Characterization of the L-arginine–NO–cGMP pathway in spontaneously hypertensive rat platelets: the effects of pregnancy
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[1] L. Myatt. Review: Reactive oxygen and nitrogen species and functional adaptation of the placenta. , 2010, Placenta.
[2] P. Kaufmann,et al. Vasodilator factors in the systemic and local adaptations to pregnancy , 2009, Reproductive biology and endocrinology : RB&E.
[3] J. Garthwaite,et al. Mechanisms of Activity-dependent Plasticity in Cellular Nitric Oxide-cGMP Signaling* , 2009, The Journal of Biological Chemistry.
[4] G. Jackson,et al. Aspirin acetylates nitric oxide synthase type 3 in platelets thereby increasing its activity. , 2009, Cardiovascular research.
[5] S. Cuzzocrea,et al. Maternal adaptation in pregnant hypertensive rats: improvement of vascular and inflammatory variables and oxidative damage in the kidney. , 2009, American journal of hypertension.
[6] K. Naseem,et al. Globular adiponectin increases cGMP formation in blood platelets independently of nitric oxide , 2008, Journal of thrombosis and haemostasis : JTH.
[7] J. Zweier,et al. Activation of Hsp90-eNOS and increased NO generation attenuate respiration of hypoxia-treated endothelial cells. , 2008, American journal of physiology. Cell physiology.
[8] D. Schwartz,et al. A profound decrease in maternal arginine uptake provokes endothelial nitration in the pregnant rat. , 2008, American journal of physiology. Heart and circulatory physiology.
[9] G. Mann,et al. Chronic exercise reduces platelet activation in hypertension: upregulation of the l‐arginine‐nitric oxide pathway , 2008, Scandinavian journal of medicine & science in sports.
[10] E. Melloni,et al. Proteolytic degradation of nitric oxide synthase isoforms by calpain is modulated by the expression levels of HSP90 , 2007, The FEBS journal.
[11] G. Lip,et al. Hypertension: endothelial dysfunction, the prothrombotic state and antithrombotic therapy , 2007, Expert review of cardiovascular therapy.
[12] G. Mann,et al. Platelet nitric oxide synthesis in uremia and malnutrition: a role for L-arginine supplementation in vascular protection? , 2007, Cardiovascular research.
[13] C. Page,et al. Increased platelet aggregation in vivo in the Zucker Diabetic Fatty rat: differences from the streptozotocin diabetic rat , 2007, British journal of pharmacology.
[14] E. Ha,et al. Reduced L-arginine level and decreased placental eNOS activity in preeclampsia. , 2006, Placenta.
[15] J. Joles,et al. blood pressure in adult SHR by shifting the perinatal balance of NO and reactive oxygen species towards NO : the inverted Barker phenomenon , 2004 .
[16] P. Jurasz,et al. Platelet–leukocyte aggregation induced by PAR agonists: regulation by nitric oxide and matrix metalloproteinases , 2004, British journal of pharmacology.
[17] R. Soares de Moura,et al. CAPTOPRIL REVERSES THE REDUCED VASODILATOR RESPONSE TO BRADYKININ IN HYPERTENSIVE PREGNANT RATS , 2004, Clinical and experimental pharmacology & physiology.
[18] G. Mann,et al. Diminished L-arginine bioavailability in hypertension. , 2004, Clinical science.
[19] J. Ellory,et al. Inhibition of l‐arginine transport in platelets by asymmetric dimethylarginine and NG‐monomethyl‐l‐arginine: Effects of arterial hypertension , 2004, Clinical and experimental pharmacology & physiology.
[20] D. Merrill,et al. Enhanced expression of Ang-(1-7) during pregnancy. , 2004, Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas.
[21] M. C. Rueda,et al. Intrauterine Growth Restriction in Spontaneously Hypertensive Rats , 2004 .
[22] P. Baker,et al. An overview of endothelium-derived hyperpolarising factor (EDHF) in normal and compromised pregnancies. , 2003, European journal of obstetrics, gynecology, and reproductive biology.
[23] F. Hofmann,et al. Direct activation of PDE5 by cGMP , 2003, The Journal of cell biology.
[24] E. Coelho,et al. Pregnancy-associated increase in rat systemic arteries endothelial nitric oxide production diminishes vasoconstrictor but does not enhance vasodilator responses. , 2002, Life sciences.
