Respiratory infections and asthma.
暂无分享,去创建一个
[1] W. Jusko,et al. Steroid dose sparing: pharmacodynamic responses to single versus divided doses of methylprednisolone in man. , 1990, The Journal of allergy and clinical immunology.
[2] A. Kagey‐Sobotka,et al. Cutaneous late-phase response to allergen. Mediator release and inflammatory cell infiltration. , 1989, The Journal of clinical investigation.
[3] I. Tager. Epidemiology of Respiratory Infections in the Development of Airway Hyperreactivity , 1990 .
[4] S. Baron,et al. Role of interferon in leukocyte histamine release caused by common respiratory viruses. , 1988, The Journal of infectious diseases.
[5] D. McDonald. Respiratory tract infections increase susceptibility to neurogenic inflammation in the rat trachea. , 1988, The American review of respiratory disease.
[6] P. Ogra,et al. The appearance of cell-bound IgE in respiratory-tract epithelium after respiratory-syncytial-virus infection. , 1980, The New England journal of medicine.
[7] N. Flavahan,et al. The Respiratory Epithelium Releases a Smooth Muscle Relaxing Factor , 1985 .
[8] A. Notkins,et al. Enhancement of IgE-mediated histamine release from human basophils by viruses: role of interferon , 1977, The Journal of experimental medicine.
[9] F. Sánchez‐Madrid,et al. The functional significance, distribution, and structure of LFA-1, LFA-2, and LFA-3: cell surface antigens associated with CTL-target interactions. , 1983, Journal of immunology.
[10] J. D. Capra,et al. Monoclonal antibodies to immunoglobulin G4 induce histamine release from human basophils in vitro. , 1982, The Journal of allergy and clinical immunology.
[11] D. Hahn,et al. Association of Chlamydia pneumoniae (strain TWAR) infection with wheezing, asthmatic bronchitis, and adult-onset asthma. , 1991, JAMA.
[12] P. Barnes. ASTHMA AS AN AXON REFLEX , 1986, The Lancet.
[13] A. Mcclelland,et al. The major human rhinovirus receptor is ICAM-1 , 1989, Cell.
[14] W. Busse. Decreased granulocyte response to isoproterenol in asthma during upper respiratory infections. , 2015, The American review of respiratory disease.
[15] W. Busse,et al. Effect of influenza A virus on leukocyte histamine release. , 1983, The Journal of allergy and clinical immunology.
[16] D. Littman,et al. Viral receptors of the immunoglobulin superfamily , 1989, Cell.
[17] P. Ogra,et al. Predictive value of respiratory syncytial virus-specific IgE responses for recurrent wheezing following bronchiolitis. , 1986, The Journal of pediatrics.
[18] K. Mcintosh,et al. The association of viral and bacterial respiratory infections with exacerbations of wheezing in young asthmatic children+ , 1973, The Journal of Pediatrics.
[19] D. Wong,et al. Parainfluenza virus bronchiolitis. Epidemiology and pathogenesis. , 1986, American journal of diseases of children.
[20] B. Bierer,et al. T cell adhesion molecules , 1988, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[21] J. Nadel,et al. Mechanisms of bronchial hyperreactivity in normal subjects after upper respiratory tract infection. , 1976, The American review of respiratory disease.
[22] J. Nadel,et al. Influenza infection causes airway hyperresponsiveness by decreasing enkephalinase. , 1988, Journal of applied physiology.
[23] D. Wong,et al. The development of respiratory syncytial virus-specific IgE and the release of histamine in nasopharyngeal secretions after infection. , 1981, The New England journal of medicine.
[24] J. Nadel,et al. Virus induces airway hyperresponsiveness to tachykinins: role of neutral endopeptidase. , 1989, Journal of applied physiology.
[25] J. Hooks,et al. Enhancement of basophil chemotaxis in vitro by virus-induced interferon. , 1981, The Journal of clinical investigation.
