BRCC3 mutations in myeloid neoplasms
暂无分享,去创建一个
H. Aburatani | S. Miyano | Y. Shiraishi | K. Chiba | S. Ogawa | T. Haferlach | Y. Shiozawa | S. Schnittger | W. Kern | Y. Okuno | G. Nagae | Y. Nagata | Kenichi Yoshida | A. Kon | M. Sanada | H. Klein | C. Haferlach | H. Koeffler | M. Dugas | M. Sekeres | H. Makishima | N. Hosono | B. Przychodzen | James G. Phillips | T. Radivoyevitch | M. Clemente | V. Grossmann | C. Polprasert | T. Alpermann | W. Shen | N. Nguyen | Hiroko Tanaka | Yang Du | N. Nadarajah | K. Oakley | Dayong Huang | Wenyi Shen | Da-yong Huang
[1] P. Jeggo,et al. DNA DSB repair pathway choice: an orchestrated handover mechanism. , 2014, The British journal of radiology.
[2] M. Stratton,et al. Clinical and biological implications of driver mutations in myelodysplastic syndromes. , 2013, Blood.
[3] C Haferlach,et al. Landscape of genetic lesions in 944 patients with myelodysplastic syndromes , 2013, Leukemia.
[4] S. Miyano,et al. Somatic SETBP1 mutations in myeloid malignancies , 2013, Nature Genetics.
[5] A. Jankowska,et al. Multiple mechanisms deregulate EZH2 and histone H3 lysine 27 epigenetic changes in myeloid malignancies , 2013, Leukemia.
[6] Benjamin J. Raphael,et al. Genomic and epigenomic landscapes of adult de novo acute myeloid leukemia. , 2013, The New England journal of medicine.
[7] Angela G. Fleischman,et al. Oncogenic CSF3R mutations in chronic neutrophilic leukemia and atypical CML. , 2013, The New England journal of medicine.
[8] Kumar Somyajit,et al. ATM- and ATR-Mediated Phosphorylation of XRCC3 Regulates DNA Double-Strand Break-Induced Checkpoint Activation and Repair , 2013, Molecular and Cellular Biology.
[9] Jiandong Chen,et al. p53 promotes repair of heterochromatin DNA by regulating JMJD2b and SUV39H1 expression , 2013, Oncogene.
[10] Masao Nagasaki,et al. Recurrent mutations in multiple components of the cohesin complex in myeloid neoplasms , 2012, Nature Genetics.
[11] C. Richter-Landsberg,et al. The small molecule inhibitor PR-619 of deubiquitinating enzymes affects the microtubule network and causes protein aggregate formation in neural cells: implications for neurodegenerative diseases. , 2012, Biochimica et biophysica acta.
[12] N. Copeland,et al. Setbp1 promotes the self-renewal of murine myeloid progenitors via activation of Hoxa9 and Hoxa10. , 2012, Blood.
[13] Junjie Chen,et al. RAP80 Protein Is Important for Genomic Stability and Is Required for Stabilizing BRCA1-A Complex at DNA Damage Sites in Vivo* , 2012, The Journal of Biological Chemistry.
[14] A. Jankowska,et al. Mutations in the spliceosome machinery, a novel and ubiquitous pathway in leukemogenesis. , 2012, Blood.
[15] Ken Chen,et al. Clonal architecture of secondary acute myeloid leukemia. , 2012, The New England journal of medicine.
[16] C. O'keefe,et al. CBL mutation-related patterns of phosphorylation and sensitivity to tyrosine kinase inhibitors , 2012, Leukemia.
[17] M. Stratton,et al. Somatic SF3B1 mutation in myelodysplasia with ring sideroblasts. , 2011, The New England journal of medicine.
[18] S. Sugano,et al. Frequent pathway mutations of splicing machinery in myelodysplasia , 2011, Nature.
[19] D. Neuberg,et al. Clinical effect of point mutations in myelodysplastic syndromes. , 2011, The New England journal of medicine.
[20] J. Licht,et al. DNMT3A mutations in acute myeloid leukemia , 2011, Nature Genetics.
[21] R. Greenberg,et al. The BRCA1-RAP80 Complex Regulates DNA Repair Mechanism Utilization by Restricting End Resection* , 2011, The Journal of Biological Chemistry.
[22] Junjie Chen,et al. The Lys63-specific Deubiquitinating Enzyme BRCC36 Is Regulated by Two Scaffold Proteins Localizing in Different Subcellular Compartments* , 2010, The Journal of Biological Chemistry.
[23] J. Soulier,et al. Mutation in TET2 in myeloid cancers. , 2009, The New England journal of medicine.
[24] R. Greenberg,et al. The Rap80-BRCC36 de-ubiquitinating enzyme complex antagonizes RNF8-Ubc13-dependent ubiquitination events at DNA double strand breaks , 2009, Proceedings of the National Academy of Sciences.
[25] C. O'keefe,et al. Chromosomal lesions and uniparental disomy detected by SNP arrays in MDS, MDS/MPD, and MDS-derived AML. , 2008, Blood.
[26] N. Shinton. WHO Classification of Tumors of Hematopoietic and Lymphoid Tissues , 2007 .
[27] M. Hollstein,et al. p53 gain-of-function cancer mutants induce genetic instability by inactivating ATM , 2007, Nature Cell Biology.
[28] Shigeru Chiba,et al. A robust algorithm for copy number detection using high-density oligonucleotide single nucleotide polymorphism genotyping arrays. , 2005, Cancer research.
[29] P. Olive,et al. Radiation Sensitivity, H2AX Phosphorylation, and Kinetics of Repair of DNA Strand Breaks in Irradiated Cervical Cancer Cell Lines , 2004, Cancer Research.
[30] D. Ramsden,et al. The DNA‐dependent protein kinase: the director at the end , 2004, Immunological reviews.
[31] D. Lane,et al. The p53 tumour suppressor gene , 1998, The British journal of surgery.
[32] C. Croce,et al. ATM mutations in cancer families. , 1996, Cancer research.
[33] M. Jasin,et al. Genetic manipulation of genomes with rare-cutting endonucleases. , 1996, Trends in genetics : TIG.
[34] G. Lenoir,et al. Increasing incidence of breast cancer in family with BRCA1 mutation , 1993, The Lancet.
[35] F. Collins,et al. Genetic counseling for families with inherited susceptibility to breast and ovarian cancer. , 1993, JAMA.
[36] J. Fraumeni,et al. Rhabdomyosarcoma in children: epidemiologic study and identification of a familial cancer syndrome. , 1969, Journal of the National Cancer Institute.