Statistical analysis of clinical risk factors for coronary artery spasm: identification of the most important determinant.

[1]  A. Kuroiwa,et al.  Comparison of serum lipid values in variant angina pectoris and fixed coronary artery disease with normal subjects. , 1989, The American journal of cardiology.

[2]  S. Narumiya,et al.  Cholesteryl ester accumulation in macrophages incubated with low density lipoprotein pretreated with cigarette smoke extract. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[3]  A. Ooshima,et al.  Probucol prevents the progression of atherosclerosis in Watanabe heritable hyperlipidemic rabbit, an animal model for familial hypercholesterolemia. , 1987, Proceedings of the National Academy of Sciences of the United States of America.

[4]  Toru Kita,et al.  Stimulation of cholesteryl ester synthesis in mouse peritoneal macrophages by cholesterol-rich very low density lipoproteins from the Watanabe heritable hyperlipidemic rabbit, an animal model of familial hypercholesterolemia. , 1986, The Journal of clinical investigation.

[5]  M. Winniford,et al.  Smoking-induced coronary vasoconstriction in patients with atherosclerotic coronary artery disease: evidence for adrenergically mediated alterations in coronary artery tone. , 1986, Circulation.

[6]  A. Gotto.  Interactions of the major risk factors for coronary heart disease. , 1986, The American journal of medicine.

[7]  P. Ducimetiere,et al.  Comparison of risk factors in vasospastic angina without significant fixed coronary narrowing to significant fixed coronary narrowing and no vasospastic angina. , 1986, The American journal of cardiology.

[8]  M. de Lorgeril,et al.  Acute influence of cigarette smoke in platelets, catecholamines and neurophysins in the normal conditions of daily life. , 1985, European heart journal.

[9]  K. Tanaka,et al.  Coronary artery spasm induced in miniature swine: angiographic evidence and relation to coronary atherosclerosis. , 1985, American heart journal.

[10]  M. Winniford,et al.  Acute systemic and coronary hemodynamic and serologic responses to cigarette smoking in long-term smokers with atherosclerotic coronary artery disease. , 1984, Journal of the American College of Cardiology.

[11]  T. Rink,et al.  The role of cytoplasmic free calcium in the responses of quin2-loaded human platelets to vasopressin. , 1984, The Biochemical journal.

[12]  W. Laskey,et al.  Acute coronary vasoconstrictive effects of cigarette smoking in coronary heart disease. , 1984, The American journal of cardiology.

[13]  M. Berridge,et al.  Rapid mobilization of Ca2+ from rat insulinoma microsomes by inositol-1,4,5-trisphosphate , 1984, Nature.

[14]  K. Tanaka,et al.  Selective hypercontraction caused by ergonovine in the canine coronary artery under conditions of induced atherosclerosis. , 1984, Circulation.

[15]  F. Fernández,et al.  Diffuse or segmental narrowing (spasm) of the coronary arteries during smoking demonstrated on angiography. , 1984, The American journal of cardiology.

[16]  R. Kronmal,et al.  Coronary Arteriography and Coronary Artery Bypass Surgery: Morbidity and Mortality in Patients Ages 65 Years or Older A Report from the Coronary Artery Surgery Study , 1983, Circulation.

[17]  T. Kita,et al.  Cellular Pathology of Progressive Atherosclerosis in the WHHL Rabbit: An Animal Model of Familial Hypercholesterolemia , 1983, Arteriosclerosis.

[18]  P. Weber,et al.  Plasma Catecholamines, Platelet Aggregation and Associated Thromboxane Formation After Physical Exercise, Smoking or Norepinephrine Infusion , 1982, Circulation.

[19]  S. Chierchia,et al.  [The role of spasm in angina pectoris, myocardial infarction and sudden death. Indications for future research and treatment]. , 1982, Archives des maladies du coeur et des vaisseaux.

[20]  M. Inoue,et al.  Elevation of Thromboxane B2 Levels in Patients with Classic and Variant Angina Pectoris , 1981, Circulation.

