs: biological implications. , 1998, Annual review of immunology.
[30] R. Snell,et al. Requirement of STAT5b for sexual dimorphism of body growth rates and liver gene expression. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[31] L. Hennighausen,et al. Stat5a is mandatory for adult mammary gland development and lactogenesis. , 1997, Genes & development.
[32] J. Griffin,et al. Tyrosyl phosphorylation and DNA binding activity of signal transducers and activators of transcription (STAT) proteins in hematopoietic cell lines transformed by Bcr/Abl , 1996, The Journal of experimental medicine.
[33] A. Miyajima,et al. Interleukin‐3, granulocyte‐macrophage colony‐stimulating factor, and interleukin‐5 transduce signals through two forms of STAT5 , 1995, Journal of leukocyte biology.
[34] A. Miyajima,et al. Interleukin‐3, granulocyte‐macrophage colony stimulating factor and interleukin‐5 transduce signals through two STAT5 homologs. , 1995, The EMBO journal.
[35] B. Groner,et al. Prolactin induces phosphorylation of Tyr694 of Stat5 (MGF), a prerequisite for DNA binding and induction of transcription. , 1994, The EMBO journal.
[36] T. Loughran,et al. Clonal diseases of large granular lymphocytes. , 1993, Blood.
[37] C. Disteche,et al. Leukemia of large granular lymphocytes: association with clonal chromosomal abnormalities and autoimmune neutropenia, thrombocytopenia, and hemolytic anemia. , 1985, Annals of internal medicine.