Role for TGF-beta in cyclosporine-induced modulation of renal epithelial barrier function.
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T. McMorrow | M. Ryan | Gemma Feldman | Breda Kiely | G. Ryan | N. Martin
[1] D. Balkovetz. Opening Pandora's box in the tight junction. , 2007, Journal of the American Society of Nephrology : JASN.
[2] D. McKay,et al. Transforming growth factor-beta regulation of epithelial tight junction proteins enhances barrier function and blocks enterohemorrhagic Escherichia coli O157:H7-induced increased permeability. , 2005, The American journal of pathology.
[3] T. McMorrow,et al. Cyclosporine A-induced renal fibrosis: a role for epithelial-mesenchymal transition. , 2005, The American journal of pathology.
[4] J. Lipschutz,et al. Extracellular Signal-regulated Kinases 1/2 Control Claudin-2 Expression in Madin-Darby Canine Kidney Strain I and II Cells* , 2005, Journal of Biological Chemistry.
[5] H. Moses,et al. Activation of the Erk pathway is required for TGF-beta1-induced EMT in vitro. , 2004, Neoplasia.
[6] T. Tsuruo,et al. Transforming Growth Factor-β1 Upregulates the Tight Junction and P-glycoprotein of Brain Microvascular Endothelial Cells , 2004, Cellular and Molecular Neurobiology.
[7] C. Cheng,et al. Regulation of blood-testis barrier dynamics: an in vivo study , 2004, Journal of Cell Science.
[8] R. Harris,et al. Epidermal Growth Factor Receptor Activation Differentially Regulates Claudin Expression and Enhances Transepithelial Resistance in Madin-Darby Canine Kidney Cells* , 2004, Journal of Biological Chemistry.
[9] J. Matthews,et al. Bryostatin-1 enhances barrier function in T84 epithelia through PKC-dependent regulation of tight junction proteins. , 2003, American journal of physiology. Cell physiology.
[10] C. Cheng,et al. TGF-betas: their role in testicular function and Sertoli cell tight junction dynamics. , 2003, International journal of andrology.
[11] C. Cheng,et al. TGF-beta3 regulates the blood-testis barrier dynamics via the p38 mitogen activated protein (MAP) kinase pathway: an in vivo study. , 2003, Endocrinology.
[12] M. Ryan,et al. Modulation of renal epithelial barrier function by mitogen-activated protein kinases (MAPKs): mechanism of cyclosporine A-induced increase in transepithelial resistance. , 2003, Kidney international.
[13] J. Gauldie,et al. TGF-beta effects on epithelial ion transport and barrier: reduced Cl- secretion blocked by a p38 MAPK inhibitor. , 2002, American journal of physiology. Cell physiology.
[14] L. Ericson,et al. Transforming growth factor-β and epidermal growth factor synergistically stimulate epithelial to mesenchymal transition (EMT) through a MEK-dependent mechanism in primary cultured pig thyrocytes , 2002, Journal of Cell Science.
[15] K. Budde,et al. Cyclosporine A up-regulates the expression of TGF-beta1 and its receptors type I and type II in rat mesangial cells. , 2002, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[16] L. González-Mariscal,et al. Nuclear localization of the tight junction protein ZO-2 in epithelial cells. , 2002, Experimental cell research.
[17] W. Lui,et al. Transforming Growth Factor-β3 Perturbs the Inter-Sertoli Tight Junction Permeability Barrier in Vitro Possibly Mediated via Its Effects on Occludin, Zonula Occludens-1, and Claudin-11. , 2001, Endocrinology.
[18] S. Tsukita,et al. Conversion of Zonulae Occludentes from Tight to Leaky Strand Type by Introducing Claudin-2 into Madin-Darby Canine Kidney I Cells , 2001, The Journal of cell biology.
[19] K. Sharma,et al. Effect of anti-transforming growth factor-beta antibodies in cyclosporine-induced renal dysfunction. , 2001, Kidney international.
