Sepsis-Induced Cardiac Mitochondrial Dysfunction Involves Altered Mitochondrial-Localization of Tyrosine Kinase Src and Tyrosine Phosphatase SHP2
暂无分享,去创建一个
[1] J. Minei,et al. Specific inhibition of mitochondrial oxidative stress suppresses inflammation and improves cardiac function in a rat pneumonia-related sepsis model. , 2012, American journal of physiology. Heart and circulatory physiology.
[2] C. Rocher,et al. Preservation of NADH ubiquinone-oxidoreductase activity by Src kinase-mediated phosphorylation of NDUFB10. , 2012, Biochimica et biophysica acta.
[3] M. Singer,et al. Organ Failure in the ICU: Cellular Alterations , 2011, Seminars in respiratory and critical care medicine.
[4] G. Dulan,et al. Regulation of VEGF-induced endothelial cell migration by mitochondrial reactive oxygen species. , 2011, American journal of physiology. Cell physiology.
[5] T. Letellier,et al. Functional impact of PTP1B-mediated Src regulation on oxidative phosphorylation in rat brain mitochondria , 2011, Cellular and Molecular Life Sciences.
[6] Xiangmei Zhou,et al. A role for mitochondria in NLRP3 inflammasome activation , 2011, Nature.
[7] F. Bozza,et al. Bioenergetic failure of human peripheral blood monocytes in patients with septic shock is mediated by reduced F1Fo adenosine-5′-triphosphate synthase activity* , 2011, Critical care medicine.
[8] P. Lyu,et al. Proteomics study of oxidative stress and Src kinase inhibition in H9C2 cardiomyocytes: A cell model of heart ischemia reperfusion injury and treatment , 2010 .
[9] C. Deutschman,et al. Mitochondrial dysfunction and resuscitation in sepsis. , 2010, Critical care clinics.
[10] M. Disatnik,et al. Mitochondrial import of PKCε is mediated by HSP90: a role in cardioprotection from ischaemia and reperfusion injury , 2010, Cardiovascular research.
[11] F. Nwariaku,et al. Burn serum causes a CD14-dependent mitochondrial damage in primary cardiomyocytes. , 2010, American journal of physiology. Heart and circulatory physiology.
[12] R. Apweiler,et al. Phosphoproteome Analysis Reveals Regulatory Sites in Major Pathways of Cardiac Mitochondria* , 2010, Molecular & Cellular Proteomics.
[13] D. Harrison,et al. Therapeutic targeting of mitochondrial superoxide in hypertension , 2010, Circulation research.
[14] D. Maass,et al. Age-dependent differences of interleukin-6 activity in cardiac function after burn complicated by sepsis. , 2010, Burns : journal of the International Society for Burn Injuries.
[15] E. Tajkhorshid,et al. Tyrosine phosphorylation by Src within the cavity of the adenine nucleotide translocase 1 regulates ADP/ATP exchange in mitochondria. , 2010, American journal of physiology. Cell physiology.
[16] Richard Beale,et al. The Surviving Sepsis Campaign: Results of an international guideline-based performance improvement program targeting severe sepsis* , 2010, Critical care medicine.
[17] Richard Beale,et al. The Surviving Sepsis Campaign: results of an international guideline-based performance improvement program targeting severe sepsis , 2010, Intensive Care Medicine.
[18] W. Junger,et al. Circulating Mitochondrial DAMPs Cause Inflammatory Responses to Injury , 2009, Nature.
[19] S. Hollenberg,et al. Cardiac dysfunction in severe sepsis and septic shock , 2009, Current opinion in critical care.
[20] J. Altschmied,et al. "Shping 2" different cellular localizations - a potential new player in aging processes , 2009, Aging.
[21] Jesús Blanco,et al. Incidence, organ dysfunction and mortality in severe sepsis: a Spanish multicentre study , 2008, Critical care.
