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Glucotoxicity promotes aberrant activation and mislocalization of Ras-related C3 botulinum toxin substrate 1 [Rac1] and metabolic dysfunction in pancreatic islet β-cells: reversal of such metabolic defects by metformin
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A. Kowluru
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D. L. Hynds
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Anil Chekuri
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Sartaj S. Baidwan
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