Tumor-derived granulocyte-macrophage colony-stimulating factor and granulocyte colony-stimulating factor prolong the survival of neutrophils infiltrating bronchoalveolar subtype pulmonary adenocarcinoma.

[1]  C. Haslett.  Granulocyte apoptosis and its role in the resolution and control of lung inflammation. , 1999, American journal of respiratory and critical care medicine.

[2]  I. Takanami,et al.  Tumor-Associated Macrophage Infiltration in Pulmonary Adenocarcinoma: Association with Angiogenesis and Poor Prognosis , 1999, Oncology.

[3]  T. Hugli,et al.  Multiple epithelial cell-derived factors enhance neutrophil survival. Regulation by glucocorticoids and tumor necrosis factor-alpha. , 1999, American journal of respiratory cell and molecular biology.

[4]  Takaaki Ito,et al.  Atypical adenomatous hyperplasia of the lung. Implications for the pathogenesis of peripheral lung adenocarcinoma. , 1999, American journal of clinical pathology.

[5]  K. Kubo,et al.  Alveolar type II-like cells release G-CSF as neutrophil chemotactic activity. , 1998, American journal of physiology. Lung cellular and molecular physiology.

[6]  L. Wakefield,et al.  Transforming growth factor-β1 is a new form of tumor suppressor with true haploid insufficiency , 1998, Nature Medicine.

[7]  G. Dranoff,et al.  GM-CSF enhances lung growth and causes alveolar type II epithelial cell hyperplasia in transgenic mice. , 1997, American journal of physiology. Lung cellular and molecular physiology.

[8]  C. Haslett,et al.  Regulation of neutrophil apoptosis by tumor necrosis factor-alpha: requirement for TNFR55 and TNFR75 for induction of apoptosis in vitro. , 1997, Blood.

[9]  C. Angeletti,et al.  Angiogenesis as a prognostic indicator of survival in non-small-cell lung carcinoma: a prospective study. , 1997, Journal of the National Cancer Institute.

[10]  J. Jackson,et al.  Stimulated human leukocytes cause activating mutations in the K-ras proto-oncogene , 1997, Oncogene.

[11]  J. Marshall,et al.  Neutrophil apoptosis is modulated by endothelial transmigration and adhesion molecule engagement. , 1997, Journal of immunology.

[12]  T. Takano,et al.  Sequential morphologic events during apoptosis of human neutrophils. Modulation by lipoxygenase-derived eicosanoids. , 1996, Journal of immunology.

[13]  H Suzuki,et al.  The changes of the stromal elastotic framework in the growth of peripheral lung adenocarcinomas , 1996, Cancer.

[14]  E. Moore,et al.  Interleukin‐6 suppression of neutrophil apoptosis is neutrophil concentration dependent , 1995, Journal of leukocyte biology.

[15]  P. Musiani,et al.  Role of cytokines in distribution and differentiation of dendritic cell/Langerhans' cell lineage in human primary carcinomas of the lung. , 1995, Human Pathology.

[16]  G. Stamp,et al.  The detection and localization of monocyte chemoattractant protein-1 (MCP-1) in human ovarian cancer. , 1995, The Journal of clinical investigation.

[17]  A. Verhoeven,et al.  Human neutrophils lose their surface Fc gamma RIII and acquire Annexin V binding sites during apoptosis in vitro. , 1995, Blood.

[18]  A. Mantovani,et al.  Tumor-associated macrophages in neoplastic progression: a paradigm for the in vivo function of chemokines. , 1994, Laboratory investigation; a journal of technical methods and pathology.

[19]  S. Barsky,et al.  Rising incidence of bronchioloalveolar lung carcinoma and its unique clinicopathologic features , 1994, Cancer.

[20]  C. Haslett,et al.  Inhibition of apoptosis and prolongation of neutrophil functional longevity by inflammatory mediators , 1993, Journal of leukocyte biology.

[21]  M. Pébusque,et al.  TNF alpha acts in synergy with GM-CSF to induce proliferation of acute myeloid leukemia cells by up-regulating the GM-CSF receptor and GM-CSF gene expression. , 1993, Leukemia.

