Kidney injury molecule‐1 and N‐acetyl‐ß‐d‐glucosaminidase in chronic heart failure: possible biomarkers of cardiorenal syndrome

Patients with chronic heart failure are often characterized by impaired renal function, also referred to as cardiorenal syndrome (CRS). The aim of this study was to assess whether novel markers of kidney injury are elevated in chronic heart failure and CRS.

[1]  L. Bongartz.  The Severe Cardiorenal Syndrome , 2011 .

[2]  A. Passantino,et al.  The cardiorenal anaemia syndrome in systolic heart failure: prevalence, clinical correlates, and long‐term survival , 2011, European journal of heart failure.

[3]  C. Ronco,et al.  Volume overload and cardiorenal syndromes. , 2010, Congestive heart failure.

[4]  G. Navis,et al.  Tubular damage in chronic systolic heart failure is associated with reduced survival independent of glomerular filtration rate , 2010, Heart.

[5]  O. Shaker,et al.  Early Diagnostic Markers for Contrast Nephropathy in Patients Undergoing Coronary Angiography , 2010, Angiology.

[6]  H. Ince,et al.  The impact of impaired renal function on mortality in patients with acutely decompensated chronic heart failure , 2010, European journal of heart failure.

[7]  Nobuaki Kobayashi,et al.  Acute kidney injury and outcomes in acute decompensated heart failure: evaluation of the RIFLE criteria in an acutely ill heart failure population , 2010, European journal of heart failure.

[8]  P. Ponikowski,et al.  Cardio-renal syndromes: report from the consensus conference of the Acute Dialysis Quality Initiative , 2009, European heart journal.

[9]  D. J. Veldhuisen,et al.  Both in‐ and out‐hospital worsening of renal function predict outcome in patients with heart failure: results from the Coordinating Study Evaluating Outcome of Advising and Counseling in Heart Failure (COACH) , 2009, European journal of heart failure.

[10]  J. Małyszko,et al.  Serum Neutrophil Gelatinase-Associated Lipocalin as a Marker of Renal Function in Patients with Chronic Heart Failure and Coronary Artery Disease , 2009, Kidney and Blood Pressure Research.

[11]  D. Bolignano,et al.  Increased plasma neutrophil gelatinase-associated lipocalin levels predict mortality in elderly patients with chronic heart failure. , 2009, Rejuvenation research.

[12]  G. Navis,et al.  Increased central venous pressure is associated with impaired renal function and mortality in a broad spectrum of patients with cardiovascular disease. , 2009, Journal of the American College of Cardiology.

[13]  D. J. Veldhuisen,et al.  Urinary neutrophil gelatinase associated lipocalin (NGAL), a marker of tubular damage, is increased in patients with chronic heart failure , 2008, European journal of heart failure.

[14]  Michael Böhm,et al.  Kommentar zu den ESC-Guidelines for the Diagnosis and Treatment of Acute and Chronic Heart Failure 2008 , 2008, European journal of heart failure.

[15]  J Concato,et al.  Biomarkers for the diagnosis and risk stratification of acute kidney injury: a systematic review. , 2008, Kidney international.

[16]  T. Nickolas,et al.  Biomarkers in acute and chronic kidney disease , 2008, Current opinion in nephrology and hypertension.

[17]  Qing Ma,et al.  Neutrophil gelatinase-associated lipocalin (NGAL) as a biomarker for acute renal injury after cardiac surgery , 2005, The Lancet.

[18]  B. Massie,et al.  The clinical challenge of cardiorenal syndrome. , 2004, Circulation.

[19]  R. Schrier,et al.  Acute renal failure: definitions, diagnosis, pathogenesis, and therapy. , 2004, The Journal of clinical investigation.

[20]  T. MacDonald,et al.  Review of aldosterone- and angiotensin II-induced target organ damage and prevention. , 2004, Cardiovascular research.

[21]  L. Feligioni,et al.  Activity and isoenzyme profile of N-acetyl-beta-D-glucosaminidase in urine from workers exposed to cadmium. , 2000, Clinica chimica acta; international journal of clinical chemistry.

[22]  Joseph V. Bonventre,et al.  Kidney Injury Molecule-1 (KIM-1), a Putative Epithelial Cell Adhesion Molecule Containing a Novel Immunoglobulin Domain, Is Up-regulated in Renal Cells after Injury* , 1998, The Journal of Biological Chemistry.

[23]  S. Takebayashi,et al.  Idiopathic membranous glomerulonephritis: a clinicopathologic and quantitative morphometric study. , 1992, Clinical nephrology.

[24]  R. Arora,et al.  Cardiorenal syndrome: A literature review. , 2008, Experimental and clinical cardiology.

[25]  P. Doevendans,et al.  Renal function; , 2004 .

[26]  Price Rg.  The role of NAG (N-acetyl-beta-D-glucosaminidase) in the diagnosis of kidney disease including the monitoring of nephrotoxicity. , 1992 .

[27]  R. G. Price.  The role of NAG (N-acetyl-beta-D-glucosaminidase) in the diagnosis of kidney disease including the monitoring of nephrotoxicity. , 1992, Clinical nephrology.