Toll-like receptors activate programmed necrosis in macrophages through a receptor-interacting kinase-3–mediated pathway
暂无分享,去创建一个
[1] G. Häcker,et al. cIAPs Block Ripoptosome Formation, a RIP1/Caspase-8 Containing Intracellular Cell Death Complex Differentially Regulated by cFLIP Isoforms , 2011, Molecular cell.
[2] T. Tenev,et al. The Ripoptosome, a signaling platform that assembles in response to genotoxic stress and loss of IAPs. , 2011, Molecular cell.
[3] Guy S. Salvesen,et al. Catalytic activity of the caspase-8-FLIPL complex inhibits RIPK3-dependent necrosis , 2011, Nature.
[4] Joan W. Miller,et al. Receptor interacting protein kinases mediate retinal detachment-induced photoreceptor necrosis and compensate for inhibition of apoptosis , 2010, Proceedings of the National Academy of Sciences.
[5] P. Vandenabeele,et al. Molecular mechanisms of necroptosis: an ordered cellular explosion , 2010, Nature Reviews Molecular Cell Biology.
[6] L. O’Neill,et al. New Insights into the Regulation of Signalling by Toll-Like Receptors and Nod-Like Receptors , 2010, Journal of Innate Immunity.
[7] S. Akira,et al. The role of pattern-recognition receptors in innate immunity: update on Toll-like receptors , 2010, Nature Immunology.
[8] W. Kaiser,et al. Virus inhibition of RIP3-dependent necrosis. , 2010, Cell host & microbe.
[9] Na Zhang,et al. RIP3, an Energy Metabolism Regulator That Switches TNF-Induced Cell Death from Apoptosis to Necrosis , 2009, Science.
[10] F. Chan,et al. Phosphorylation-Driven Assembly of the RIP1-RIP3 Complex Regulates Programmed Necrosis and Virus-Induced Inflammation , 2009, Cell.
[11] Tao Wang,et al. Receptor Interacting Protein Kinase-3 Determines Cellular Necrotic Response to TNF-α , 2009, Cell.
[12] T. Kawamoto,et al. TAK-242 selectively suppresses Toll-like receptor 4-signaling mediated by the intracellular domain. , 2008, European journal of pharmacology.
[13] Yide Mei,et al. Cleavage of RIP3 inactivates its caspase-independent apoptosis pathway by removal of kinase domain. , 2007, Cellular signalling.
[14] You-Sun Kim,et al. TNF-induced activation of the Nox1 NADPH oxidase and its role in the induction of necrotic cell death. , 2007, Molecular cell.
[15] H. Osada,et al. Inhibition of ADP/ATP Exchange in Receptor-Interacting Protein-Mediated Necrosis , 2006, Molecular and Cellular Biology.
[16] R. Pope,et al. NF-kappaB protects macrophages from lipopolysaccharide-induced cell death: the role of caspase 8 and receptor-interacting protein. , 2005, The Journal of biological chemistry.
[17] Alexei Degterev,et al. Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury , 2005, Nature chemical biology.
[18] Xiaodong Wang,et al. A Small Molecule Smac Mimic Potentiates TRAIL- and TNFα-Mediated Cell Death , 2004, Science.
[19] S. Akira,et al. Functions of toll-like receptors: lessons from KO mice. , 2004, Comptes rendus biologies.
[20] F. Martinon,et al. RIP1 is an essential mediator of Toll-like receptor 3–induced NF-κB activation , 2004, Nature Immunology.
[21] Xiaodong Wang,et al. A small molecule Smac mimic potentiates TRAIL- and TNFalpha-mediated cell death. , 2004, Science.
[22] Kay Hofmann,et al. RIP1 is an essential mediator of Toll-like receptor 3-induced NF-kappa B activation. , 2004, Nature immunology.
[23] B. Moss,et al. A Role for Tumor Necrosis Factor Receptor-2 and Receptor-interacting Protein in Programmed Necrosis and Antiviral Responses* , 2003, Journal of Biological Chemistry.
[24] Jiahuai Han,et al. Identification of Lps2 as a key transducer of MyD88-independent TIR signalling , 2003, Nature.
[25] S. Akira,et al. Role of Adaptor TRIF in the MyD88-Independent Toll-Like Receptor Signaling Pathway , 2003, Science.
[26] P. Vandenabeele,et al. Tipping the balance between necrosis and apoptosis in human and murine cells treated with interferon and dsRNA , 2002, Cell Death and Differentiation.
[27] R. Flavell,et al. Recognition of double-stranded RNA and activation of NF-κB by Toll-like receptor 3 , 2001, Nature.
[28] S. Akira,et al. Unresponsiveness of MyD88-deficient mice to endotoxin. , 1999, Immunity.
[29] S. Akira,et al. Cutting edge: Toll-like receptor 4 (TLR4)-deficient mice are hyporesponsive to lipopolysaccharide: evidence for TLR4 as the Lps gene product. , 1999, Journal of immunology.
[30] P. Ricciardi-Castagnoli,et al. Defective LPS signaling in C3H/HeJ and C57BL/10ScCr mice: mutations in Tlr4 gene. , 1998, Science.
[31] Stefan Grimm,et al. The Death Domain Kinase RIP Mediates the TNF-Induced NF-κB Signal , 1998 .
[32] P. Leder,et al. The death domain kinase RIP mediates the TNF-induced NF-kappaB signal. , 1998, Immunity.