A high Mr factor in human blood which confers serum resistance on gonococci: some properties and synergism with CMP-NANA.
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C. Constantinidou | J. Cole | H. Smith | N. Parsons | C. Sammons | D. Beadle | X. H. Zhou
[1] J. Cole,et al. The serum resistance of gonococci in the majority of urethral exudates is due to sialylated lipopolysaccharide seen as a surface coat , 1992 .
[2] J. Cole,et al. The surface structure seen on gonococci after treatment with CMP-NANA is due to sialylation of surface lipopolysaccharide previously described as a 'capsule'. , 1991, Microbial pathogenesis.
[3] M. Wilson,et al. Modification by sialic acid of Neisseria gonorrhoeae lipooligosaccharide epitope expression in human urethral exudates: an immunoelectron microscopic analysis. , 1990, The Journal of infectious diseases.
[4] J. Cole,et al. Resistance to human serum of gonococci in urethral exudates is reduced by neuraminidase , 1990, Proceedings of the Royal Society of London. Series B: Biological Sciences.
[5] J. Griffiss,et al. In vitro and in vivo modification of Neisseria gonorrhoeae lipooligosaccharide epitope structure by sialylation , 1990, The Journal of experimental medicine.
[6] J. Cole,et al. A surface polysaccharide forms when gonococci are converted to serum resistance by cytidine 5'-monophospho-N-acetyl neuraminic acid. , 1990, FEMS microbiology letters.
[7] J. R. Andrade,et al. Sialylation of lipopolysaccharide and loss of absorption of bactericidal antibody during conversion of gonococci to serum resistance by cytidine 5'-monophospho-N-acetyl neuraminic acid. , 1989, Microbial pathogenesis.
[8] J. Cole,et al. Cytidine 5'-monophospho-N-acetylneuraminic acid or a related compound is the low Mr factor from human red blood cells which induces gonococcal resistance to killing by human serum. , 1988, Journal of general microbiology.
[9] G. Larson,et al. Purification and characterization of a CMP-sialic:LcOse4Cer sialyltransferase from human colorectal carcinoma cell membranes. , 1988, Biochemistry.
[10] J. Cole,et al. Cytidine 5'-monophospho-N-acetyl neuraminic acid and a low molecular weight factor from human blood cells induce lipopolysaccharide alteration in gonococci when conferring resistance to killing by human serum. , 1988, Microbial pathogenesis.
[11] J. Cole,et al. White blood cells including polymorphonuclear phagocytes contain a factor which induces gonococcal resistance to complement‐mediated serum killing , 1988 .
[12] H. Smith,et al. Lipopolysaccharide alteration is associated with induced resistance of Neisseria gonorrhoeae to killing by human serum. , 1986, Journal of general microbiology.
[13] H. Smith,et al. Fractionation of guinea pig serum for an inducer of gonococcal resistance to killing by human serum: active fractions containing glucopeptides similar to those from human red blood cells. , 1984, Journal of general microbiology.
[14] H. Smith,et al. Red blood cells, a source of factors which induce Neisseria gonorrhoeae to resistance to complement-mediated killing by human serum. , 1984, Journal of general microbiology.
[15] C. Penn,et al. Factors affecting the induction of phenotypically determined serum resistance of Neisseria gonorrhoeae grown in media containing serum or its diffusible components. , 1981, Journal of general microbiology.
[16] C. Penn,et al. Selection from gonococci grown in vitro of a colony type with some virulence properties of organisms adapted in vivo. , 1977, Journal of general microbiology.
[17] C. Penn,et al. Morphological, biological and antigenic properties of Neisseria gonorrhoeae adapted to growth in guinea-pig subcutaneous chambers. , 1976, Journal of general microbiology.
[18] K. Witt,et al. DIFFERENTIAL ABILITY OF COLONIAL TYPES OF NEISSERIA GONORRHOEAE TO PRODUCE INFECTION AND AN INFLAMMATORY RESPONSE IN SUBCUTANEOUS PERFORATED PLASTIC CHAMBERS IN GUINEA-PIGS AND RABBITS , 1975 .
[19] M. Ward,et al. Gonococci in Urethral Exudates possess a Virulence Factor lost on Subculture , 1970, Nature.