Effects of Cholesterol and Nuclear Hormone Receptor Agonists on the Production of Transforming Growth Factor-β in Macrophages

[1]  M. Dushkin,et al.  Cholesterol-induced stimulation of postinflammatory liver fibrosis , 2008, Bulletin of Experimental Biology and Medicine.

[2]  P. Edwards,et al.  Regulation of hepatic Insig-2 by the farnesoid X receptor. , 2007, Molecular endocrinology.

[3]  M. E. Gazzar HMGB1 modulates inflammatory responses in LPS-activated macrophages , 2007, Inflammation Research.

[4]  Joseph L Goldstein,et al.  Insig-dependent Ubiquitination and Degradation of Mammalian 3-Hydroxy-3-methylglutaryl-CoA Reductase Stimulated by Sterols and Geranylgeraniol* , 2003, Journal of Biological Chemistry.

[5]  J. Cavaillon,et al.  Endotoxin tolerance: is there a clinical relevance? , 2003, Journal of endotoxin research.

[6]  P. Claus,et al.  Differential Intranuclear Localization of Fibroblast Growth Factor-2 Isoforms and Specific Interaction with the Survival of Motoneuron Protein* , 2003, The Journal of Biological Chemistry.

[7]  M. Di Nisio,et al.  Increased transforming growth factor-beta(1) circulating levels and production in human monocytes after 3-hydroxy-3-methyl-glutaryl-coenzyme a reductase inhibition with pravastatin. , 2002, Journal of the American College of Cardiology.

[8]  B. Hamilton,et al.  Decreased production of TGF-beta 1 by human alveolar macrophages compared with blood monocytes. , 1996, Journal of immunology.

[9]  K. Feingold,et al.  Discordant regulation of proteins of cholesterol metabolism during the acute phase response. , 1995, Journal of lipid research.

[10]  M. Parmely,et al.  Transforming growth factor beta 1 and gamma interferon provide opposing signals to lipopolysaccharide-activated mouse macrophages , 1994, Infection and immunity.

[11]  H. Shimokawa,et al.  Different Effects of Simvastatin and Losartan on Cytokine Levels in Coronary Artery Disease , 2006, American journal of cardiovascular drugs : drugs, devices, and other interventions.

[12]  V. Fadok,et al.  Phosphatidylserine-dependent ingestion of apoptotic cells promotes TGF-beta1 secretion and the resolution of inflammation. , 2002, The Journal of clinical investigation.

[13]  T. Shimokama,et al.  Effects of hypercholesterolemia on initial and chronic phases of rat nephrotoxic serum nephritis: development of focal segmental glomerulosclerosis, analogous to atherosclerosis , 1993, Virchows Archiv. B, Cell pathology including molecular pathology.