The chandelier neuron in schizophrenia
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[1] J. Gold. Cognitive deficits as treatment targets in schizophrenia , 2004, Schizophrenia Research.
[2] M. Rasband,et al. βIV spectrin is recruited to axon initial segments and nodes of Ranvier by ankyrinG , 2007, The Journal of cell biology.
[3] T. Woo,et al. Density of glutamic acid decarboxylase 67 messenger RNA-containing neurons that express the N-methyl-D-aspartate receptor subunit NR2A in the anterior cingulate cortex in schizophrenia and bipolar disorder. , 2004, Archives of general psychiatry.
[4] D. Barch,et al. Working memory and prefrontal cortex dysfunction: specificity to schizophrenia compared with major depression , 2003, Biological Psychiatry.
[5] T. Yagi,et al. Cleft palate and decreased brain gamma-aminobutyric acid in mice lacking the 67-kDa isoform of glutamic acid decarboxylase. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[6] T. Woo,et al. A subclass of prefrontal gamma-aminobutyric acid axon terminals are selectively altered in schizophrenia. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[7] D. Melchitzky,et al. Synaptic targets of calretinin-containing axon terminals in macaque monkey prefrontal cortex , 2005, Neuroscience.
[8] Jonathan D. Cohen,et al. Specificity of prefrontal dysfunction and context processing deficits to schizophrenia in never-medicated patients with first-episode psychosis. , 2005, American Journal of Psychiatry.
[9] J. Morrison,et al. Ultrastructural analysis of somatostatin‐immunoreactive neurons and synapses in the temporal and occipital cortex of the macaque monkey , 1989, The Journal of comparative neurology.
[10] L. Iversen,et al. GLUTAMIC-ACID DECARBOXYLASE IN SCHIZOPHRENIA , 1978, The Lancet.
[11] D. Lewis,et al. Schizophrenia from a neural circuitry perspective: advancing toward rational pharmacological therapies. , 2009, The Journal of clinical investigation.
[12] M. Webster,et al. Prefrontal GABA(A) receptor alpha-subunit expression in normal postnatal human development and schizophrenia. , 2010, Journal of psychiatric research.
[13] F. Benes,et al. Up-regulation of GABAA receptor binding on neurons of the prefrontal cortex in schizophrenic subjects , 1996, Neuroscience.
[14] S. Lévi,et al. Gephyrin Is Critical for Glycine Receptor Clustering But Not for the Formation of Functional GABAergic Synapses in Hippocampal Neurons , 2004, The Journal of Neuroscience.
[15] P. Haydon,et al. Gephyrin Regulates the Cell Surface Dynamics of Synaptic GABAA Receptors , 2005, The Journal of Neuroscience.
[16] T. Hashimoto,et al. Molecular mechanisms contributing to dendritic spine alterations in the prefrontal cortex of subjects with schizophrenia , 2006, Molecular Psychiatry.
[17] P. Goldman-Rakic,et al. The synaptology of parvalbumin‐immunoreactive neurons in the primate prefrontal cortex , 1992, The Journal of comparative neurology.
[18] M. Webster,et al. Decreased glutamic acid decarboxylase(67) mRNA expression in multiple brain areas of patients with schizophrenia and mood disorders. , 2009, Journal of psychiatric research.
[19] David A Lewis,et al. Reduced Pyramidal Cell Somal Volume in Auditory Association Cortex of Subjects with Schizophrenia , 2003, Neuropsychopharmacology.
[20] Csaba Varga,et al. Complex Events Initiated by Individual Spikes in the Human Cerebral Cortex , 2008, PLoS biology.
[21] M. L. Pucak,et al. Peripubertal refinement of the intrinsic and associational circuitry in monkey prefrontal cortex , 1997, Neuroscience.
[22] A. Agmon,et al. Distinct Subtypes of Somatostatin-Containing Neocortical Interneurons Revealed in Transgenic Mice , 2006, The Journal of Neuroscience.
[23] David A Lewis,et al. Conserved regional patterns of GABA-related transcript expression in the neocortex of subjects with schizophrenia. , 2008, The American journal of psychiatry.
[24] A. Sampson,et al. Reciprocal alterations in pre- and postsynaptic inhibitory markers at chandelier cell inputs to pyramidal neurons in schizophrenia. , 2002, Cerebral cortex.
[25] Pat Levitt,et al. Molecular Characterization of Schizophrenia Viewed by Microarray Analysis of Gene Expression in Prefrontal Cortex , 2000, Neuron.
[26] P. Goldman-Rakic,et al. Neuronal and glial somal size in the prefrontal cortex: a postmortem morphometric study of schizophrenia and Huntington disease. , 1998, Archives of general psychiatry.