[26] R. Slavin,et al. Sinusitis and bronchial asthma. , 1980, The Journal of allergy and clinical immunology.
[27] H. Gruber,et al. Ribavirin inhibits mast cell mediator release. , 1987, The Journal of pharmacology and experimental therapeutics.
[28] W. Liu,et al. Immunomodulation of influenza virus infection in the precipitating asthma attack. , 1988, Chest.
[29] W. Busse,et al. Parainfluenza 3 infection blocks the ability of a beta adrenergic receptor agonist to inhibit antigen-induced contraction of guinea pig isolated airway smooth muscle. , 1981, The Journal of clinical investigation.
[30] B. Ames,et al. Oxygen radicals and human disease. , 1987, Annals of internal medicine.
[31] E. Walsh,et al. Aerosolized ribavirin treatment of infants with respiratory syncytial viral infection. A randomized double-blind study. , 1983, The New England journal of medicine.
[32] V. Knight,et al. Selective inhibition of functional lymphocyte subpopulations by ribavirin , 1982, Antimicrobial Agents and Chemotherapy.
[33] A. Szentivanyi. The beta adrenergic theory of the atopic abnormality in bronchial asthma , 1968 .
[34] R. Rothlein,et al. Intercellular adhesion molecule-1 (ICAM-1) in the pathogenesis of asthma. , 1990, Science.
[35] W. Busse,et al. In vitro incubation with influenza virus primes human polymorphonuclear leukocyte generation of superoxide. , 1991, American journal of respiratory cell and molecular biology.
[36] A. Fryer,et al. Parainfluenza virus type 1 reduces the affinity of agonists for muscarinic receptors in guinea-pig lung and heart. , 1990, European journal of pharmacology.
[37] P. Vanhoutte. Epithelium-derived relaxing factor(s) and bronchial reactivity. , 1989, The Journal of allergy and clinical immunology.
[38] G. Rachelefsky,et al. Chronic sinus disease with associated reactive airway disease in children. , 1984, Pediatrics.
[39] D. Staunton,et al. A cell adhesion molecule, ICAM-1, is the major surface receptor for rhinoviruses , 1989, Cell.
[40] E. Bleecker,et al. Immediate and late inflammatory responses to ragweed antigen challenge of the peripheral airways in allergic asthmatics. Cellular, mediator, and permeability changes. , 1991, The American review of respiratory disease.
[41] P. Suratt,et al. Exacerbations of asthma in adults during experimental rhinovirus infection. , 1985, The American review of respiratory disease.
[42] B. Volovitz,et al. The Release of Leukotrienes in the Respiratory Tract during Infection with Respiratory Syncytial Virus: Role in Obstructive Airway Disease , 1988, Pediatric Research.
[43] C. Reed,et al. Viruses as precipitants of asthmatic attacks in children. , 1974, JAMA.
[44] E. Goetzl,et al. Predominant generation of 15-lipoxygenase metabolites of arachidonic acid by epithelial cells from human trachea. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[45] P. Edelson,et al. Effect of ribavirin therapy on respiratory syncytial virus-specific IgE and IgA responses after infection. , 1987, The Journal of infectious diseases.
[46] D. Staunton,et al. A soluble form of intercellular adhesion molecule-1 inhibits rhinovirus infection , 1990, Nature.
[47] E. Dick,et al. Enhancement by parainfluenza 3 infection of contractile responses to substance P and capsaicin in airway smooth muscle from the guinea pig. , 1987, The American review of respiratory disease.
[48] P. Barnes. Neuropeptides in the lung: localization, function, and pathophysiologic implications. , 1987, The Journal of allergy and clinical immunology.
[49] Elliot,et al. Rhinovirus upper respiratory infection increases airway hyperreactivity and late asthmatic reactions. , 1989, The Journal of clinical investigation.
[50] A. Togias,et al. Inflammatory mediators in late antigen-induced rhinitis. , 1985, The New England journal of medicine.