[22] P. Ping,et al. Reactive Oxygen Species Production in Energized Cardiac Mitochondria During Hypoxia/Reoxygenation: Modulation by Nitric Oxide , 2008, Circulation research.
[23] H. Tsutsui,et al. Oxidative stress and mitochondrial DNA damage in heart failure. , 2008, Circulation journal : official journal of the Japanese Circulation Society.
[24] E. Gontier,et al. Localization of PTP-1B, SHP-2, and Src Exclusively in Rat Brain Mitochondria and Functional Consequences* , 2008, Journal of Biological Chemistry.
[25] A. Stringaro,et al. Src‐Tyrosine kinases are major agents in mitochondrial tyrosine phosphorylation , 2008, Journal of cellular biochemistry.
[26] G. Hunninghake,et al. Constitutive ERK MAPK Activity Regulates Macrophage ATP Production and Mitochondrial Integrity1 , 2008, The Journal of Immunology.
[27] A. Brunati,et al. Identification of new tyrosine phosphorylated proteins in rat brain mitochondria , 2008, FEBS letters.
[28] A. Brunati,et al. Identification of the flavoprotein of succinate dehydrogenase and aconitase as in vitro mitochondrial substrates of Fgr tyrosine kinase , 2007, FEBS letters.
[29] R. Gamelli,et al. American Burn Association Consensus Conference to Define Sepsis and Infection in Burns , 2007, Journal of burn care & research : official publication of the American Burn Association.
[30] R. Levy. MITOCHONDRIAL DYSFUNCTION, BIOENERGETIC IMPAIRMENT, AND METABOLIC DOWN-REGULATION IN SEPSIS , 2007, Shock.
[31] M. Singer,et al. Mechanisms of sepsis-induced cardiac dysfunction , 2007, Critical care medicine.
[32] D. Maass,et al. Cardiac mitochondrial damage and inflammation responses in sepsis. , 2007, Surgical infections.
[33] D. Angus,et al. Epidemiology of severe sepsis around the world. , 2006, Endocrine, metabolic & immune disorders drug targets.
[34] Elena Bisetto,et al. Differential steady‐state tyrosine phosphorylation of two oligomeric forms of mitochondrial F0F1ATPsynthase: A structural proteomic analysis , 2006, Proteomics.
[35] D. Maass,et al. MURINE IN VIVO MYOCARDIAL CONTRACTILE DYSFUNCTION AFTER BURN INJURY IS EXACERBATED BY PNEUMONIA SEPSIS , 2005, Shock.
[36] Zhijian J. Chen,et al. Identification and Characterization of MAVS, a Mitochondrial Antiviral Signaling Protein that Activates NF-κB and IRF3 , 2005, Cell.
[37] J. Mazat,et al. Identification of tyrosine-phosphorylated proteins of the mitochondrial oxidative phosphorylation machinery , 2005, Cellular and Molecular Life Sciences CMLS.
[38] S. Ballinger. Mitochondrial dysfunction in cardiovascular disease. , 2005, Free radical biology & medicine.
[39] Mark A Sussman,et al. Cardiomyocyte Apoptosis Triggered by RAFTK/pyk2 via Src Kinase Is Antagonized by Paxillin* , 2004, Journal of Biological Chemistry.
[40] E. Crouser. Mitochondrial dysfunction in septic shock and multiple organ dysfunction syndrome. , 2004, Mitochondrion.
[41] J. Medina,et al. Mitochondrial extracellular signal‐regulated kinases 1/2 (ERK1/2) are modulated during brain development , 2004, Journal of Neurochemistry.
[42] D. Maass,et al. Cardiac effects of burn injury complicated by aspiration pneumonia-induced sepsis. , 2003, American journal of physiology. Heart and circulatory physiology.
[43] A. Sabri,et al. Mechanisms of Protease-activated Receptor-4 Actions in Cardiomyocytes , 2003, The Journal of Biological Chemistry.
