T and B cells participate in bone repair by infiltrating the fracture callus in a two-wave fashion.
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Georg N Duda | Andreas Radbruch | Hanna Schell | Thaqif El Khassawna | Hans-Dieter Volk | G. Duda | Andreas Radbruch | H. Volk | A. Serra | K. Schmidt-Bleek | A. Hauser | C. Schlundt | T. El Khassawna | H. Schell | A. Ellinghaus | Ireen Könnecke | Alessandro Serra | Claudia Schlundt | Anja Hauser | Agnes Ellinghaus | Katharina Schmidt-Bleek | I. Könnecke
[1] J. Efron,et al. Wound healing and T-lymphocytes. , 1990, The Journal of surgical research.
[2] H. Takayanagi,et al. Osteoclasts and the immune system , 2009, Journal of Bone and Mineral Metabolism.
[3] Josef M. Penninger,et al. Activated T cells regulate bone loss and joint destruction in adjuvant arthritis through osteoprotegerin ligand , 1999, Nature.
[4] O. Ljunggren,et al. Inflammatory cytokines regulate proliferation of cultured human osteoblasts. , 1997, Acta orthopaedica Scandinavica.
[5] Kenneth G. C. Smith,et al. Competence and competition: the challenge of becoming a long-lived plasma cell , 2006, Nature Reviews Immunology.
[6] Youngkyun Lee. The role of interleukin-17 in bone metabolism and inflammatory skeletal diseases , 2013, BMB reports.
[7] D. Hu,et al. Rejuvenation of the inflammatory system stimulates fracture repair in aged mice , 2010, Journal of orthopaedic research : official publication of the Orthopaedic Research Society.
[8] H. Takayanagi. Osteoimmunology: shared mechanisms and crosstalk between the immune and bone systems , 2007, Nature Reviews Immunology.
[9] S. Shi,et al. Mesenchymal Stem Cell-Based Tissue Regeneration is Governed by Recipient T Lymphocyte via IFN-γ and TNF-α , 2011, Nature Medicine.
[10] G. Duda,et al. Immunologically restricted patients exhibit a pronounced inflammation and inadequate response to hypoxia in fracture hematomas , 2011, Immunologic research.
[11] Y. Kadono,et al. Osteoimmunology: interplay between the immune system and bone metabolism. , 2006, Annual review of immunology.
[12] R. Pabst,et al. The bone marrow: a nest for migratory memory T cells. , 2005, Trends in immunology.
[13] G. Duda,et al. Human Early Fracture Hematoma Is Characterized by Inflammation and Hypoxia , 2011, Clinical orthopaedics and related research.
[14] G. Stark,et al. Characterization of β-R1, a Gene That Is Selectively Induced by Interferon β (IFN-β) Compared with IFN-α* , 1996, The Journal of Biological Chemistry.
[15] N. Verma,et al. Immunoregulation of bone remodelling , 2012, International journal of critical illness and injury science.
[16] G. Duda,et al. Initiation and early control of tissue regeneration – bone healing as a model system for tissue regeneration , 2014, Expert opinion on biological therapy.
[17] P. Giannoudis,et al. The role of inhibitory molecules in fracture healing. , 2006, Injury.
[18] S. Nourshargh,et al. Venular basement membranes ubiquitously express matrix protein low-expression regions: characterization in multiple tissues and remodeling during inflammation. , 2010, The American journal of pathology.
[19] Temple F. Smith,et al. Transcriptional Analysis of Fracture Healing and the Induction of Embryonic Stem Cell–Related Genes , 2009, PloS one.
[20] Antonios G Mikos,et al. Harnessing and modulating inflammation in strategies for bone regeneration. , 2011, Tissue engineering. Part B, Reviews.
[21] B. Alman,et al. T-Lymphocytes Enable Osteoblast Maturation via IL-17F during the Early Phase of Fracture Repair , 2012, PloS one.
[22] T. Einhorn,et al. Production of a standard closed fracture in laboratory animal bone , 1984, Journal of orthopaedic research : official publication of the Orthopaedic Research Society.
[23] W. Olszewski,et al. The healing of tibial fracture and response of the local lymphatic system. , 2007, The Journal of trauma.
[24] J. Strominger,et al. Tumor necrosis factor alpha is an autocrine growth factor for normal human B cells. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[25] Kozo Nakamura,et al. Connection Between B Lymphocyte and Osteoclast Differentiation Pathways1 , 2001, The Journal of Immunology.
[26] J. Lederer,et al. Interaction Between the Innate and Adaptive Immune Systems is Required to Survive Sepsis and Control Inflammation After Injury , 2003, Shock.
[27] J. Farber. A macrophage mRNA selectively induced by gamma-interferon encodes a member of the platelet factor 4 family of cytokines. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[28] T. Einhorn,et al. Induction of apoptosis in chondrocytes by tumor necrosis factor‐alpha , 2001, Journal of orthopaedic research : official publication of the Orthopaedic Research Society.
[29] F. Sallusto,et al. Cytokine-driven proliferation and differentiation of human naïve, central memory and effector memory CD4+ T cells. , 2003, Pathologie-biologie.
[30] Wei-Ping Qian,et al. IL-7 induces bone loss in vivo by induction of receptor activator of nuclear factor κB ligand and tumor necrosis factor α from T cells , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[31] H. Takayanagi. New developments in osteoimmunology , 2012, Nature Reviews Rheumatology.
[32] A. Cope,et al. Treg cells suppress osteoclast formation: a new link between the immune system and bone. , 2007, Arthritis and rheumatism.
[33] G. Duda,et al. The early fracture hematoma and its potential role in fracture healing. , 2010, Tissue engineering. Part B, Reviews.