[21]  R. Gerrity,et al.  The role of the monocyte in atherogenesis: II. Migration of foam cells from atherosclerotic lesions. , 1981, The American journal of pathology.

[22]  V. Fuster,et al.  Platelet Survival and the Development of Coronary Artery Disease in the Young Adult: Effects of Cigarette Smoking, Strong Family History and Medical Therapy , 1981, Circulation.

[23]  Å. Wennmalm.  NICOTINE INHIBITS HYPOXIA‐ AND ARACHIDONATE‐INDUCED RELEASE OF PROSTACYCLIN‐LIKE ACTIVITY IN RABBIT HEARTS , 1980, British journal of pharmacology.

[24]  R. Macalpin,et al.  Relation of coronary arterial spasm to sites of organic stenosis. , 1980, The American journal of cardiology.

[25]  J. Rowe,et al.  Evidence in man that cigarette smoking induces vasopressin release via an airway-specific mechanism. , 1980, The Journal of clinical endocrinology and metabolism.

[26]  M. Trivella,et al.  Some clinical considerations regarding the relation of coronary vasospasm to coronary atherosclerosis: a hypothetical pathogenesis. , 1980, The American journal of cardiology.

[27]  R. Macalpin,et al.  Contribution of Dynamic Vascular Wall Thickening to Luminal Narrowing During Coronary Arterial Constriction , 1980, Circulation.

[28]  S. Chierchia,et al.  "Variant" angina: one aspect of a continuous spectrum of vasospastic myocardial ischemia. Pathogenetic mechanisms, estimated incidence and clinical and coronary arteriographic findings in 138 patients. , 1978, The American journal of cardiology.

[29]  H. Hellstrom.  The injury-vasospasm hypothesis of ischemic heart disease, revisited. , 1977, American heart journal.

[30]  P. B. Oliva,et al.  Arteriographic Evidence of Coronary Arterial Spasm in Acute Myocardial Infarction , 1977, Circulation.

[31]  M C Hjortland,et al.  High density lipoprotein as a protective factor against coronary heart disease. The Framingham Study. , 1977, The American journal of medicine.

[32]  T. Chang,et al.  Cesarean section and genital herpes. , 1977, The New England journal of medicine.

[33]  P. Cryer,et al.  Norepinephrine and epinephrine release and adrenergic mediation of smoking-associated hemodynamic and metabolic events. , 1976, The New England journal of medicine.

[34]  H. Yasue,et al.  Prinzmetal's variant form of angina as a manifestation of alpha-adrenergic receptor-mediated coronary artery spasm: documentation by coronary arteriography. , 1976, American heart journal.

[35]  A. Frantz,et al.  Nicotine-stimulated release of neurophysin and vasopressin in humans. , 1975, The Journal of clinical endocrinology and metabolism.

[36]  R. Frye,et al.  A reporting system on patients evaluated for coronary artery disease. Report of the Ad Hoc Committee for Grading of Coronary Artery Disease, Council on Cardiovascular Surgery, American Heart Association. , 1975, Circulation.

[37]  R. Turner,et al.  Smoking and the heart. The basis for action. , 1974, Lancet.

[38]  P. Levine.  An Acute Effect of Cigarette Smoking on Platelet Function: A Possible Link Between Smoking and Arterial Thrombosis , 1973, Circulation.

[39]  L. Truelove,et al.  Antidiuretic Action of Nicotine , 1945, British medical journal.

[40]  T. Ohe,et al.  Age-related changes of clinical features and prevalences of coronary risk factors in Japanese patients with vasospastic angina. , 1986, Chest.

[41]  J. Hébert,et al.  Cigarette smoking during coronary angiography: diffuse or focal narrowing (spasm) of the coronary arteries in 13 patients with angina at rest and normal coronary angiograms. , 1986, Catheterization and cardiovascular diagnosis.

[42]  C. Conti,et al.  What is the role of coronary artery spasm in ischemic heart disease? , 1983, Cardiovascular clinics.

[43]  M. Brown,et al.  Lipoprotein metabolism in the macrophage: implications for cholesterol deposition in atherosclerosis. , 1983, Annual review of biochemistry.