[27] A. Marty,et al. Developmental Changes in Parvalbumin Regulate Presynaptic Ca2+ Signaling , 2005, The Journal of Neuroscience.
[28] Tyrone D. Cannon,et al. Dorsolateral prefrontal cortex activity during maintenance and manipulation of information in working memory in patients with schizophrenia. , 2005, Archives of general psychiatry.
[29] M. Rasband,et al. AnkyrinG is required for maintenance of the axon initial segment and neuronal polarity , 2008, The Journal of cell biology.
[30] R. Schneggenburger,et al. Parvalbumin Is a Mobile Presynaptic Ca2+ Buffer in the Calyx of Held that Accelerates the Decay of Ca2+ and Short-Term Facilitation , 2007, The Journal of Neuroscience.
[31] A. Sampson,et al. GABA transporter-1 mRNA in the prefrontal cortex in schizophrenia: decreased expression in a subset of neurons. , 2001, The American journal of psychiatry.
[32] M. Rasband,et al. Postnatal development of synaptic structure proteins in pyramidal neuron axon initial segments in monkey prefrontal cortex , 2009, The Journal of comparative neurology.
[33] Sarah E. Forster,et al. Subunit-selective modulation of GABA type A receptor neurotransmission and cognition in schizophrenia. , 2008, The American journal of psychiatry.
[34] David A. Lewis,et al. Altered Cortical CDC42 Signaling Pathways in Schizophrenia: Implications for Dendritic Spine Deficits , 2010, Biological Psychiatry.
[35] A. Sampson,et al. Relationship of Brain-Derived Neurotrophic Factor and Its Receptor TrkB to Altered Inhibitory Prefrontal Circuitry in Schizophrenia , 2005, The Journal of Neuroscience.
[36] T. C. Wang,et al. Postnatal changes in enzyme activities of rat myocardial adenine nucleotide catabolic pathway. , 1987, Life sciences.
[37] S. Eggan,et al. Postnatal development of pre‐ and postsynaptic GABA markers at chandelier cell connections with pyramidal neurons in monkey prefrontal cortex , 2003, The Journal of comparative neurology.
[38] David A Lewis,et al. Catching Up on Schizophrenia Natural History and Neurobiology , 2000, Neuron.
[39] A. Sampson,et al. Decreased somal size of deep layer 3 pyramidal neurons in the prefrontal cortex of subjects with schizophrenia. , 2001, Archives of general psychiatry.
[40] J. Paysan,et al. Switch in the expression of rat GABAA-receptor subtypes during postnatal development: an immunohistochemical study , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[41] R. Nicoll,et al. GABA Generates Excitement , 2003, Neuron.
[42] N L Harrison,et al. Activation and deactivation rates of recombinant GABA(A) receptor channels are dependent on alpha-subunit isoform. , 1997, Biophysical journal.
[43] A. Sampson,et al. Decreased glutamic acid decarboxylase67 messenger RNA expression in a subset of prefrontal cortical gamma-aminobutyric acid neurons in subjects with schizophrenia. , 2000, Archives of general psychiatry.
[44] Takanori Hashimoto,et al. Protracted Developmental Trajectories of GABA A Receptor α1 and α2 Subunit Expression in Primate Prefrontal Cortex , 2009, Biological Psychiatry.
[45] S. Hirsch,et al. Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia , 1998, Journal of neurology, neurosurgery, and psychiatry.
[46] P. McKenna,et al. Measurement of GABAergic parameters in the prefrontal cortex in schizophrenia: focus on GABA content, GABAA receptor α-1 subunit messenger RNA and human GABA transporter-1 (hGAT-1) messenger RNA expression , 1999, Neuroscience.
[47] A. Sampson,et al. Gene Expression Deficits in a Subclass of GABA Neurons in the Prefrontal Cortex of Subjects with Schizophrenia , 2003, The Journal of Neuroscience.
[48] D. Lewis,et al. Decreased dendritic spine density on prefrontal cortical pyramidal neurons in schizophrenia. , 2000, Archives of general psychiatry.
[49] S. Akbarian. MOLECULAR DETERMINANTS OF DYSREGULATED GABAERGIC GENE EXPRESSION IN THE PREFRONTAL CORTEX OF SUBJECTS WITH SCHIZOPHRENIA , 2010, Schizophrenia Research.
[50] P S Goldman-Rakic,et al. Synaptogenesis in the prefrontal cortex of rhesus monkeys. , 1994, Cerebral cortex.
[51] Carter Wendelken,et al. Neurocognitive development of the ability to manipulate information in working memory. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[52] T. Goldberg,et al. Cognitive impairment in schizophrenia is the core of the disorder. , 2000, Critical reviews in neurobiology.
[53] J. Deakin,et al. Reduced GABA uptake sites in the temporal lobe in schizophrenia , 1989, Neuroscience Letters.
