E2FBP1/DRIL1, an AT-rich interaction domain-family transcription factor, is regulated by p53.
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S. Ichwan | M. Ikeda | T. Suganuma | K. Araki | K. Ma | M. Tamamori‐Adachi | Kaiwen Ma | Tamaki Suganuma | Keigo Araki | M. Tamamori-Adachi
[1] G. Daley,et al. A functional screen identifies hDRIL1 as an oncogene that rescues RAS-induced senescence , 2002, Nature Cell Biology.
[2] Yusuke Nakamura,et al. Cyclin K as a direct transcriptional target of the p53 tumor suppressor. , 2002, Neoplasia.
[3] Y Taya,et al. p53DINP1, a p53-inducible gene, regulates p53-dependent apoptosis. , 2001, Molecular cell.
[4] R. Scheuermann,et al. Transcriptional Activation by a Matrix Associating Region-binding Protein , 2001, The Journal of Biological Chemistry.
[5] A. Levine,et al. Surfing the p53 network , 2000, Nature.
[6] P. Pandolfi,et al. Regulation of p53 activity in nuclear bodies by a specific PML isoform , 2000, The EMBO journal.
[7] Ailan Guo,et al. The function of PML in p53-dependent apoptosis , 2000, Nature Cell Biology.
[8] Yusuke Nakamura,et al. p53AIP1, a Potential Mediator of p53-Dependent Apoptosis, and Its Regulation by Ser-46-Phosphorylated p53 , 2000, Cell.
[9] P. Tucker,et al. Regulation of matrix attachment region‐dependent, lymphocyte‐restricted transcription through differential localization within promyelocytic leukemia nuclear bodies , 2000, The EMBO journal.
[10] R. Saint,et al. ARID proteins come in from the desert. , 2000, Trends in biochemical sciences.
[11] Yusuke Nakamura,et al. A ribonucleotide reductase gene involved in a p53-dependent cell-cycle checkpoint for DNA damage , 2000, Nature.
[12] Goberdhan P Dimri,et al. Regulation of a Senescence Checkpoint Response by the E2F1 Transcription Factor and p14ARF Tumor Suppressor , 2000, Molecular and Cellular Biology.
[13] J. Levine,et al. Surfing the p53 network , 2000, Nature.
[14] R. Saint,et al. The Drosophila dead ringer gene is required for early embryonic patterning through regulation of argos and buttonhead expression. , 1999, Development.
[15] A. Courey,et al. Dorsal-Mediated Repression Requires the Formation of a Multiprotein Repression Complex at the Ventral Silencer , 1998, Molecular and Cellular Biology.
[16] S. Sekiya,et al. A novel E2F binding protein with Myc-type HLH motif stimulates E2F-dependent transcription by forming a heterodimer , 1998, Oncogene.
[17] R. Saint,et al. The human dead ringer/bright homolog, DRIL1: cDNA cloning, gene structure, and mapping to D19S886, a marker on 19p13.3 that is strictly linked to the Peutz-Jeghers syndrome. , 1998, Genomics.
[18] C. Webb,et al. Expression of bright at two distinct stages of B lymphocyte development. , 1998, Journal of immunology.
[19] W. El-Deiry,et al. Regulation of p53 downstream genes. , 1998, Seminars in cancer biology.
[20] S. Lowe,et al. Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4a , 1997, Cell.
[21] J. Nevins,et al. Functional analysis of E2F transcription factor. , 1997, Methods in enzymology.
[22] K. Kinzler,et al. Genetic determinants of p53-induced apoptosis and growth arrest. , 1996, Genes & development.
[23] C. Prives,et al. p53: puzzle and paradigm. , 1996, Genes & development.
[24] J. Nevins,et al. A unique role for the Rb protein in controlling E2F accumulation during cell growth and differentiation. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[25] R. Saint,et al. Characterization of the dead ringer gene identifies a novel, highly conserved family of sequence-specific DNA-binding proteins , 1996, Molecular and cellular biology.
[26] Y. Kanegae,et al. Efficient generation of recombinant adenoviruses using adenovirus DNA-terminal protein complex and a cosmid bearing the full-length virus genome. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[27] R. Scheuermann,et al. The immunoglobulin heavy-chain matrix-associating regions are bound by Bright: a B cell-specific trans-activator that describes a new DNA-binding protein family. , 1995, Genes & development.
[28] J. Nevins,et al. Cellular targets for activation by the E2F1 transcription factor include DNA synthesis- and G1/S-regulatory genes , 1995, Molecular and cellular biology.
[29] David Beach,et al. p21 is a universal inhibitor of cyclin kinases , 1993, Nature.
[30] J. Trent,et al. WAF1, a potential mediator of p53 tumor suppression , 1993, Cell.
[31] S. Elledge,et al. The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases , 1993, Cell.