A distinct subpopulation of leukemia initiating cells in acute precursor B lymphoblastic leukemia: quiescent phenotype and unique transcriptomic profile
暂无分享,去创建一个
J. McPherson | C. Tepper | N. Satake | N. Nitin | S. Yoshida | Ryan R Davis | B. McLaughlin | C. Duong | Yueju Li | Masami Ijiri | Kyoungmi Kim | Connie P. M. Duong | H. Konishi | Ryan R. Davis | Jonathan Van Dyke | L. Beckett | Alex Q. Lee | Jonathan E. Van Dyke
[1] Huihui Liu,et al. Lipid metabolism of cancer stem cells , 2022, Oncology letters.
[2] Gary D Bader,et al. The reactome pathway knowledgebase 2022 , 2021, Nucleic Acids Res..
[3] B. Beutler,et al. Emerging roles of spliceosome in cancer and immunity , 2021, Protein & Cell.
[4] S. Ogawa,et al. Chromatin-Spliceosome Mutations in Acute Myeloid Leukemia , 2021, Cancers.
[5] Fan Yang,et al. The regulation of protein translation and its implications for cancer , 2021, Signal Transduction and Targeted Therapy.
[6] Anushya Muruganujan,et al. The Gene Ontology resource: enriching a GOld mine , 2020, Nucleic Acids Res..
[7] Y. Dou,et al. ER associated degradation preserves hematopoietic stem cell quiescence and self-renewal by restricting mTOR activity. , 2020, Blood.
[8] E. Vanzyl,et al. The spliceosome inhibitors isoginkgetin and pladienolide B induce ATF3-dependent cell death , 2019, bioRxiv.
[9] Justin Taylor,et al. Mutations in spliceosome genes and therapeutic opportunities in myeloid malignancies , 2019, Genes, chromosomes & cancer.
[10] T. Rando,et al. Stem Cell Quiescence: Dynamism, Restraint, and Cellular Idling. , 2019, Cell stem cell.
[11] Y. Ho,et al. HDAC inhibitor suppresses proliferation and tumorigenicity of drug‐resistant chronic myeloid leukemia stem cells through regulation of hsa‐miR‐196a targeting BCR/ABL1 , 2018, Experimental cell research.
[12] S. Wingett,et al. FastQ Screen: A tool for multi-genome mapping and quality control. , 2018, F1000Research.
[13] R. Mancini,et al. Metabolic features of cancer stem cells: the emerging role of lipid metabolism , 2018, Oncogene.
[14] Matthew R. Gazzara,et al. Aberrant splicing in B-cell acute lymphoblastic leukemia , 2017, bioRxiv.
[15] C. Sette,et al. Alternative splicing and cell survival: from tissue homeostasis to disease , 2016, Cell Death and Differentiation.
[16] Yangqiu Li,et al. Heterogeneity of CD34 and CD38 expression in acute B lymphoblastic leukemia cells is reversible and not hierarchically organized , 2016, Journal of Hematology & Oncology.
[17] F. Sotgia,et al. Cancer stem cell metabolism , 2016, Breast Cancer Research.
[18] S. Minucci,et al. Inhibition of histone deacetylases in cancer therapy: lessons from leukaemia , 2016, British Journal of Cancer.
[19] A. Syme,et al. Phosphorylation of eIF2α Is a Translational Control Mechanism Regulating Muscle Stem Cell Quiescence and Self-Renewal. , 2016, Cell stem cell.
[20] Minoru Kanehisa,et al. KEGG as a reference resource for gene and protein annotation , 2015, Nucleic Acids Res..
[21] David Allman,et al. Convergence of Acquired Mutations and Alternative Splicing of CD19 Enables Resistance to CART-19 Immunotherapy. , 2015, Cancer discovery.
[22] Enric Llorens-Bobadilla,et al. Single-Cell Transcriptomics Reveals a Population of Dormant Neural Stem Cells that Become Activated upon Brain Injury. , 2015, Cell stem cell.
[23] M. Rieger,et al. Stem Cell Hierarchy and Clonal Evolution in Acute Lymphoblastic Leukemia , 2015, Stem cells international.
[24] J. Li,et al. A New Strategy to Target Acute Myeloid Leukemia Stem and Progenitor Cells Using Chidamide, a Histone Deacetylase Inhibitor. , 2015, Current cancer drug targets.
[25] C. Schürch,et al. Regulation of hematopoietic and leukemic stem cells by the immune system , 2014, Cell Death and Differentiation.
[26] W. Huber,et al. Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2 , 2014, Genome Biology.
[27] S. Morrison,et al. Cellular Differences in Protein Synthesis Regulate Tissue Homeostasis , 2014, Cell.
[28] W. Du,et al. Binding to WGR domain by salidroside activates PARP1 and protects hematopoietic stem cells from oxidative stress. , 2014, Antioxidants & redox signaling.
[29] Jeffrey A. Magee,et al. Haematopoietic stem cells require a highly regulated protein synthesis rate , 2014, Nature.
[30] M. Olivotto,et al. Hypoxia-resistant profile implies vulnerability of cancer stem cells to physiological agents, which suggests new therapeutic targets , 2013, Cell cycle.
[31] M. Teitell,et al. Techniques to monitor glycolysis. , 2014, Methods in Enzymology.
[32] J. Haybaeck,et al. Eukaryotic translation initiation factors in cancer development and progression. , 2013, Cancer letters.
[33] John M. Ashton,et al. BCL-2 inhibition targets oxidative phosphorylation and selectively eradicates quiescent human leukemia stem cells. , 2013, Cell stem cell.
