Glucose and Lactate Concentrations in Cerebrospinal Fluid After Traumatic Brain Injury
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J. Vincent | S. Scolletta | R. Badenes | F. Taccone | M. Oddo | F. Franchi | A. Lozano | J. Creteur | O. Lheureux | Ramon J Seastres
[1] T. Carpenter,et al. A Comparison of Oxidative Lactate Metabolism in Traumatically Injured Brain and Control Brain , 2018, Journal of neurotrauma.
[2] K. Ho,et al. A comparison of prognostic significance of strong ion gap (SIG) with other acid-base markers in the critically ill: a cohort study , 2016, Journal of Intensive Care.
[3] L. Mascia,et al. Multivariate projection method to investigate inflammation associated with secondary insults and outcome after human traumatic brain injury: a pilot study , 2016, Journal of Neuroinflammation.
[4] U. Ungerstedt,et al. Consensus statement from the 2014 International Microdialysis Forum , 2015, Intensive Care Medicine.
[5] T. F. Group. Intensive versus conventional glucose control in critically ill patients with traumatic brain injury: long-term follow-up of a subgroup of patients from the NICE-SUGAR study , 2015 .
[6] Reto Meuli,et al. Cerebral metabolic effects of exogenous lactate supplementation on the injured human brain , 2014, Intensive Care Medicine.
[7] D. Menon,et al. Lactate uptake by the injured human brain: evidence from an arteriovenous gradient and cerebral microdialysis study. , 2013, Journal of neurotrauma.
[8] R. Meuli,et al. Cerebral Extracellular Lactate Increase is Predominantly Nonischemic in Patients with Severe Traumatic Brain Injury , 2013, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[9] S. Mayer,et al. Reduced Brain/Serum Glucose Ratios Predict Cerebral Metabolic Distress and Mortality After Severe Brain Injury , 2013, Neurocritical Care.
[10] P. Marik,et al. Stress hyperglycemia: an essential survival response! , 2013, Critical Care.
[11] Sung-Cheng Huang,et al. Tight glycemic control increases metabolic distress in traumatic brain injury: A randomized controlled within-subjects trial* , 2012, Critical care medicine.
[12] E. Yan,et al. Post-traumatic hypoxia exacerbates neurological deficit, neuroinflammation and cerebral metabolism in rats with diffuse traumatic brain injury , 2011, Journal of Neuroinflammation.
[13] J. Pickard,et al. Cerebral extracellular chemistry and outcome following traumatic brain injury: a microdialysis study of 223 patients. , 2011, Brain : a journal of neurology.
[14] S. Mayer,et al. Impact of tight glycemic control on cerebral glucose metabolism after severe brain injury: A microdialysis study* , 2008, Critical care medicine.
[15] F. Bilotta,et al. The Effect of Intensive Insulin Therapy on Infection Rate, Vasospasm, Neurologic Outcome, and Mortality in Neurointensive Care Unit After Intracranial Aneurysm Clipping in Patients With Acute Subarachnoid Hemorrhage: A Randomized Prospective Pilot Trial , 2007, Journal of neurosurgical anesthesiology.
[16] Juan Lu,et al. IMPACT database of traumatic brain injury: design and description. , 2007, Journal of neurotrauma.
[17] F Bruyninckx,et al. Insulin therapy protects the central and peripheral nervous system of intensive care patients , 2005, Neurology.
[18] L. Pellerin,et al. Cellular and subcellular distribution of monocarboxylate transporters in cultured brain cells and in the adult brain , 2005, Journal of neuroscience research.
[19] S. Wisniewski,et al. Continuous versus intermittent cerebrospinal fluid drainage after severe traumatic brain injury in children: effect on biochemical markers. , 2004, Journal of neurotrauma.
[20] M M Knudson,et al. Hypotension, hypoxia, and head injury: frequency, duration, and consequences. , 2001, Archives of surgery.
[21] M Uzura,et al. Prevention of secondary ischemic insults after severe head injury. , 1999, Critical care medicine.
[22] M Uzura,et al. Extracellular lactate and glucose alterations in the brain after head injury measured by microdialysis. , 1999, Critical care medicine.
[23] M E Phelps,et al. Cerebral hyperglycolysis following severe traumatic brain injury in humans: a positron emission tomography study. , 1997, Journal of neurosurgery.
[24] A. DeSalles,et al. Prognostic significance of ventricular CSF lactic acidosis in severe head injury. , 1986, Journal of neurosurgery.
[25] R. Hayes,et al. Regional brain metabolite levels following mild experimental head injury in the cat. , 1985, Journal of neurosurgery.