[20] P. Muriel,et al. Tight junction proteins ZO-1, ZO-2, and occludin along isolated renal tubules. , 2000, Kidney international.
[21] T. Inai,et al. Claudin-1 contributes to the epithelial barrier function in MDCK cells. , 1999, European journal of cell biology.
[22] C. Fiocchi,et al. Transforming growth factor‐β1 preserves epithelial barrier function: Identification of receptors, biochemical intermediates, and cytokine antagonists , 1999, Journal of cellular physiology.
[23] C. Hawkey,et al. Human colonic subepithelial myofibroblasts modulate transepithelial resistance and secretory response. , 1999, American journal of physiology. Cell physiology.
[24] K. Stelwagen,et al. An anti-inflammatory component derived from milk of hyperimmunised cows reduces tight junction permeability in vitro , 1998, Inflammation Research.
[25] V. Sharma,et al. In vivo expression of transforming growth factor-beta1 in humans: stimulation by cyclosporine. , 1998 .
[26] D. McKay,et al. Superantigen activation of immune cells evokes epithelial (T84) transport and barrier abnormalities via IFN-gamma and TNF alpha: inhibition of increased permeability, but not diminished secretory responses by TGF-beta2. , 1997, Journal of immunology.
[27] M. Suthanthiran,et al. In vivo hyperexpression of transforming growth factor-beta1 in mice: stimulation by cyclosporine. , 1997, Transplantation.
[28] M. Balda,et al. Functional dissociation of paracellular permeability and transepithelial electrical resistance and disruption of the apical- basolateral intramembrane diffusion barrier by expression of a mutant tight junction membrane protein , 1996, The Journal of cell biology.
[29] C. Marano,et al. Tissue remodeling during tumor necrosis factor-induced apoptosis in LLC-PK1 renal epithelial cells. , 1996, The American journal of physiology.
[30] G. Firestone,et al. Antagonistic Regulation of Tight Junction Dynamics by Glucocorticoids and Transforming Growth Factor- in Mouse Mammary Epithelial Cells (*) , 1996, The Journal of Biological Chemistry.
[31] S. Lewis,et al. Cell division does not increase transepithelial permeability of LLC-PK1 cell sheets. , 1995, Experimental cell research.
[32] L. M. Russo,et al. Long‐term effects of tumor necrosis factor on LLC‐PK1 transepithelial resistance , 1993, Journal of cellular physiology.
[33] D. Podolsky,et al. Cytokine modulation of intestinal epithelial cell restitution: central role of transforming growth factor beta. , 1993, Gastroenterology.
[34] M. Welsh,et al. Cadmium in vivo causes disruption of tight junction-associated microfilaments in rat Sertoli cells. , 1993, Biology of reproduction.
[35] A. Thomson,et al. Cyclosporine: toxicity, metabolism, and drug interactions--implications from animal studies. , 1985, Transplantation proceedings.
[36] S H MADIN,et al. Established Kidney Cell Lines of Normal Adult Bovine and Ovine Origin.∗ , 1958, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.
[37] T. Tsuruo,et al. Transforming growth factor-beta1 upregulates the tight junction and P-glycoprotein of brain microvascular endothelial cells. , 2004, Cellular and molecular neurobiology.
[38] C. Cheng,et al. Transforming Growth Factor-b 3 Perturbs the Inter-Sertoli Tight Junction Permeability Barrier in Vitro Possibly Mediated via Its Effects on Occludin , Zonula Occludens-1 , and Claudin-11 * , 2001 .
[39] W. Bennett,et al. Nephrotoxocity of immunosuppressive drugs , 1994 .
[40] B. Ryffel,et al. Light and electron microscopic changes in the kidney of Wistar rats following treatment with cyclosporine A. , 1987, Ultrastructural pathology.
[41] B. Ryffel,et al. Morphology of cyclosporine nephrotoxicity in the rat. , 1986, Clinical nephrology.