[34] Thomas R. Gingeras,et al. STAR: ultrafast universal RNA-seq aligner , 2013, Bioinform..
[35] W. Du,et al. Salidroside stimulates DNA repair enzyme Parp-1 activity in mouse HSC maintenance. , 2012, Blood.
[36] H. Coller,et al. Staying alive , 2012, Cell cycle.
[37] H. Boswell,et al. Novel combination treatments targeting chronic myeloid leukemia stem cells. , 2012, Clinical lymphoma, myeloma & leukemia.
[38] Stephen L. Abrams,et al. Targeting the translational apparatus to improve leukemia therapy: roles of the PI3K/PTEN/Akt/mTOR pathway , 2011, Leukemia.
[39] D. Charron,et al. Age‐related changes in human hematopoietic stem/progenitor cells , 2011, Aging cell.
[40] M. Assanah,et al. HnRNP proteins controlled by c-Myc deregulate pyruvate kinase mRNA splicing in cancer , 2010, Nature.
[41] I. Sánchez-García,et al. B‐cell acute lymphoblastic leukaemia: towards understanding its cellular origin , 2009, BioEssays : news and reviews in molecular, cellular and developmental biology.
[42] Timothy J. Muldoon,et al. Molecular imaging of glucose uptake in oral neoplasia following topical application of fluorescently labeled deoxy‐glucose , 2009, International journal of cancer.
[43] Jing Chen,et al. ToppGene Suite for gene list enrichment analysis and candidate gene prioritization , 2009, Nucleic Acids Res..
[44] W. Evans,et al. Inhibition of glycolysis modulates prednisolone resistance in acute lymphoblastic leukemia cells. , 2009, Blood.
[45] Y. Liu,et al. p53 regulates hematopoietic stem cell quiescence. , 2009, Cell stem cell.
[46] Scott A Armstrong,et al. Leukemia stem cells and human acute lymphoblastic leukemia. , 2009, Seminars in hematology.
[47] M. Baker. Cancer stem cells, becoming common , 2008 .
[48] Rob Pieters,et al. In childhood acute lymphoblastic leukemia, blasts at different stages of immunophenotypic maturation have stem cell properties. , 2008, Cancer cell.
[49] M. Loh,et al. Risk- and response-based classification of childhood B-precursor acute lymphoblastic leukemia: a combined analysis of prognostic markers from the Pediatric Oncology Group (POG) and Children's Cancer Group (CCG). , 2007, Blood.
[50] Chi V Dang,et al. Cancer's molecular sweet tooth and the Warburg effect. , 2006, Cancer research.
[51] Martin J Firth,et al. Altered glucose metabolism in childhood pre-B acute lymphoblastic leukaemia , 2006, Leukemia.
[52] Pablo Tamayo,et al. Gene set enrichment analysis: A knowledge-based approach for interpreting genome-wide expression profiles , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[53] Dagmar Ringe,et al. “Sleeping Beauty”: Quiescence in Saccharomyces cerevisiae , 2004, Microbiology and Molecular Biology Reviews.
[54] M. Trigg. Hematopoietic stem cells. , 2004, Pediatrics.
[55] Ioannis S Vizirianakis,et al. Mechanisms involved in the induced differentiation of leukemia cells. , 2003, Pharmacology & therapeutics.
[56] M. D. Boer,et al. Patient stratification based on prednisolone-vincristine-asparaginase resistance profiles in children with acute lymphoblastic leukemia. , 2003, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[57] T. Flores,et al. A primitive hematopoietic cell is the target for the leukemic transformation in human philadelphia-positive acute lymphoblastic leukemia. , 2000, Blood.
[58] K. Moore,et al. Single adult human CD34(+)/Lin-/CD38(-) progenitors give rise to natural killer cells, B-lineage cells, dendritic cells, and myeloid cells. , 1999, Blood.
[59] Ph,et al. Primordial role of CD34+38− cells in early and late trilineage haemopoietic engraftment after autologous blood cell transplantation , 1998, British journal of haematology.
[60] R. Pieters,et al. Prednisolone resistance in childhood acute lymphoblastic leukemia: vitro-vivo correlations and cross-resistance to other drugs. , 1998, Blood.
[61] L. Ailles,et al. Detection and characterization of primitive malignant and normal progenitors in patients with acute myelogenous leukemia using long-term coculture with supportive feeder layers and cytokines. , 1997, Blood.
[62] J. Dick,et al. Human acute myeloid leukemia is organized as a hierarchy that originates from a primitive hematopoietic cell , 1997, Nature Medicine.
[63] T. Hongo,et al. In vitro drug sensitivity testing can predict induction failure and early relapse of childhood acute lymphoblastic leukemia. , 1997, Blood.
[64] M. Caligiuri,et al. A cell initiating human acute myeloid leukaemia after transplantation into SCID mice , 1994, Nature.
[65] C. Jordan,et al. Primitive hemopoietic stem cells: direct assay of most productive populations by competitive repopulation with simple binomial, correlation and covariance calculations. , 1993, Experimental hematology.
[66] A. Eaves,et al. Rapid decline of chronic myeloid leukemic cells in long-term culture due to a defect at the leukemic stem cell level. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[67] C. Eaves,et al. Quantitative assay for totipotent reconstituting hematopoietic stem cells by a competitive repopulation strategy. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[68] D. Harrison. Competitive repopulation: a new assay for long-term stem cell functional capacity. , 1980, Blood.
[69] M. Minden,et al. Self-renewal in culture of proliferative blast progenitor cells in acute myeloblastic leukemia. , 1979, Blood.