[44] Sakae Tanaka,et al. Regulation of cytochrome c oxidase activity by c-Src in osteoclasts , 2003, The Journal of cell biology.
[45] Anand Kumar,et al. Clinical review: Myocardial depression in sepsis and septic shock , 2002, Critical care.
[46] M. Fink. Bench-to-bedside review: Cytopathic hypoxia , 2002, Critical care.
[47] John Land,et al. Association between mitochondrial dysfunction and severity and outcome of septic shock , 2002, The Lancet.
[48] A. Brunati,et al. Characterization and location of Src-dependent tyrosine phosphorylation in rat brain mitochondria. , 2002, Biochimica et biophysica acta.
[49] S. Kudoh,et al. Growth hormone signalling and apoptosis in neonatal rat cardiomyocytes , 2001, Molecular and Cellular Biochemistry.
[50] E. Cadenas,et al. Mitochondrial respiratory chain-dependent generation of superoxide anion and its release into the intermembrane space. , 2001, The Biochemical journal.
[51] Y. Zou,et al. Both Gs and Gi Proteins Are Critically Involved in Isoproterenol-induced Cardiomyocyte Hypertrophy* , 1999, The Journal of Biological Chemistry.
[52] M. Pucéat,et al. Src Family Tyrosine Kinase Regulates Intracellular pH in Cardiomyocytes , 1998, The Journal of cell biology.
[53] D. White,et al. Aspiration pneumonia-induced sepsis increases cardiac dysfunction after burn trauma. , 1998, The Journal of surgical research.
[54] C. Cairns,et al. Evidence for early supply independent mitochondrial dysfunction in patients developing multiple organ failure after trauma. , 1997, The Journal of trauma.
[55] W. Knaus,et al. Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM Consensus Conference Committee. American College of Chest Physicians/Society of Critical Care Medicine. , 1992, Chest.
[56] G. Attardi,et al. Differential regulation of expression of the multiple ADP/ATP translocase genes in human cells. , 1991, The Journal of biological chemistry.
[57] F. Ognibene,et al. Septic shock in humans. Advances in the understanding of pathogenesis, cardiovascular dysfunction, and therapy. , 1990, Annals of internal medicine.
[58] N. Banchero,et al. Volume density and distribution of mitochondria in myocardial growth and hypertrophy. , 1987, Respiration physiology.
[59] A. Starkov. Measurement of mitochondrial ROS production. , 2010, Methods in molecular biology.
[60] L. Cong,et al. Protein kinase C- (cid:1) regulates the subcellular localization of Shc in H 2 O 2 -treated cardiomyocytes , 2010 .
[61] G. Supinski,et al. Diaphragm and cardiac mitochondrial creatine kinases are impaired in sepsis. , 2007, Journal of applied physiology.
[62] D. Maass,et al. Cardiac mitochondrial damage and loss of ROS defense after burn injury: the beneficial effects of antioxidant therapy. , 2007, Journal of applied physiology.
[63] Zhijian J. Chen,et al. Identification and characterization of MAVS, a mitochondrial antiviral signaling protein that activates NF-kappaB and IRF 3. , 2005, Cell.
[64] J. Kline,et al. Metabolic dysfunction and depletion of mitochondria in hearts of septic rats. , 2004, Journal of molecular and cellular cardiology.
[65] D. Brdiczka,et al. The Function of Complexes between the Outer Mitochondrial Membrane Pore (vdac) and the Adenine Nucleotide Translocase in Regulation of Energy Metabolism and Apoptosis , 2003 .
[66] M. Fink. Cytopathic Hypoxia: Mitochondrial Dysfunction as a Potential Mechanism Contributing to Organ Failure in Sepsis , 2002 .
[67] M. Fink. Cytopathic hypoxia. Mitochondrial dysfunction as mechanism contributing to organ dysfunction in sepsis. , 2001, Critical care clinics.