Inhibition of Chitinase-3-like-1 expression by K284 ameliorates lipopolysaccharide-induced acute liver injury through down regulation of CXCL3
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D. Son | In Jun Yeo | Hee-Pom Lee | J. Yun | S. Han | Minji Kim | Jin-Tae Hong | I. Yeo
[1] Jui-Ching Chen,et al. New Horizons of Macrophage Immunomodulation in the Healing of Diabetic Foot Ulcers , 2022, Pharmaceutics.
[2] Hanyu Qiu,et al. The Value of Serum CHI3L1 for the Diagnosis of Chronic Liver Diseases , 2022, International journal of general medicine.
[3] Chih-Chiang Chen,et al. Restoring Prohealing/Remodeling-Associated M2a/c Macrophages Using ON101 Accelerates Diabetic Wound Healing , 2022, JID innovations : skin science from molecules to population health.
[4] Ting Zhang,et al. Samotolisib Attenuates Acute Liver Injury Through Inhibiting Caspase-11-Mediated Pyroptosis Via Regulating E3 Ubiquitin Ligase Nedd4 , 2021, Frontiers in Pharmacology.
[5] J. Hong,et al. Inhibition of Chitinase-3-like-1 by K284-6111 Reduces Atopic Skin Inflammation via Repressing Lactoferrin , 2021, Immune network.
[6] J. Hong,et al. A small molecule targeting CHI3L1 inhibits lung metastasis by blocking IL‐13Rα2‐mediated JNK‐AP‐1 signals , 2021, Molecular oncology.
[7] Q. Yuan,et al. Senescent thyroid tumor cells promote their migration by inducing the polarization of M2-like macrophages , 2021, Clinical and Translational Oncology.
[8] Yingying Xu,et al. CHI3L1 (Chitinase 3 Like 1) upregulation is associated with macrophage signatures in esophageal cancer , 2021, Bioengineered.
[9] Jian Sun,et al. Gut-liver crosstalk in sepsis-induced liver injury , 2020, Critical Care.
[10] Xiaoren Zhang,et al. Chitinase-3 like-protein-1 function and its role in diseases , 2020, Signal Transduction and Targeted Therapy.
[11] M. N. Kim,et al. Chitinase 3-Like 1 Contributes to Food Allergy via M2 Macrophage Polarization , 2020, Allergy, asthma & immunology research.
[12] J. Hong,et al. K284-6111 alleviates memory impairment and neuroinflammation in Tg2576 mice by inhibition of Chitinase-3-like 1 regulating ERK-dependent PTX3 pathway , 2020, Journal of neuroinflammation.
[13] Wei-Hsin Sun,et al. Temporal expression patterns of distinct cytokines and M1/M2 macrophage polarization regulate rheumatoid arthritis progression , 2020, Molecular Biology Reports.
[14] Haibing Lan,et al. Downregulated CHI3L1 alleviates skeletal muscle stem cell injury in a mouse model of sepsis , 2020, IUBMB life.
[15] Xiaodong Sun,et al. Chitinase-3-Like-1 Promotes M2 Macrophage Differentiation and Induces Choroidal Neovascularization in Neovascular Age-Related Macular Degeneration. , 2019, Investigative ophthalmology & visual science.
[16] K. Tomita,et al. Chitinase 3‐like 1 deficiency ameliorates liver fibrosis by promoting hepatic macrophage apoptosis , 2019, Hepatology research : the official journal of the Japan Society of Hepatology.
[17] J. Hong,et al. Chitinase-3-like-1 deficiency attenuates ethanol-induced liver injury by inhibition of sterol regulatory element binding protein 1-dependent triglyceride synthesis. , 2019, Metabolism: clinical and experimental.
[18] Hyeongjin Cho,et al. Glucosamine improves survival in a mouse model of sepsis and attenuates sepsis-induced lung injury and inflammation , 2018, The Journal of Biological Chemistry.
[19] Yu Cao,et al. Park 7: A Novel Therapeutic Target for Macrophages in Sepsis-Induced Immunosuppression , 2018, Front. Immunol..
[20] Qingchong Qiu,et al. CHI3L1 promotes tumor progression by activating TGF-β signaling pathway in hepatocellular carcinoma , 2018, Scientific Reports.
[21] Jin Zhang,et al. Review: the Role and Mechanisms of Macrophage Autophagy in Sepsis , 2018, Inflammation.
[22] J. Hong,et al. K284-6111 prevents the amyloid beta-induced neuroinflammation and impairment of recognition memory through inhibition of NF-κB-mediated CHI3L1 expression , 2018, Journal of Neuroinflammation.
[23] Chris C. Miller,et al. Chitinase-3-like 1 is a biomarker of acute kidney injury and mortality in paediatric severe malaria , 2018, Malaria Journal.
[24] K. Yanagihara,et al. M1 and M2 Monocytes in Rheumatoid Arthritis: A Contribution of Imbalance of M1/M2 Monocytes to Osteoclastogenesis , 2018, Front. Immunol..
[25] K. Chayama,et al. Serum YKL-40 as a marker of liver fibrosis in patients with non-alcoholic fatty liver disease , 2016, Scientific Reports.
[26] S. Andrews,et al. Crosstalk between the lipopolysaccharide and phospholipid pathways during outer membrane biogenesis in Escherichia coli , 2016, Proceedings of the National Academy of Sciences.
[27] G. von Heijne,et al. Tissue-based map of the human proteome , 2015, Science.
[28] S. Bojesen,et al. YKL-40 and alcoholic liver and pancreas damage and disease in 86,258 individuals from the general population: cohort and mendelian randomization studies. , 2014, Clinical chemistry.
[29] R. Beelen,et al. Human macrophage polarization in vitro: maturation and activation methods compared. , 2014, Immunobiology.
