Effect of calcium hydroxide on proinflammatory cytokines and neuropeptides.

[1]  A. Akopian,et al.  Tumor necrosis factor α enhances the sensitivity of rat trigeminal neurons to capsaicin , 2008, Neuroscience.

[2]  J. Prpić,et al.  Tumor Necrosis Factor-Alpha in Peripical Tissue Exudates of Teeth with Apical Periodontitis , 2008, Mediators of Inflammation.

[3]  O. Zmener,et al.  An in vitro study of the pH of three calcium hydroxide dressing materials. , 2007, Dental traumatology : official publication of International Association for Dental Traumatology.

[4]  Wonkyung Yang,et al.  Effect of calcium hydroxide-treated Prevotella nigrescens on the gene expression of matrix metalloproteinase and its inhibitor in MG63 cells. , 2006, Journal of endodontics.

[5]  S. Kopp,et al.  Insufficient endogenous control of tumor necrosis factor-alpha contributes to temporomandibular joint pain and tissue destruction in rheumatoid arthritis. , 2006, The Journal of rheumatology.

[6]  I. Moro,et al.  Expression of inflammatory cytokine genesin vivo by human alveolar bone-derived polymorphonuclear leukocytes isolated from chronically inflamed sites of bone resorption , 1996, Calcified Tissue International.

[7]  H. Messer,et al.  An evidence-based analysis of the antibacterial effectiveness of intracanal medicaments. , 2004, Journal of endodontics.

[8]  Jin Jiang,et al.  Calcium hydroxide reduces lipopolysaccharide-stimulated osteoclast formation. , 2003, Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics.

[9]  P. Lamey,et al.  Quantitative analysis of substance P, neurokinin A and calcitonin gene-related peptide in gingival crevicular fluid associated with painful human teeth. , 2002, European journal of oral sciences.

[10]  K. Nagata,et al.  Involvement of substance P, mast cells, TNF-alpha and ICAM-1 in the infiltration of inflammatory cells in human periapical granulomas. , 2002, Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology.

[11]  H. Ari,et al.  Interleukin-1beta and tumour necrosis factor-alpha levels in periapical exudates. , 2002, International endodontic journal.

[12]  M. Franchi,et al.  Effect of calcium hydroxide form and placement on root dentine pH. , 2001, International endodontic journal.

[13]  T. Arai,et al.  Calcium concentration and pH of the periapical environment after applying calcium hydroxide into root canals in vitro. , 2001, Journal of endodontics.

[14]  A. Markham,et al.  Infliximab: a review of its use in the management of rheumatoid arthritis. , 2000, Drugs.

[15]  S. Kopp,et al.  Tumor necrosis factor-alpha in synovial fluid and plasma from patients with chronic connective tissue disease and its relation to temporomandibular joint pain. , 2000, Journal of oral and maxillofacial surgery : official journal of the American Association of Oral and Maxillofacial Surgeons.

[16]  H. Junger,et al.  Nociceptive and inflammatory effects of subcutaneous TNFα , 2000, Pain.

[17]  J. Siqueira,et al.  Mechanisms of antimicrobial activity of calcium hydroxide: a critical review. , 1999, International endodontic journal.

[18]  Donald Voet,et al.  Fundamentals of Biochemistry , 1999 .

[19]  N. Kawashima,et al.  Expression of bone-resorptive and regulatory cytokines in murine periapical inflammation. , 1999, Archives of oral biology.

[20]  B. Bresnihan,et al.  Interleukin-1 receptor antagonist. , 1998, Rheumatic diseases clinics of North America.

[21]  Tetsu Takahashi,et al.  Proinflammatory cytokines detectable in synovial fluids from patients with temporomandibular disorders. , 1998, Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics.

[22]  H. Shimauchi,et al.  Balance of interleukin-1 beta and interleukin-1 receptor antagonist in human periapical lesions. , 1998, Journal of endodontics.

[23]  C. Y. Wang,et al.  Bone-resorptive cytokine gene expression in periapical lesions in the rat. , 1997, Oral microbiology and immunology.

