c-Jun is essential for normal mouse development and hepatogenesis

[1]  E. Wagner,et al.  Osteoblasts are target cells for transformation in c-fos transgenic mice , 1993, The Journal of cell biology.

[2]  D. Cohen,et al.  Transcriptional regulation in the testis: a role for transcription factor AP-1 complexes at various stages of spermatogenesis. , 1993, Oncogene.

[3]  E. Wagner,et al.  Bone and haematopoietic defects in mice lacking c-fos , 1992, Nature.

[4]  E. Wagner,et al.  Embryonic stem (ES) cells lacking functional c-jun: consequences for growth and differentiation, AP-1 activity and tumorigenicity. , 1992, Oncogene.

[5]  B. Spiegelman,et al.  Pleiotropic effects of a null mutation in the c-fos proto-oncogene , 1992, Cell.

[6]  R. Weinberg,et al.  Effects of an Rb mutation in the mouse , 1992, Nature.

[7]  A. Berns,et al.  Requirement for a functional Rb-1 gene in murine development , 1992, Nature.

[8]  A. Bradley,et al.  Mice deficient for Rb are nonviable and show defects in neurogenesis and haematopoiesis , 1992, Nature.

[9]  S. Tapscott,et al.  Functional antagonism between c-Jun and MyoD proteins: A direct physical association , 1992, Cell.

[10]  M. Karin,et al.  The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformation. , 1991, Biochimica et biophysica acta.

[11]  M. Yaniv,et al.  Overexpression of c-jun, junB, or junD affects cell growth differently. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[12]  U. Möhle-Steinlein,et al.  A novel target cell for c‐fos‐induced oncogenesis: development of chondrogenic tumours in embryonic stem cell chimeras. , 1991, The EMBO journal.

[13]  R. Bravo,et al.  The jun and fos protein families are both required for cell cycle progression in fibroblasts , 1991, Molecular and cellular biology.

[14]  D. Soprano,et al.  A potential role for c-jun in cell cycle progression through late G1 and S. , 1991, Oncogene.

[15]  F. Kruyt,et al.  Ectopic expression of c‐jun leads to differentiation of P19 embryonal carcinoma cells. , 1990, The EMBO journal.

[16]  K. Zatloukal,et al.  High molecular weight component of Mallory bodies detected by a monoclonal antibody. , 1990, Laboratory investigation; a journal of technical methods and pathology.

[17]  D. Wilkinson,et al.  Tissue-specific expression of c-jun and junB during organogenesis in the mouse. , 1989, Development.

[18]  M. Yaniv,et al.  Characterization of junD: a new member of the jun proto‐oncogene family. , 1989, The EMBO journal.

[19]  P. Vogt,et al.  Avian sarcoma virus 17 carries the jun oncogene. , 1987, Proceedings of the National Academy of Sciences of the United States of America.

[20]  M. M. Bradford A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. , 1976, Analytical biochemistry.

[21]  M. Capecchi,et al.  Mice homozygous for a targeted disruption of the proto-oncogene int-2 have developmental defects in the tail and inner ear. , 1993, Development.

[22]  E. Robertson Teratocarcinomas and embryonic stem cells : a practical approach , 1987 .