beta-Arrestins facilitate ubiquitin-dependent degradation of apoptosis signal-regulating kinase 1 (ASK1) and attenuate H2O2-induced apoptosis.

[1]  J. Benovic,et al.  Arrestin-2 Interacts with the Ubiquitin-Protein Isopeptide Ligase Atrophin-interacting Protein 4 and Mediates Endosomal Sorting of the Chemokine Receptor CXCR4* , 2007, Journal of Biological Chemistry.

[2]  Lan Ma,et al.  β-arrestin signaling and regulation of transcription , 2007, Journal of Cell Science.

[3]  Y. Jiang,et al.  Human glutathione S-transferase P1-1 interacts with TRAF2 and regulates TRAF2–ASK1 signals , 2006, Oncogene.

[4]  M. Bogoyevitch The isoform‐specific functions of the c‐Jun N‐terminal Kinases (JNKs): differences revealed by gene targeting , 2006, BioEssays : news and reviews in molecular, cellular and developmental biology.

[5]  G. Pei,et al.  Association of β-arrestin and TRAF6 negatively regulates Toll-like receptor–interleukin 1 receptor signaling , 2006, Nature Immunology.

[6]  O. Larsson,et al.  β-Arrestin Is Crucial for Ubiquitination and Down-regulation of the Insulin-like Growth Factor-1 Receptor by Acting as Adaptor for the MDM2 E3 Ligase* , 2005, Journal of Biological Chemistry.

[7]  T. Dawson,et al.  Mediation of cell death by poly(ADP-ribose) polymerase-1. , 2005, Pharmacological research.

[8]  W. Min,et al.  Hsp90–Akt phosphorylates ASK1 and inhibits ASK1-mediated apoptosis , 2005, Oncogene.

[9]  Robert J. Lefkowitz,et al.  Transduction of Receptor Signals by ß-Arrestins , 2005, Science.

[10]  Wang Min,et al.  AIP1/DAB2IP, a Novel Member of the Ras-GAP Family, Transduces TRAF2-induced ASK1-JNK Activation* , 2004, Journal of Biological Chemistry.

[11]  H. Ichijo,et al.  The ASK1-MAP kinase cascades in mammalian stress response. , 2004, Journal of biochemistry.

[12]  W. Min,et al.  Thioredoxin-2 Inhibits Mitochondria-Located ASK1-Mediated Apoptosis in a JNK-Independent Manner , 2004, Circulation research.

[13]  D. Cimino,et al.  Arrestins Block G Protein-coupled Receptor-mediated Apoptosis* , 2004, Journal of Biological Chemistry.

[14]  Yue Sun,et al.  Identification of β-Arrestin2 as a G Protein-Coupled Receptor-Stimulated Regulator of NF-κB Pathways , 2004 .

[15]  Robert J. Lefkowitz,et al.  beta-arrestins: traffic cops of cell signaling. , 2004, Current opinion in cell biology.

[16]  Ping Wang,et al.  Subcellular Localization of β-Arrestins Is Determined by Their Intact N Domain and the Nuclear Export Signal at the C Terminus* , 2003, The Journal of Biological Chemistry.

[17]  Ping Wang,et al.  β-Arrestin 2 Functions as a G-Protein-coupled Receptor-activated Regulator of Oncoprotein Mdm2* , 2003, The Journal of Biological Chemistry.

[18]  M. Suntharalingam,et al.  Role of Glutaredoxin in Metabolic Oxidative Stress , 2002, The Journal of Biological Chemistry.

[19]  G. Johnson,et al.  Signalling: Mixed-lineage kinase control of JNK and p38 MAPK pathways , 2002, Nature Reviews Molecular Cell Biology.

[20]  Kiyoshi Inoue,et al.  ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats. , 2002, Genes & development.

[21]  H. Ichijo,et al.  Activation of apoptosis signal‐regulating kinase 1 by the stress‐induced activating phosphorylation of pre‐formed oligomer , 2002, Journal of cellular physiology.

[22]  H. Ichijo,et al.  Negative feedback regulation of ASK1 by protein phosphatase 5 (PP5) in response to oxidative stress , 2001, The EMBO journal.

[23]  K. Kang,et al.  Glutathione S-Transferase Mu Modulates the Stress-activated Signals by Suppressing Apoptosis Signal-regulating Kinase 1* , 2001, The Journal of Biological Chemistry.

[24]  W. Min,et al.  Laminar flow inhibits TNF-induced ASK1 activation by preventing dissociation of ASK1 from its inhibitor 14-3-3. , 2001, The Journal of clinical investigation.

[25]  Colin Adrain,et al.  Executioner Caspase-3, -6, and -7 Perform Distinct, Non-redundant Roles during the Demolition Phase of Apoptosis* , 2001, The Journal of Biological Chemistry.

[26]  Jeong-Sun Seo,et al.  Hsp72 functions as a natural inhibitory protein of c‐Jun N‐terminal kinase , 2001, The EMBO journal.

[27]  R. Lefkowitz,et al.  Beta-arrestin 2: a receptor-regulated MAPK scaffold for the activation of JNK3. , 2000, Science.

[28]  R. Davis,et al.  Signal Transduction by the JNK Group of MAP Kinases , 2000, Cell.

[29]  K. Tew,et al.  Glutathione S-transferase p elicits protection against H2O2-induced cell death via coordinated regulation of stress kinases. , 2000, Cancer research.

[30]  D. Bar-Sagi,et al.  Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway. , 2000, Science.

[31]  Hong Liu,et al.  Activation of Apoptosis Signal-Regulating Kinase 1 (ASK1) by Tumor Necrosis Factor Receptor-Associated Factor 2 Requires Prior Dissociation of the ASK1 Inhibitor Thioredoxin , 2000, Molecular and Cellular Biology.

[32]  H. Ichijo,et al.  From receptors to stress-activated MAP kinases , 1999, Oncogene.

[33]  M. Caron,et al.  Beta-arrestin-dependent formation of beta2 adrenergic receptor-Src protein kinase complexes. , 1999, Science.

[34]  D. Baltimore,et al.  Activation of apoptosis signal-regulating kinase 1 (ASK1) by the adapter protein Daxx. , 1998, Science.

[35]  K. Miyazono,et al.  ASK1 is essential for JNK/SAPK activation by TRAF2. , 1998, Molecular cell.

[36]  Kohei Miyazono,et al.  Mammalian thioredoxin is a direct inhibitor of apoptosis signal‐regulating kinase (ASK) 1 , 1998, The EMBO journal.

[37]  H. K. Sluss,et al.  Selective interaction of JNK protein kinase isoforms with transcription factors. , 1996, The EMBO journal.

[38]  D. Whitmore,et al.  Cone arrestin identified by targeting expression of a functional family. , 1994, The Journal of biological chemistry.

[39]  J L Benovic,et al.  Visual arrestin interaction with rhodopsin. Sequential multisite binding ensures strict selectivity toward light-activated phosphorylated rhodopsin. , 1993, The Journal of biological chemistry.

[40]  M. Sallese,et al.  Molecular analysis of human beta-arrestin-1: cloning, tissue distribution, and regulation of expression. Identification of two isoforms generated by alternative splicing. , 1993, The Journal of biological chemistry.

[41]  C. Patterson,et al.  C-terminus of heat shock protein 70– interacting protein facilitates degradation of apoptosis signal-regulating kinase 1 and inhibits apoptosis signal-regulating kinase 1– dependent apoptosis , 2005, Cell stress & chaperones.

[42]  R. Davis,et al.  Signal transduction by the c-Jun N-terminal kinase. , 1999, Biochemical Society symposium.