[54] Graham V. Williams,et al. Reversal of Ketamine-Induced Working Memory Impairments by the GABAAα2/3 Agonist TPA023 , 2010, Biological Psychiatry.
[55] E. Ginns,et al. Molecular Determinants of Dysregulated GABAergic Gene Expression in the Prefrontal Cortex of Subjects with Schizophrenia , 2009, Biological Psychiatry.
[56] J. Lund,et al. Local circuit neurons of developing and mature macaque prefrontal cortex: Golgi and immunocytochemical characteristics , 1993, The Journal of comparative neurology.
[57] H. M. Morris,et al. Alterations in GABA-related transcriptome in the dorsolateral prefrontal cortex of subjects with schizophrenia , 2008, Molecular Psychiatry.
[58] V. Bennett,et al. AnkyrinG. A new ankyrin gene with neural-specific isoforms localized at the axonal initial segment and node of Ranvier. , 1995, The Journal of biological chemistry.
[59] J. DeFelipe. Types of neurons, synaptic connections and chemical characteristics of cells immunoreactive for calbindin-D28K, parvalbumin and calretinin in the neocortex , 1997, Journal of Chemical Neuroanatomy.
[60] P. Somogyi,et al. Differential synaptic localization of two major gamma-aminobutyric acid type A receptor alpha subunits on hippocampal pyramidal cells. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[61] Peter Somogyi,et al. Interneurons hyperpolarize pyramidal cells along their entire somatodendritic axis , 2009, Nature Neuroscience.
[62] A. Sampson,et al. Pyramidal cell size reduction in schizophrenia: evidence for involvement of auditory feedforward circuits , 2004, Biological Psychiatry.
[63] Yogesh K. Dwivedi,et al. Decrease in reelin and glutamic acid decarboxylase67 (GAD67) expression in schizophrenia and bipolar disorder: a postmortem brain study. , 2000, Archives of general psychiatry.
[64] Alan C. Evans,et al. Brain development during childhood and adolescence: a longitudinal MRI study , 1999, Nature Neuroscience.
[65] Vann Bennett,et al. AnkyrinG Is Required for Clustering of Voltage-gated Na Channels at Axon Initial Segments and for Normal Action Potential Firing , 1998, The Journal of cell biology.
[66] D. Lewis,et al. Parvalbumin‐immunoreactive axon terminals in macaque monkey and human prefrontal cortex: Laminar, regional, and target specificity of type I and type II synapses , 1999, The Journal of comparative neurology.
[67] J. S. Lund,et al. Synchronous development of pyramidal neuron dendritic spines and parvalbumin-immunoreactive chandelier neuron axon terminals in layer III of monkey prefrontal cortex , 1995, Neuroscience.
[68] C. Tanaka,et al. [3H]muscimol binding sites increased in autopsied brains of chronic schizophrenics. , 1987, Life sciences.
[69] David A Lewis,et al. Lamina-specific reductions in dendritic spine density in the prefrontal cortex of subjects with schizophrenia. , 2005, The American journal of psychiatry.
[70] Françoise Condé,et al. Local circuit neurons immunoreactive for calretinin, calbindin D‐28k or parvalbumin in monkey prefronatal cortex: Distribution and morphology , 1994, The Journal of comparative neurology.
[71] H. Lester,et al. Steady states, charge movements, and rates for a cloned GABA transporter expressed in Xenopus oocytes , 1993, Neuron.
[72] A. Diamond. Normal development of prefrontal cortex from birth to young adulthood: Cognitive functions, anatomy, and biochemistry. , 2002 .
[73] David A. Lewis,et al. Selective Alterations in Postsynaptic Markers of Chandelier Cell Inputs to Cortical Pyramidal Neurons in Subjects with Schizophrenia , 2009, Neuropsychopharmacology.
[74] G. Tamás,et al. Excitatory Effect of GABAergic Axo-Axonic Cells in Cortical Microcircuits , 2006, Science.
[75] Jared X. Van Snellenberg,et al. Functional neuroimaging of working memory in schizophrenia: task performance as a moderating variable. , 2006, Neuropsychology.
[76] M. Vreugdenhil,et al. Parvalbumin-deficiency facilitates repetitive IPSCs and gamma oscillations in the hippocampus. , 2003, Journal of neurophysiology.
[77] Michael B. Mayhew,et al. Allelic variation in GAD1 (GAD67) is associated with schizophrenia and influences cortical function and gene expression , 2007, Molecular Psychiatry.
[78] M. Chee,et al. fMRI study of maintenance and manipulation processes within working memory in first-episode schizophrenia. , 2005, The American journal of psychiatry.
[79] Richie Poulton,et al. Static and dynamic cognitive deficits in childhood preceding adult schizophrenia: a 30-year study. , 2010, The American journal of psychiatry.
