Peli1 facilitates TRIF-dependent Toll-like receptor signaling and proinflammatory cytokine production

[1]  George Kollias,et al.  Function of TRADD in tumor necrosis factor receptor 1 signaling and in TRIF-dependent inflammatory responses , 2008, Nature Immunology.

[2]  Zheng‐gang Liu,et al.  The function of TRADD in signaling through tumor necrosis factor receptor 1 and TRIF-dependent Toll-like receptors , 2008, Nature Immunology.

[3]  T. Mak,et al.  Beyond tumor necrosis factor receptor: TRADD signaling in toll-like receptors , 2008, Proceedings of the National Academy of Sciences.

[4]  Shao-Cong Sun,et al.  Regulation of IκB Kinase-related Kinases and Antiviral Responses by Tumor Suppressor CYLD* , 2008, Journal of Biological Chemistry.

[5]  Osamu Takeuchi,et al.  Sequential control of Toll-like receptor–dependent responses by IRAK1 and IRAK2 , 2008, Nature Immunology.

[6]  C. Libert,et al.  Tumor necrosis factor alpha mediates the lethal hepatotoxic effects of poly(I:C) in D-galactosamine-sensitized mice. , 2008, Cytokine.

[7]  S. Ghosh,et al.  Shared Principles in NF-κB Signaling , 2008, Cell.

[8]  E. Pietras,et al.  A Deubiquitinase That Regulates Type I Interferon Production , 2007, Science.

[9]  P. Moynagh,et al.  Kinase-active Interleukin-1 Receptor-associated Kinases Promote Polyubiquitination and Degradation of the Pellino Family , 2007, Journal of Biological Chemistry.

[10]  S. Gerondakis,et al.  Regulating B‐cell activation and survival in response to TLR signals , 2007, Immunology and cell biology.

[11]  Zhijian J. Chen,et al.  Ubiquitin-mediated activation of TAK1 and IKK , 2007, Oncogene.

[12]  S. Akira,et al.  Essential role of IRAK-4 protein and its kinase activity in Toll-like receptor–mediated immune responses but not in TCR signaling , 2007, The Journal of experimental medicine.

[13]  S. Janssens,et al.  Pellino Proteins: Novel Players in TLR and IL-1R Signalling , 2007, Journal of cellular and molecular medicine.

[14]  A. Bowie,et al.  The family of five: TIR-domain-containing adaptors in Toll-like receptor signalling , 2007, Nature Reviews Immunology.

[15]  S. Janssens,et al.  Pellino proteins are more than scaffold proteins in TLR/IL‐1R signalling: A role as novel RING E3–ubiquitin‐ligases , 2006, FEBS letters.

[16]  K. Ishii,et al.  Differential roles of MDA5 and RIG-I helicases in the recognition of RNA viruses , 2006, Nature.

[17]  Hong-Hsing Liu,et al.  The specific and essential role of MAVS in antiviral innate immune responses. , 2006, Immunity.

[18]  Shao-Cong Sun,et al.  Regulation of T cell development by the deubiquitinating enzyme CYLD , 2006, Nature Immunology.

[19]  A. Lanzavecchia,et al.  Toll‐like receptor stimulation as a third signal required for activation of human naive B cells , 2006, European journal of immunology.

[20]  S. Akira,et al.  Pathogen Recognition and Innate Immunity , 2006, Cell.

[21]  Ruslan Medzhitov,et al.  Control of B-cell responses by Toll-like receptors , 2005, Nature.

[22]  Katherine A. Fitzgerald,et al.  Rip1 Mediates the Trif-dependent Toll-like Receptor 3- and 4-induced NF-κB Activation but Does Not Contribute to Interferon Regulatory Factor 3 Activation* , 2005, Journal of Biological Chemistry.

[23]  M. Offermann,et al.  Apoptosis Induced by the Toll-Like Receptor Adaptor TRIF Is Dependent on Its Receptor Interacting Protein Homotypic Interaction Motif1 , 2005, The Journal of Immunology.

[24]  Shao-Cong Sun,et al.  Negative Regulation of JNK Signaling by the Tumor Suppressor CYLD* , 2004, Journal of Biological Chemistry.

[25]  J. Inoue,et al.  Cutting Edge: TNFR-Associated Factor (TRAF) 6 Is Essential for MyD88-Dependent Pathway but Not Toll/IL-1 Receptor Domain-Containing Adaptor-Inducing IFN-β (TRIF)-Dependent Pathway in TLR Signaling1 , 2004, Journal of Immunology.

[26]  Douglas R. McDonald,et al.  RIP Links TLR4 to Akt and Is Essential for Cell Survival in Response to LPS Stimulation , 2004, The Journal of experimental medicine.