[34] K Yano,et al. Severe osteoporosis in mice lacking osteoclastogenesis inhibitory factor/osteoprotegerin. , 1998, Biochemical and biophysical research communications.
[35] C. Manferdini,et al. T cell subsets differently regulate osteogenic differentiation of human mesenchymal stromal cells in vitro , 2016, Journal of tissue engineering and regenerative medicine.
[36] S. Opal. Phylogenetic and functional relationships between coagulation and the innate immune response. , 2000, Critical care medicine.
[37] M. Terauchi,et al. T cells potentiate PTH-induced cortical bone loss through CD40L signaling. , 2008, Cell metabolism.
[38] W. Dougall,et al. RANK is essential for osteoclast and lymph node development. , 1999, Genes & development.
[39] Xudong Huang,et al. The role of T cells in osteoporosis, an update. , 2009, International journal of clinical and experimental pathology.
[40] Wei-Ping Qian,et al. B cells and T cells are critical for the preservation of bone homeostasis and attainment of peak bone mass in vivo. , 2007, Blood.
[41] M. Feldmann,et al. TNF-α promotes fracture repair by augmenting the recruitment and differentiation of muscle-derived stromal cells , 2011, Proceedings of the National Academy of Sciences.
[42] S. Nedospasov,et al. Intracellular signals and events activated by cytokines of the tumor necrosis factor superfamily: From simple paradigms to complex mechanisms. , 2006, International Review of Cytology.
[43] T A Einhorn,et al. The cell and molecular biology of fracture healing. , 1998, Clinical orthopaedics and related research.
[44] N. Haas. Callusmodulation – Fiktion oder Realität? , 2000, Der Chirurg.
[45] G. Duda,et al. Initial immune reaction and angiogenesis in bone healing , 2014, Journal of tissue engineering and regenerative medicine.
[46] É. Hajduch,et al. The Role of Inflammatory Cytokines, the RANKL/OPG Axis, and the Immunoskeletal Interface in Physiological Bone Turnover and Osteoporosis , 2013, Scientifica.
[47] Subburaman Mohan,et al. Microarray analysis of gene expression during the inflammation and endochondral bone formation stages of rat femur fracture repair. , 2006, Bone.
[48] W. Olszewski,et al. Lymphoscintigraphic monitoring of the lower limb lymphatic system response to bone fracture and healing. , 2005, Lymphatic research and biology.
[49] L. Donahue,et al. Genetic variability in adult bone density among inbred strains of mice. , 1996, Bone.
[50] M. Rossol,et al. Cytokine-induced human IFN-gamma-secreting effector-memory Th cells in chronic autoimmune inflammation. , 2009, Blood.
[51] Andrew D. Luster,et al. γ-Interferon transcriptionally regulates an early-response gene containing homology to platelet proteins , 1985, Nature.
[52] L. Flamand,et al. Normal human primary CD4+ T lymphocytes synthesize and release functional osteoprotegerin in vitro , 2008, Laboratory Investigation.
[53] G. Duda,et al. Fracture healing is accelerated in the absence of the adaptive immune system , 2010, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[54] Georg N Duda,et al. Differential regulation of blood vessel formation between standard and delayed bone healing , 2009, Journal of orthopaedic research : official publication of the Orthopaedic Research Society.
[55] S. Abrignani,et al. Human naive T cells activated by cytokines differentiate into a split phenotype with functional features intermediate between naive and memory T cells. , 1995, International immunology.
[56] W. Olszewski,et al. Limb lymph node response to bone fracture. , 2004, Lymphatic research and biology.
[57] D. Marsh,et al. Inflammatory cells in normal human fracture healing. , 1994, Acta orthopaedica Scandinavica.
[58] I. Chaudry,et al. The role of γδ T cells in the regulation of neutrophil‐mediated tissue damage after thermal injury , 2004 .
[59] Paul Martin,et al. Inflammatory cells during wound repair: the good, the bad and the ugly. , 2005, Trends in cell biology.
[60] G. Franzoso,et al. Structure, regulation and function of NF-kappa B. , 1994, Annual review of cell biology.
[61] C. Murphy,et al. TNFα Potently Activates Osteoclasts, through a Direct Action Independent of and Strongly Synergistic with RANKL. , 2002, Endocrinology.
[62] Navrag B. Singh,et al. Terminally Differentiated CD8+ T Cells Negatively Affect Bone Regeneration in Humans , 2013, Science Translational Medicine.
[63] Thomas A Einhorn,et al. Differential Temporal Expression of Members of the Transforming Growth Factor β Superfamily During Murine Fracture Healing , 2002, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[64] P. Pileri,et al. Antigen-independent activation of naive and memory resting T cells by a cytokine combination , 1994, The Journal of experimental medicine.
[65] Thomas A Einhorn,et al. Fracture healing as a post‐natal developmental process: Molecular, spatial, and temporal aspects of its regulation , 2003, Journal of cellular biochemistry.
[66] G. Duda,et al. Cellular composition of the initial fracture hematoma compared to a muscle hematoma: A study in sheep , 2009, Journal of orthopaedic research : official publication of the Orthopaedic Research Society.
[67] P M Rommens,et al. [Osteoblasts : cellular and molecular regulatory mechanisms in fracture healing]. , 2009, Der Orthopade.
[68] Georg N. Duda,et al. Inflammatory phase of bone healing initiates the regenerative healing cascade , 2011, Cell and Tissue Research.
[69] I. Ofotokun,et al. Alterations in the immuno-skeletal interface drive bone destruction in HIV-1 transgenic rats , 2010, Proceedings of the National Academy of Sciences.
[70] I. Kalajzic,et al. Role of B Lymphocytes in New Bone Formation , 2000, Laboratory Investigation.