[24]  D. Carnes,et al.  Changes in pH at the dentin surface in roots obturated with calcium hydroxide pastes. , 1996, Journal of endodontics.

[25]  R. Walton,et al.  Anaerobic tissue-dissolving abilities of calcium hydroxide and sodium hypochlorite. , 1995, Journal of endodontics.

[26]  C. Y. Wang,et al.  Immunolocalization of bone-resorptive cytokines in rat pulp and periapical lesions following surgical pulp exposure. , 1995, Oral microbiology and immunology.

[27]  E. Kontakiotis,et al.  In vitro evaluation of the effectiveness of calcium hydroxide and paramonochlorophenol on anaerobic bacteria from the root canal. , 1993, Endodontics & dental traumatology.

[28]  H. Messer,et al.  pH changes in root dentin over a 4-week period following root canal dressing with calcium hydroxide. , 1993, Journal of endodontics.

[29]  J. Andreasen,et al.  In vitro solubility of human pulp tissue in calcium hydroxide and sodium hypochlorite. , 1992, Endodontics & dental traumatology.

[30]  C. E. Brown,et al.  The comparative antimicrobial effect of calcium hydroxide. , 1991, Oral surgery, oral medicine, and oral pathology.

[31]  T. Williams,et al.  Time‐dependent synergistic interactions between the vasodilator neuropeptide, calcitonin gene‐related peptide (CGRP) and mediators of inflammation , 1991, British journal of pharmacology.

[32]  S. Brain,et al.  Inflammatory edema induced by interactions between IL-1 and the neuropeptide calcitonin gene-related peptide. , 1991, Journal of immunology.

[33]  G. Sundqvist,et al.  The antimicrobial effect of calcium hydroxide as a short-term intracanal dressing. , 1991, International endodontic journal.

[34]  E. Rossomando,et al.  Tumor necrosis factor identified in periapical tissue exudates of teeth with apical periodontitis. , 1991, Journal of endodontics.

[35]  P. Foreman,et al.  Review of calcium hydroxide. , 1990, International endodontic journal.

[36]  P. Stashenko Role of immune cytokines in the pathogenesis of periapical lesions. , 1990, Endodontics & dental traumatology.

[37]  M. Byers,et al.  Effects of injury and inflammation on pulpal and periapical nerves. , 1990, Journal of endodontics.

[38]  B. Henderson,et al.  Inhibition by neuropeptides of interleukin‐1β‐induced, prostaglandin‐independent hyperalgesia , 1989, British journal of pharmacology.

[39]  M. Byers,et al.  Responses of nerve fibers to pulpal inflammation and periapical lesions in rat molars demonstrated by calcitonin gene-related peptide immunocytochemistry. , 1988, Journal of endodontics.

[40]  M. Byers,et al.  Inflammation of rat molar pulp and periodontium causes increased calcitonin gene‐related peptide and axonal sprouting , 1988, The Anatomical record.

[41]  M. Cvek,et al.  Effects of calcium hydroxide and sodium hypochlorite on the dissolution of necrotic porcine muscle tissue. , 1988, Journal of endodontics.

[42]  T. Williams,et al.  Inflammatory oedema induced by synergism between calcitonin gene‐related peptide (CGRP) and mediators of increased vascular permeability , 1985, British journal of pharmacology.

[43]  R. Claesson,et al.  The antibacterial effect of camphorated paramonochlorophenol, camphorated phenol and calcium hydroxide in the treatment of infected root canals. , 1985, Endodontics & dental traumatology.

[44]  H. Morris,et al.  Calcitonin gene-related peptide is a potent vasodilator , 1985, Nature.

[45]  G. Heithersay Replantation of avulsed teeth. A review. , 1975, Australian dental journal.

[46]  H. Stanley,et al.  THE EFFECTS OF SURGICAL EXPOSURES OF DENTAL PULPS IN GERM-FREE AND CONVENTIONAL LABORATORY RATS. , 1965, Oral surgery, oral medicine, and oral pathology.