[30] Dawei Wang,et al. Advances in sepsis-associated liver dysfunction , 2014, Burns & Trauma.
[31] N. Tennagels,et al. Chitinase-3-like protein 1 protects skeletal muscle from TNFα-induced inflammation and insulin resistance. , 2014, The Biochemical journal.
[32] Frank Tacke,et al. Macrophage heterogeneity in liver injury and fibrosis. , 2014, Journal of hepatology.
[33] S. Bicciato,et al. Transcriptomic Profiling of the Development of the Inflammatory Response in Human Monocytes In Vitro , 2014, PloS one.
[34] Cheng Huang,et al. The significance of YKL-40 protein in liver fibrosis , 2014, Inflammation Research.
[35] P. Kubes,et al. Neutrophil recruitment and function in health and inflammation , 2013, Nature Reviews Immunology.
[36] O. Fardel,et al. Polarization profiles of human M-CSF-generated macrophages and comparison of M1-markers in classically activated macrophages from GM-CSF and M-CSF origin. , 2013, Cellular immunology.
[37] Marco Manca,et al. Distribution of macrophage polarization markers in human atherosclerosis. , 2012, Atherosclerosis.
[38] Hai Qian,et al. LPS-induced nuclear translocation of RhoA is dependent on NF-κB in the human lung cancer cell line A549. , 2012, Oncology letters.
[39] P. Tak,et al. Intimal lining layer macrophages but not synovial sublining macrophages display an IL-10 polarized-like phenotype in chronic synovitis , 2012, Arthritis Research & Therapy.
[40] Hartmut Jaeschke,et al. Acetaminophen hepatotoxicity and repair: the role of sterile inflammation and innate immunity , 2012, Liver international : official journal of the International Association for the Study of the Liver.
[41] R. Schumann. Old and new findings on lipopolysaccharide-binding protein: a soluble pattern-recognition molecule. , 2011, Biochemical Society transactions.
[42] S. Perron,et al. Alveolar macrophages in allergic asthma: An expression signature characterized by heat shock protein pathways , 2009, Human Immunology.
[43] Ronald G. Crystal,et al. Smoking-Dependent Reprogramming of Alveolar Macrophage Polarization: Implication for Pathogenesis of Chronic Obstructive Pulmonary Disease1 , 2009, The Journal of Immunology.
[44] I. N. Crispe,et al. The liver as a lymphoid organ. , 2009, Annual review of immunology.
[45] C. Lee. Chitin, Chitinases and Chitinase-like Proteins in Allergic Inflammation and Tissue Remodeling , 2009, Yonsei medical journal.
[46] J. Johansen,et al. YKL-40: a novel marker shared by chronic inflammation and oncogenic transformation. , 2009, Methods in molecular biology.
[47] J. Mellors,et al. YKL-40, a marker of simian immunodeficiency virus encephalitis, modulates the biological activity of basic fibroblast growth factor. , 2008, The American journal of pathology.
[48] F. Coffman. Chitinase 3-Like-1 (CHI3L1): A Putative Disease Marker at the Interface of Proteomics and Glycomics , 2008, Critical reviews in clinical laboratory sciences.
[49] P. Metnitz,et al. Incidence and prognosis of early hepatic dysfunction in critically ill patients—A prospective multicenter study , 2007, Critical care medicine.
[50] P. Kristjansen,et al. Expression of YKL-40 by peritumoral macrophages in human small cell lung cancer. , 2005, Lung cancer.
[51] B. Levy,et al. Cyclooxygenase 2 Plays a Pivotal Role in the Resolution of Acute Lung Injury1 , 2005, The Journal of Immunology.
[52] P. Kristjansen,et al. Regulation of YKL‐40 expression during genotoxic or microenvironmental stress in human glioblastoma cells , 2005, Cancer science.
[53] H. Ling,et al. The chitinase 3-like protein human cartilage glycoprotein 39 inhibits cellular responses to the inflammatory cytokines interleukin-1 and tumour necrosis factor-alpha. , 2004, The Biochemical journal.
[54] H. Ling,et al. The chitinase 3-like protein human cartilage glycoprotein 39 (HC-gp39) stimulates proliferation of human connective-tissue cells and activates both extracellular signal-regulated kinase- and protein kinase B-mediated signalling pathways. , 2002, The Biochemical journal.
[55] D. Carter,et al. Characterization of a Murine Model of Endotoxin-Induced Acute Lung Injury , 2002, Shock.
[56] K. Ostergaard,et al. Studies on YKL-40 in knee joints of patients with rheumatoid arthritis and osteoarthritis. Involvement of YKL-40 in the joint pathology. , 2001, Osteoarthritis and cartilage.
[57] E. Rietschel,et al. Bacterial lipopolysaccharides and innate immunity. , 2001, Journal of endotoxin research.
[58] G. Verbruggen,et al. Comparative study of the synovial histology in rheumatoid arthritis, spondyloarthropathy, and osteoarthritis: influence of disease duration and activity , 2000, Annals of the rheumatic diseases.
[59] J. Henriksen,et al. Serum YKL-40 is increased in patients with hepatic fibrosis. , 2000, Journal of hepatology.
[60] C. D. de Vries,et al. Strong induction of members of the chitinase family of proteins in atherosclerosis: chitotriosidase and human cartilage gp-39 expressed in lesion macrophages. , 1999, Arteriosclerosis, thrombosis, and vascular biology.
[61] A. Muijsers,et al. Chitotriosidase, a chitinase, and the 39-kDa human cartilage glycoprotein, a chitin-binding lectin, are homologues of family 18 glycosyl hydrolases secreted by human macrophages. , 1998, European journal of biochemistry.