Suppression of complement regulatory protein C1 inhibitor in vascular endothelial activation by inhibiting vascular cell adhesion molecule-1 action.

[1]  Dongxu Liu,et al.  N-Linked Glycosylation at Asn3 and the Positively Charged Residues within the Amino-Terminal Domain of the C1 Inhibitor Are Required for Interaction of the C1 Inhibitor with Salmonella enterica Serovar Typhimurium Lipopolysaccharide and Lipid A , 2005, Infection and Immunity.

[2]  Dongxu Liu,et al.  C1 inhibitor prevents Gram-negative bacterial lipopolysaccharide-induced vascular permeability. , 2005, Blood.

[3]  C. Herfarth,et al.  C1-inhibitor reduces hepatic leukocyte-endothelial interaction and the expression of VCAM-1 in LPS-induced sepsis in the rat. , 2004, Microvascular research.

[4]  R. Gaynor,et al.  IκB kinases: key regulators of the NF-κB pathway , 2004 .

[5]  S. Ziemer,et al.  Effects of C1 esterase inhibitor administration on intestinal functional capillary density, leukocyte adherence and mesenteric plasma extravasation during experimental endotoxemia , 2004, Intensive Care Medicine.

[6]  A. Davis,et al.  Complement Regulatory Protein C1 Inhibitor Binds to Selectins and Interferes with Endothelial-Leukocyte Adhesion 1 , 2003, The Journal of Immunology.

[7]  Dongxu Liu,et al.  C1 Inhibitor Prevents Endotoxin Shock Via a Direct Interaction with Lipopolysaccharide 1 2 , 2003, The Journal of Immunology.

[8]  S. Goldblum,et al.  Mechanisms of bacterial lipopolysaccharide-induced endothelial apoptosis. , 2003, American journal of physiology. Lung cellular and molecular physiology.

[9]  W. Aird.  The role of the endothelium in severe sepsis and multiple organ dysfunction syndrome. , 2003, Blood.

[10]  G. Horstick,et al.  C1‐Esterase‐Inhibitor Treatment at Early Reperfusion of Hemorrhagic Shock Reduces Mesentery Leukocyte Adhesion and Rolling , 2001, Microcirculation.

[11]  C. F. Bennett,et al.  A Constitutive Cytoprotective Pathway Protects Endothelial Cells from Lipopolysaccharide-induced Apoptosis* , 2001, The Journal of Biological Chemistry.

[12]  A. Aderem,et al.  Toll-like receptors in the induction of the innate immune response , 2000, Nature.

[13]  R. de Caterina,et al.  Estrogens and glucocorticoids inhibit endothelial vascular cell adhesion molecule-1 expression by different transcriptional mechanisms. , 2000, Circulation research.

[14]  M. Heger,et al.  In vivo microscopy reveals that complement inhibition by C1‐esterase inhibitor reduces ischemia/reperfusion injury in the liver , 2000, Transplant international : official journal of the European Society for Organ Transplantation.

[15]  C. Hack,et al.  C1-Esterase inhibitor: an anti-inflammatory agent and its potential use in the treatment of diseases other than hereditary angioedema. , 2000, Pharmacological reviews.

[16]  R. Colman,et al.  Biologic Activities of the Contact Factors In Vivo , 1999, Thrombosis and Haemostasis.

[17]  P. Ward,et al.  Protective effects of C5a blockade in sepsis , 1999, Nature Medicine.

[18]  W. Erl,et al.  Monocyte Arrest and Transmigration on Inflamed Endothelium in Shear Flow Is Inhibited by Adenovirus-Mediated Gene Transfer of IκB- , 1999 .

[19]  E. Martin,et al.  C1-esterase inhibitor and its effects on endotoxin-induced leukocyte adherence and plasma extravasation in postcapillary venules. , 1999, Surgery.

[20]  J. Liao,et al.  Differential regulation of endothelial cell adhesion molecule expression by nitric oxide donors and antioxidants , 1998, Journal of leukocyte biology.

[21]  J. Loscalzo,et al.  Endothelial cells in physiology and in the pathophysiology of vascular disorders. , 1998, Blood.

[22]  C. Cordon-Cardo,et al.  Lipopolysaccharide Induces Disseminated Endothelial Apoptosis Requiring Ceramide Generation , 1997, The Journal of experimental medicine.

[23]  R. Knuechel,et al.  Activated transcription factor nuclear factor-kappa B is present in the atherosclerotic lesion. , 1996, The Journal of clinical investigation.

[24]  K. Ohba,et al.  Role of nitric oxide and prostaglandins in lipopolysaccharide-induced increase in vascular permeability in mouse skin. , 1996, European journal of pharmacology.

[25]  P. Libby,et al.  Nitric oxide decreases cytokine-induced endothelial activation. Nitric oxide selectively reduces endothelial expression of adhesion molecules and proinflammatory cytokines. , 1995, The Journal of clinical investigation.

[26]  T. Su,et al.  A nuclear post-transcriptional event responsible for overproduction of argininosuccinate synthetase in a canavanine-resistant variant of a human epithelial cell line. , 1995, European journal of biochemistry.

[27]  X. Ding,et al.  Lipopolysaccharide (LPS)-binding protein and soluble CD14 function as accessory molecules for LPS-induced changes in endothelial barrier function, in vitro. , 1994, The Journal of clinical investigation.

[28]  X. Ding,et al.  Bacterial lipopolysaccharide induces actin reorganization, intercellular gap formation, and endothelial barrier dysfunction in pulmonary vascular endothelial cells: Concurrent F‐actin depolymerization and new actin synthesis , 1993, Journal of cellular physiology.

[29]  B. Masinovsky,et al.  IL-4 acts synergistically with IL-1 beta to promote lymphocyte adhesion to microvascular endothelium by induction of vascular cell adhesion molecule-1. , 1990, Journal of immunology.

[30]  T. Carlos,et al.  Vascular cell adhesion molecule-1 mediates lymphocyte adherence to cytokine-activated cultured human endothelial cells. , 1990, Blood.

[31]  R. Tizard,et al.  Direct expression cloning of vascular cell adhesion molecule 1, a cytokine-induced endothelial protein that binds to lymphocytes , 1989, Cell.

[32]  P. Brandtzaeg,et al.  Plasma endotoxin as a predictor of multiple organ failure and death in systemic meningococcal disease. , 1989, The Journal of infectious diseases.

[33]  Larry V. McIntire,et al.  Effect of flow on polymorphonuclear leukocyte/endothelial cell adhesion , 1987 .

[34]  R. Colman,et al.  Inactivation of factor XII active fragment in normal plasma. Predominant role of C-1-inhibitor. , 1984, The Journal of clinical investigation.

[35]  R. Colman,et al.  Contribution of plasma protease inhibitors to the inactivation of kallikrein in plasma. , 1982, The Journal of clinical investigation.

[36]  Robert B Sim,et al.  Interaction of 125I‐labelled complement subcomponents C r and C s with protease inhibitors in plasma , 1979, FEBS letters.

[37]  A. Karsan,et al.  Lipopolysaccharide signaling in endothelial cells , 2006, Laboratory Investigation.

[38]  C. Hack,et al.  Multiple organ failure in septic shock. , 1996, Current topics in microbiology and immunology.

[39]  A. Manning,et al.  Transcriptional regulation of endothelial cell adhesion molecules: a dominant role for NF-kappa B. , 1995, Agents and actions. Supplements.

[40]  R. Tukey,et al.  Quantitation of related gene products by nuclear run-on and northern blot analysis. , 1991, Methods in enzymology.