Oxidative stress in pregnant women and birth weight reduction.
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E. Ha | Yun-Chul Hong | K. Lee | H. Park | E. Park | Young-Ju Kim | Hye-Sung Moon
[1] E. Ha,et al. Effects of the GSTM1 and GSTT1 Polymorphisms on the Relationship Between Maternal Exposure to Environmental Tobacco Smoke and Neonatal Birth Weight , 2003, Journal of occupational and environmental medicine.
[2] K. Maruyama,et al. Erratum: Oxidative stress in very low birth weight infants as measured by urinary 8-OhdG (Free Radical Research vol. 36 (2) (189-193)) , 2002 .
[3] D. Christiani,et al. Genetic susceptibility of term pregnant women to oxidative damage. , 2002, Toxicology Letters.
[4] J. Suzin,et al. Evaluation of oxidative stress indices during treatment in pregnant women with intrauterine growth retardation. , 2002, Medical science monitor : international medical journal of experimental and clinical research.
[5] D. Christiani,et al. Effects of air pollutants on acute stroke mortality. , 2002, Environmental health perspectives.
[6] M. Tanimura,et al. Oxidative Stress in Very Low Birth Weight Infants As Measured by Urinary 8-OHdG , 2002, Free radical research.
[7] J. Kendrick,et al. Effects of smoking reduction during pregnancy on the birth weight of term infants. , 2001, American journal of epidemiology.
[8] J. Moodley,et al. Oxidative stress in pre‐eclampsia , 2001, Acta obstetricia et gynecologica Scandinavica.
[9] J. Jaakkola,et al. Fetal growth and length of gestation in relation to prenatal exposure to environmental tobacco smoke assessed by hair nicotine concentration. , 2001, Environmental health perspectives.
[10] U. Eriksson,et al. Combined Treatment with Vitamin E and Vitamin C Decreases Oxidative Stress and Improves Fetal Outcome in Experimental Diabetic Pregnancy , 2001, Pediatric Research.
[11] S. Walsh,et al. Increased superoxide generation is associated with decreased superoxide dismutase activity and mRNA expression in placental trophoblast cells in pre-eclampsia. , 2001, Placenta.
[12] T. Stein,et al. Oxidant damage to DNA and pregnancy outcome , 2001, The Journal of maternal-fetal medicine.
[13] T. Inder,et al. Protein Carbonyls and Lipid Peroxidation Products as Oxidation Markers in Preterm Infant Plasma: Associations with Chronic Lung Disease and Retinopathy and Effects of Selenium Supplementation , 2000, Pediatric Research.
[14] H. Thompson,et al. Effect of increased vegetable and fruit consumption on markers of oxidative cellular damage. , 1999, Carcinogenesis.
[15] G. Aykaç-toker,et al. Imbalance between Lipid Peroxidation and Antioxidant Status in Preeclampsia , 1998, Gynecologic and Obstetric Investigation.
[16] R. Cooke,et al. Comparison of urinary and plasma malondialdehyde in preterm infants. , 1997, Clinica chimica acta; international journal of clinical chemistry.
[17] A. Tjønneland,et al. Oxidative DNA damage estimated by 8-hydroxydeoxyguanosine excretion in humans: influence of smoking, gender and body mass index. , 1992, Carcinogenesis.
[18] M. Hallman,et al. Correlation of free oxygen radical-induced lipid peroxidation with outcome in very low birth weight infants. , 1990, The Journal of pediatrics.
[19] S. Nishimura,et al. Hydroxylation of deoxyguanosine at the C-8 position by ascorbic acid and other reducing agents. , 1984, Nucleic acids research.
[20] P. Leanderson,et al. Cigarette smoke-induced DNA damage in cultured human lung cells: role of hydroxyl radicals and endonuclease activation. , 1992, Chemico-biological interactions.