α2-Adrenoceptor signaling in cardiomyocytes of spontaneously hypertensive rats starts to impair already at early age.

[1]  E. V. Evdokimovskii,et al.  Synergism of myocardial β-adrenoceptor blockade and I1-imidazoline receptor-driven signaling: Kinase-phosphatase switching. , 2019, Biochemical and biophysical research communications.

[2]  A. Maltsev.  Agmatine modulates calcium handling in cardiomyocytes of hibernating ground squirrels through calcium-sensing receptor signaling. , 2018, Cellular signalling.

[3]  A. Natali,et al.  Physical Exercise and Regulation of Intracellular Calcium in Cardiomyocytes of Hypertensive Rats , 2018, Arquivos brasileiros de cardiologia.

[4]  Y. Shiferaw,et al.  T‐tubule remodeling and increased heterogeneity of calcium release during the progression to heart failure in intact rat ventricle , 2017, Physiological reports.

[5]  A. S. Averin,et al.  Sarcolemmal α2-adrenoceptors control protective cardiomyocyte-delimited sympathoadrenal response. , 2016, Journal of molecular and cellular cardiology.

[6]  A. Alekseev,et al.  Alpha-2 adrenoceptors and imidazoline receptors in cardiomyocytes mediate counterbalancing effect of agmatine on NO synthesis and intracellular calcium handling. , 2014, Journal of molecular and cellular cardiology.

[7]  J. Piletz,et al.  Agmatine: clinical applications after 100 years in translation. , 2013, Drug discovery today.

[8]  A. Sarandol,et al.  Increased plasma agmatine levels in patients with schizophrenia. , 2013, Journal of psychiatric research.

[9]  M. Nenov,et al.  Modulation of L-type Ca2+ currents and intracellular calcium by agmatine in rat cardiomyocytes , 2013, Biochemistry (Moscow) Supplement Series A: Membrane and Cell Biology.

[10]  E. V. Evdokimovskii,et al.  Regulation of potential-dependent L-type Ca2+ currents by agmatine. Imidazoline receptors in isolated cardiomyocytes , 2012, Biochemistry (Moscow) Supplement Series A: Membrane and Cell Biology.

[11]  Z. Massy,et al.  The onset of left ventricular diastolic dysfunction in SHR rats is not related to hypertrophy or hypertension. , 2012, American journal of physiology. Heart and circulatory physiology.

[12]  R. Cohen,et al.  Targeting the redox regulation of SERCA in vascular physiology and disease. , 2010, Current opinion in pharmacology.

[13]  J. Borlak,et al.  Impaired tissue clearance of verapamil in rat cardiac hypertrophy results in transcriptional repression of ion channels , 2010, Xenobiotica; the fate of foreign compounds in biological systems.

[14]  B. Tota,et al.  Phospholamban S-nitrosylation modulates Starling response in fish heart , 2009, Proceedings of the Royal Society B: Biological Sciences.

[15]  O. Krizanova,et al.  Effect of 7-nitroindazole on the expression of intracellular calcium channels in the kidney of spontaneously hypertensive rats. , 2009, General physiology and biophysics.

[16]  J. Stamler,et al.  Regulation of the cardiac muscle ryanodine receptor by O(2) tension and S-nitrosoglutathione. , 2008, Biochemistry.

[17]  J. Hare,et al.  Deficient ryanodine receptor S-nitrosylation increases sarcoplasmic reticulum calcium leak and arrhythmogenesis in cardiomyocytes , 2007, Proceedings of the National Academy of Sciences.

[18]  A. Halaris,et al.  Agmatine : metabolic pathway and spectrum of activity in brain. , 2007, CNS drugs.

[19]  J. Privratsky,et al.  Inhibition of sarco(endo)plasmic reticulum Ca2+-ATPase differentially regulates contractile function in cardiac myocytes from normotensive and spontaneously hypertensive rats , 2007, Cell Biochemistry and Biophysics.

[20]  B. Fiedler,et al.  Interference of antihypertrophic molecules and signaling pathways with the Ca2+-calcineurin-NFAT cascade in cardiac myocytes. , 2004, Cardiovascular research.

[21]  J. Balligand,et al.  Nitric Oxide and Cardiac Function: Ten Years After, and Continuing , 2003, Circulation research.

[22]  J. Balligand,et al.  Differential regulation of nitric oxide synthases and their allosteric regulators in heart and vessels of hypertensive rats. , 2003, Cardiovascular research.

[23]  Tadeusz Malinski,et al.  Dynamics of nitric oxide release in the cardiovascular system. , 2003, Acta biochimica Polonica.

[24]  L. Giovannelli,et al.  Guanosine 3′: 5′‐cyclic monophosphate‐dependent pathway alterations in ventricular cardiomyocytes of spontaneously hypertensive rats , 2001, British journal of pharmacology.

[25]  P. Dominiak,et al.  Biological significance of agmatine, an endogenous ligand at imidazoline binding sites , 2001, British journal of pharmacology.

[26]  I. Shapira,et al.  The Effect of Agmatine Administration on Ischemic-Reperfused Isolated Rat Heart , 2001, Journal of cardiovascular pharmacology and therapeutics.

[27]  M. Corretti,et al.  Cellular mechanisms of altered contractility in the hypertrophied heart: big hearts, big sparks. , 1999, Circulation Research.

[28]  A. Shah,et al.  Selective dysregulation of nitric oxide synthase type 3 in cardiac myocytes but not coronary microvascular endothelial cells of spontaneously hypertensive rat. , 1998, Cardiovascular research.

[29]  D. Reis,et al.  Agmatine: a novel neurotransmitter? , 1998, Advances in pharmacology.

[30]  A. Swislocki,et al.  Insulin Resistance and Hypertension: Glucose Intolerance, Hyperinsulinemia, and Elevated Free Fatty Acids in the Lean Spontaneously Hypertensive Rat , 1993, The American journal of the medical sciences.