[27]  F. Martinon,et al.  RIP1 is an essential mediator of Toll-like receptor 3–induced NF-κB activation , 2004, Nature Immunology.

[28]  Shizuo Akira,et al.  Toll/IL-1 Receptor Domain-Containing Adaptor Inducing IFN-β (TRIF) Associates with TNF Receptor-Associated Factor 6 and TANK-Binding Kinase 1, and Activates Two Distinct Transcription Factors, NF-κB and IFN-Regulatory Factor-3, in the Toll-Like Receptor Signaling 1 , 2003, The Journal of Immunology.

[29]  S. Akira,et al.  Role of Adaptor TRIF in the MyD88-Independent Toll-Like Receptor Signaling Pathway , 2003, Science.

[30]  Guo-Ping Zhou,et al.  Triggering the Interferon Antiviral Response Through an IKK-Related Pathway , 2003, Science.

[31]  B. Williams,et al.  Poly(dI·dC)-induced Toll-like Receptor 3 (TLR3)-mediated Activation of NFκB and MAP Kinase Is through an Interleukin-1 Receptor-associated Kinase (IRAK)-independent Pathway Employing the Signaling Components TLR3-TRAF6-TAK1-TAB2-PKR* , 2003, The Journal of Biological Chemistry.

[32]  T. Maniatis,et al.  IKKε and TBK1 are essential components of the IRF3 signaling pathway , 2003, Nature Immunology.

[33]  Zhengfan Jiang,et al.  Pellino 1 Is Required for Interleukin-1 (IL-1)-mediated Signaling through Its Interaction with the IL-1 Receptor-associated Kinase 4 (IRAK4)-IRAK-Tumor Necrosis Factor Receptor-associated Factor 6 (TRAF6) Complex* , 2003, The Journal of Biological Chemistry.

[34]  C. Sousa Faculty Opinions recommendation of TICAM-1, an adaptor molecule that participates in Toll-like receptor 3-mediated interferon-beta induction. , 2003 .

[35]  Sophie Janssens,et al.  Functional diversity and regulation of different interleukin-1 receptor-associated kinase (IRAK) family members. , 2003, Molecular cell.

[36]  S. Akira,et al.  Cutting Edge: A Novel Toll/IL-1 Receptor Domain-Containing Adapter That Preferentially Activates the IFN-β Promoter in the Toll-Like Receptor Signaling1 , 2002, The Journal of Immunology.

[37]  E. Harhaj,et al.  NF-κB-Inducing Kinase Regulates the Processing of NF-κB2 p100 , 2001 .

[38]  G. Xiao,et al.  The NF-κB Signaling Pathway Is Not Required for Fas Ligand Gene Induction but Mediates Protection from Activation-induced Cell Death* , 2000, The Journal of Biological Chemistry.

[39]  Karolina Palucka,et al.  Linking innate and adaptive immunity , 1999, Nature Medicine.

[40]  C. Nüsslein-Volhard,et al.  Oligomerisation of Tube and Pelle leads to nuclear localisation of Dorsal , 1999, Mechanisms of Development.

[41]  A. Krieg,et al.  CpG oligodeoxyribonucleotides rescue mature spleen B cells from spontaneous apoptosis and promote cell cycle entry. , 1998, Journal of immunology.

[42]  G. Kollias,et al.  Immune and inflammatory responses in TNF alpha-deficient mice: a critical requirement for TNF alpha in the formation of primary B cell follicles, follicular dendritic cell networks and germinal centers, and in the maturation of the humoral immune response , 1996, The Journal of experimental medicine.

[43]  T. Mak,et al.  Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection , 1993, Cell.

[44]  J. Farber,et al.  Identification of CRG-2. An interferon-inducible mRNA predicted to encode a murine monokine. , 1990, The Journal of biological chemistry.

[45]  J. Swanson,et al.  Macrophage colony-stimulating factor (rM-CSF) stimulates pinocytosis in bone marrow-derived macrophages , 1989, The Journal of experimental medicine.

[46]  W. Reutter,et al.  Galactosamine-induced sensitization to the lethal effects of endotoxin. , 1979, Proceedings of the National Academy of Sciences of the United States of America.

[47]  P. Cohen,et al.  The IRAK-catalysed activation of the E3 ligase function of Pellino isoforms induces the Lys63-linked polyubiquitination of IRAK1. , 2008, The Biochemical journal.

[48]  S. Akira,et al.  TLR signaling. , 2006, Current topics in microbiology and immunology.

[49]  S. Akira,et al.  TLR signalling and the function of dendritic cells. , 2005, Chemical immunology and allergy.

[50]  R. Medzhitov,et al.  Toll-like receptors: linking innate and adaptive immunity. , 2004, Microbes and infection.