The ferredoxin reductase gene is regulated by the p53 family and sensitizes cells to oxidative stress-induced apoptosis

[1]  A. Yang,et al.  On the shoulders of giants: p63, p73 and the rise of p53. , 2002, Trends in genetics : TIG.

[2]  C. Rongo.  Disparate cell types use a shared complex of PDZ proteins for polarized protein localization. , 2001, Cytokine & growth factor reviews.

[3]  Michael Dohn,et al.  Receptor tyrosine kinase EphA2 is regulated by p53-family proteins and induces apoptosis , 2001, Oncogene.

[4]  K. Kinzler,et al.  Ferredoxin reductase affects p53-dependent, 5-fluorouracil–induced apoptosis in colorectal cancer cells , 2001, Nature Medicine.

[5]  Xinbin Chen,et al.  p63α and ΔNp63α can induce cell cycle arrest and apoptosis and differentially regulate p53 target genes , 2001, Oncogene.

[6]  A. Wolffe,et al.  Chromatin remodeling and transcriptional activation: the cast (in order of appearance) , 2001, Oncogene.

[7]  J. Pietenpol,et al.  Kinetics of p53 Binding to Promoter Sites In Vivo , 2001, Molecular and Cellular Biology.

[8]  K. Vousden,et al.  PUMA, a novel proapoptotic gene, is induced by p53. , 2001, Molecular cell.

[9]  K. Kinzler,et al.  PUMA induces the rapid apoptosis of colorectal cancer cells. , 2001, Molecular cell.

[10]  Gleb Bourenkov,et al.  Adrenodoxin Reductase-Adrenodoxin Complex Structure Suggests Electron Transfer Path in Steroid Biosynthesis* , 2001, The Journal of Biological Chemistry.

[11]  Xinbin Chen,et al.  Definition of the p53 Functional Domains Necessary for Inducing Apoptosis* , 2000, The Journal of Biological Chemistry.

[12]  A. Yang,et al.  p63 and p73: p53 mimics, menaces and more , 2000, Nature Reviews Molecular Cell Biology.

[13]  A. Levine,et al.  Surfing the p53 network , 2000, Nature.

[14]  S. Orrenius,et al.  Triggering and modulation of apoptosis by oxidative stress. , 2000, Free radical biology & medicine.

[15]  Xinbin Chen,et al.  MCG10, a Novel p53 Target Gene That Encodes a KH Domain RNA-Binding Protein, Is Capable of Inducing Apoptosis and Cell Cycle Arrest in G2-M , 2000, Molecular and Cellular Biology.

[16]  R. G. Allen,et al.  Oxidative stress and gene regulation. , 2000, Free radical biology & medicine.

[17]  R. Evans,et al.  Regulation of Hormone-Induced Histone Hyperacetylation and Gene Activation via Acetylation of an Acetylase , 1999, Cell.

[18]  X. Chen,et al.  The p53 family: same response, different signals? , 1999, Molecular medicine today.

[19]  W. Kaelin.  The emerging p53 gene family. , 1999, Journal of the National Cancer Institute.

[20]  C. Harris,et al.  GADD45 induction of a G2/M cell cycle checkpoint. , 1999, Proceedings of the National Academy of Sciences of the United States of America.

[21]  Jieyuan Jiang,et al.  Differential regulation of cellular target genes by p53 devoid of the PXXP motifs with impaired apoptotic activity , 1999, Oncogene.

[22]  K. Kinzler,et al.  Requirement for p53 and p21 to sustain G2 arrest after DNA damage. , 1998, Science.

[23]  A. Yang,et al.  p63, a p53 homolog at 3q27-29, encodes multiple products with transactivating, death-inducing, and dominant-negative activities. , 1998, Molecular cell.

[24]  Xinbin Chen,et al.  Identification of a Novel p53 Functional Domain That Is Necessary for Mediating Apoptosis* , 1998, The Journal of Biological Chemistry.

[25]  K. Kinzler,et al.  14-3-3σ Is a p53-Regulated Inhibitor of G2/M Progression , 1997 .

[26]  X. Chen,et al.  p53 levels, functional domains, and DNA damage determine the extent of the apoptotic response of tumor cells. , 1996, Genes & development.

[27]  C. Prives,et al.  p53: puzzle and paradigm. , 1996, Genes & development.

[28]  K. Kinzler,et al.  p21 is necessary for the p53-mediated G1 arrest in human cancer cells. , 1995, Cancer research.

[29]  John Calvin Reed,et al.  Tumor suppressor p53 is a direct transcriptional activator of the human bax gene , 1995, Cell.

[30]  M. Kastan,et al.  DNA strand breaks: the DNA template alterations that trigger p53-dependent DNA damage response pathways , 1994, Molecular and cellular biology.

[31]  David Beach,et al.  p21 is a universal inhibitor of cyclin kinases , 1993, Nature.

[32]  J. Trent,et al.  WAF1, a potential mediator of p53 tumor suppression , 1993, Cell.

[33]  S. Elledge,et al.  The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases , 1993, Cell.

[34]  J. Doroshow,et al.  DNA base modifications induced in isolated human chromatin by NADH dehydrogenase-catalyzed reduction of doxorubicin. , 1992, Biochemistry.

[35]  B. Vogelstein,et al.  p53 mutations in human cancers. , 1991, Science.

[36]  R. Schlegel,et al.  The E6 and E7 genes of the human papillomavirus type 16 together are necessary and sufficient for transformation of primary human keratinocytes , 1989, Journal of virology.

[37]  Israel Hanukoglu,et al.  cDNA sequence of adrenodoxin reductase. Identification of NADP-binding sites in oxidoreductases. , 1989, European journal of biochemistry.

[38]  M. Waterman,et al.  Cell-free synthesis of precursor forms of mitochondrial steroid hydroxylase enzymes of the bovine adrenal cortex. , 1982, Archives of biochemistry and biophysics.