[22]  F. Herrmann,et al.  Prolongation of survival of human polymorphonuclear neutrophils by granulocyte-macrophage colony-stimulating factor is caused by inhibition of programmed cell death. , 1992, Blood.

[23]  A. Kay,et al.  Expression and generation of interleukin-8, IL-6 and granulocyte-macrophage colony-stimulating factor by bronchial epithelial cells and enhancement by IL-1 beta and tumour necrosis factor-alpha. , 1992, Immunology.

[24]  A. Mantovani,et al.  Modulation of granulocyte survival and programmed cell death by cytokines and bacterial products. , 1992, Blood.

[25]  M. Matthay,et al.  Elevated levels of NAP-1/interleukin-8 are present in the airspaces of patients with the adult respiratory distress syndrome and are associated with increased mortality. , 1992, The American review of respiratory disease.

[26]  L. Garfinkel,et al.  The changing pattern of lung carcinoma , 1991, Cancer.

[27]  T. Standiford,et al.  Interleukin-8 gene expression by a pulmonary epithelial cell line. A model for cytokine networks in the lung. , 1990, The Journal of clinical investigation.

[28]  Alan D. Lopez,et al.  International Trends in Cancer Mortality in France, West Germany, Italy, Japan, England and Wales, and the United States a , 1990, Annals of the New York Academy of Sciences.

[29]  A. Lopez,et al.  International trends in cancer mortality in France, West Germany, Italy, Japan, England and Wales, and the USA , 1990, The Lancet.

[30]  J F Silverman,et al.  Morphometric and morphologic evaluations in stage III non‐small cell lung cancers. Prognostic significance of quantitative assessment of infiltrating lymphoid cells , 1989, Cancer.

[31]  K. Suemasu,et al.  Histopathologic prognostic factors in adenocarcinomas of the peripheral lung less than 2 CM in diameter , 1988, Cancer.

[32]  S. Piantadosi,et al.  Postoperative T1 N0 non-small cell lung cancer. Squamous versus nonsquamous recurrences. The Lung Cancer Study Group. , 1987, The Journal of thoracic and cardiovascular surgery.

[33]  S D Greenberg,et al.  Lung cancer heterogeneity: a blinded and randomized study of 100 consecutive cases. , 1985, Human pathology.

[34]  J. Mcclellan,et al.  Bronchiolo-alveolar cell carcinoma diagnosed by bronchoalveolar lavage. , 1983, Chest.

[35]  S. Latt,et al.  Stimulated human phagocytes produce cytogenetic changes in cultured mammalian cells. , 1983, The New England journal of medicine.

[36]  Y. Nishiwaki,et al.  Prognostic implications of fibrotic focus (scar) in small peripheral lung cancers , 1980, The American journal of surgical pathology.

[37]  Barry H. Smith,et al.  A continuous tumor‐cell line from a human lung carcinoma with properties of type II alveolar epithelial cells , 1976, International journal of cancer.

[38]  A. Flahault,et al.  Neutrophil alveolitis in bronchioloalveolar carcinoma: induction by tumor-derived interleukin-8 and relation to clinical outcome. , 1998, The American journal of pathology.

[39]  F. Luft,et al.  Interleukin-8 delays spontaneous and tumor necrosis factor-alpha-mediated apoptosis of human neutrophils. , 1998, Kidney international.

[40]  A. Gazdar,et al.  Cell culture methods for the establishment of the NCI series of lung cancer cell lines , 1996, Journal of cellular biochemistry. Supplement.

[41]  S. Hirohashi,et al.  Small adenocarcinoma of the lung. Histologic characteristics and prognosis. , 1995, Cancer.

[42]  C. Haslett,et al.  Phagocyte recognition of cells undergoing apoptosis. , 1993, Immunology today.

[43]  P. Barbini,et al.  Bronchiolo-alveolar carcinoma: an analysis of survival predictors. , 1992, European journal of cancer.