Oral cicaprost protects from hypercholesterolaemia-induced impairment of coronary vasodilation.

[1]  M. Nakamura,et al.  Putative mechanisms of the impairment of endothelium-dependent relaxation of the aorta with atheromatous plaque in heritable hyperlipidemic rabbits. , 1991, Circulation research.

[2]  D. Harrison,et al.  Diet-induced atherosclerosis increases the release of nitrogen oxides from rabbit aorta. , 1990, The Journal of clinical investigation.

[3]  D. Harrison,et al.  Endothelium-dependent vascular relaxation is abnormal in the coronary microcirculation of atherosclerotic primates. , 1990, Circulation.

[4]  A. M. Lefer,et al.  Lack of endothelium-dependent relaxation in coronary resistance arteries of cholesterol-fed rabbits. , 1989, The American journal of physiology.

[5]  A. Herman,et al.  Biphasic response of intimal prostacyclin production during the development of experimental atherosclerosis. , 1986, Prostaglandins.

[6]  A. Herman,et al.  Effect of Hypercholesterolemia on Vascular Reactivity in the Rabbit: I. Endothelium‐Dependent and Endothelium‐Independent Contractions and Relaxations in Isolated Arteries of Control and Hypercholesterolemic Rabbits , 1986, Circulation research.

[7]  V. Tertov,et al.  PROSTACYCLIN ANALOGUES AS ANTIATHEROSCLEROTIC DRUGS , 1983, The Lancet.

[8]  K. Schrör,et al.  PGI2 Prevents Ischemia‐Induced Alterations in Cardiac Catecholamines Without Influencing Nerve‐Stimulation-Induced Catecholamine Release in Nonischemic Conditions , 1982, Journal of cardiovascular pharmacology.

[9]  A. Dembińska-kieć,et al.  The generation of prostacyclin by arteries and by the coronary vascular bed is reduced in experimental atherosclerosis in rabbits. , 1977, Prostaglandins.

[10]  S. Stürzebecher,et al.  Pharmacological profile of a novel carbacyclin derivative with high metabolic stability and oral activity in the rat. , 1986, Prostaglandins.