[25] N. Eissa,et al. Inducible Nitric-oxide Synthase Is Regulated by the Proteasome Degradation Pathway* , 2001, The Journal of Biological Chemistry.
[26] R. Fischmeister,et al. Cyclic GMP regulation of the L‐type Ca2+ channel current in human atrial myocytes , 2001, The Journal of physiology.
[27] C. Stefanadis,et al. Therapeutic modification of the L-arginine-eNOS pathway in cardiovascular diseases. , 2001, Atherosclerosis.
[28] U. Walter,et al. KT5823 Inhibits cGMP-dependent Protein Kinase Activity in Vitro but Not in Intact Human Platelets and Rat Mesangial Cells* , 2000, The Journal of Biological Chemistry.
[29] A. Ferro,et al. beta(2)-adrenoceptors activate nitric oxide synthase in human platelets. , 2000, Circulation research.
[30] A. Bender,et al. Ubiquitination of Neuronal Nitric-oxide Synthase in Vitro and in Vivo * , 2000, The Journal of Biological Chemistry.
[31] D. Levy,et al. Association of blood pressure with fibrinolytic potential in the Framingham offspring population. , 2000, Circulation.
[32] R. Haslam,et al. Cyclic Nucleotides and Phosphodiesterases in Platelets , 1999, Thrombosis and Haemostasis.
[33] J. Bonnar,et al. Nitric oxide in the uteroplacental, fetoplacental, and peripheral circulations in preeclampsia. , 1999, Obstetrics and gynecology.
[34] C. Boyd,et al. Transporters for cationic amino acids in animal cells: discovery, structure, and function. , 1998, Physiological reviews.
[35] H. Morris,et al. Hypothalamic hypertensive factor: an inhibitor of nitric oxide synthase activity. , 1997, Hypertension.
[36] B. Morris,et al. Influence of family history on frequency of glucagon receptor Gly40Ser mutation in hypertensive subjects. , 1997, Hypertension.
[37] E. Coelho,et al. Nitric oxide blunts sympathetic response of pregnant normotensive and hypertensive rat arteries. , 1997, Hypertension.
[38] J. Loscalzo,et al. Nitric oxide released from activated platelets inhibits platelet recruitment. , 1997, The Journal of clinical investigation.
[39] L. Poston,et al. Bradykinin-mediated relaxation of isolated maternal resistance arteries in normal pregnancy and preeclampsia. , 1996, American journal of obstetrics and gynecology.
[40] B. Sibai,et al. Cardiology patient page. Chronic hypertension in pregnancy. , 2007, Circulation.
[41] S. Moncada,et al. The L-arginine-nitric oxide pathway. , 1993, The New England journal of medicine.
[42] T. Lüscher. Platelet-vessel wall interaction: role of nitric oxide, prostaglandins and endothelins. , 1993, Bailliere's clinical haematology.
[43] U. Walter,et al. The nitric oxide and cGMP signal transduction system: regulation and mechanism of action. , 1993, Biochimica et biophysica acta.
[44] S Moncada,et al. Regulation of Vascular Homeostasis by Nitric Oxide , 1993, Thrombosis and Haemostasis.
[45] D. Heistad,et al. Seminars in medicine of the Beth Israel Hospital, Boston. Platelet-endothelium interactions. , 1993 .
[46] C. Boyd,et al. Identification of a new transport system (y+L) in human erythrocytes that recognizes lysine and leucine with high affinity. , 1992, The Journal of physiology.
[47] S. Moncada,et al. An L-arginine/nitric oxide pathway present in human platelets regulates aggregation. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[48] B. Brüne,et al. Inhibition of platelet aggregation by carbon monoxide is mediated by activation of guanylate cyclase. , 1987, Molecular pharmacology.
[49] M. Paller. Mechanism of decreased pressor responsiveness to ANG II, NE, and vasopressin in pregnant rats. , 1984, The American journal of physiology.
[50] S. Moncada,et al. Nitric oxide and the vascular endothelium. , 2006, Handbook of experimental pharmacology.
[51] I. Johnson,et al. Mechanisms of endothelium-dependent relaxation in myometrial resistance vessels and their alteration in preeclampsia. , 1999, Hypertension in pregnancy.
[52] J. Aronson,et al. Identification of system y+L as the high-affinity transporter for l-arginine in human platelets: up-regulation of l-arginine influx in uraemia , 1999, Pflügers Archiv.