[80] D. Lewis,et al. Cluster analysis-based physiological classification and morphological properties of inhibitory neurons in layers 2-3 of monkey dorsolateral prefrontal cortex. , 2005, Journal of neurophysiology.
[81] Paul Leonard Gabbott,et al. Local circuit neurons in the medial prefrontal cortex (areas 24a,b,c, 25 and 32) in the monkey: II. Quantitative areal and laminar distributions , 1996, The Journal of comparative neurology.
[82] T. Yagi,et al. Mice lacking the 65 kDa isoform of glutamic acid decarboxylase (GAD65) maintain normal levels of GAD67 and GABA in their brains but are susceptible to seizures. , 1996, Biochemical and biophysical research communications.
[83] T. Woo,et al. Schizophrenia and the parvalbumin-containing class of cortical local circuit neurons. , 1997, The American journal of psychiatry.
[84] R. Yuste,et al. Depolarizing effect of neocortical chandelier neurons , 2022 .
[85] Daniel R Weinberger,et al. Microarray analysis of gene expression in the prefrontal cortex in schizophrenia: a preliminary study , 2002, Schizophrenia Research.
[86] G. Westbrook,et al. Synapse Density Regulates Independence at Unitary Inhibitory Synapses , 2003, The Journal of Neuroscience.
[87] A. Zaitsev,et al. Localization of calcium-binding proteins in physiologically and morphologically characterized interneurons of monkey dorsolateral prefrontal cortex. , 2005, Cerebral cortex.
[88] J. Pierri,et al. Alterations in chandelier neuron axon terminals in the prefrontal cortex of schizophrenic subjects. , 1999, The American journal of psychiatry.
[89] Juha Voipio,et al. GABAergic Depolarization of the Axon Initial Segment in Cortical Principal Neurons Is Caused by the Na–K–2Cl Cotransporter NKCC1 , 2008, The Journal of Neuroscience.
[90] M. Rasband,et al. Spectrin and Ankyrin-Based Cytoskeletons at Polarized Domains in Myelinated Axons , 2008, Experimental biology and medicine.
[91] Michael F. Green,et al. What are the functional consequences of neurocognitive deficits in schizophrenia? , 1996, The American journal of psychiatry.
[92] Priscilla Wu,et al. Ankyrin-Based Subcellular Gradient of Neurofascin, an Immunoglobulin Family Protein, Directs GABAergic Innervation at Purkinje Axon Initial Segment , 2004, Cell.
[93] Y. Kubota,et al. Physiological and morphological identification of somatostatin- or vasoactive intestinal polypeptide-containing cells among GABAergic cell subtypes in rat frontal cortex , 1996, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[94] P. De Camilli,et al. βIVΣ1 spectrin stabilizes the nodes of Ranvier and axon initial segments , 2004, The Journal of cell biology.
[95] A. Zaitsev,et al. Interneuron diversity in layers 2-3 of monkey prefrontal cortex. , 2009, Cerebral cortex.
[96] G. Curzon,et al. TRANSMITTERS AND RELATED SUBSTANCES IN BRAIN MATERIAL: AN INDEX OF TERMINAL AND/OR POST-MORTEM BIOCHEMICAL CHANGE , 1978, The Lancet.
[97] N. Lazar,et al. Maturation of cognitive processes from late childhood to adulthood. , 2004, Child development.
[98] R. Nicoll,et al. Local and diffuse synaptic actions of GABA in the hippocampus , 1993, Neuron.
[99] Robert J. Harvey,et al. Gephyrin: where do we stand, where do we go? , 2008, Trends in Neurosciences.
[100] Wei Zhang,et al. Reduced Dendritic Spine Density in Auditory Cortex of Subjects with Schizophrenia , 2009, Neuropsychopharmacology.
[101] P. Somogyi,et al. Differential synaptic localization of two major y-aminobutyric acid type A receptor a subunits on hippocampal pyramidal cells , 1996 .
[102] Yogesh K. Dwivedi,et al. A decrease of reelin expression as a putative vulnerability factor in schizophrenia. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[103] M. Pangalos,et al. The Clustering of GABAA Receptor Subtypes at Inhibitory Synapses is Facilitated via the Direct Binding of Receptor α2 Subunits to Gephyrin , 2008, The Journal of Neuroscience.
[104] K. Mirnics,et al. Molecular markers distinguishing supragranular and infragranular layers in the human prefrontal cortex , 2007, The European journal of neuroscience.
[105] E. G. Jones,et al. Gene expression for glutamic acid decarboxylase is reduced without loss of neurons in prefrontal cortex of schizophrenics. , 1995, Archives of general psychiatry.
[106] T. Insel,et al. Cure therapeutics and strategic prevention: raising the bar for mental health research , 2006, Molecular Psychiatry.