Lineage tracing of cells involved in atherosclerosis.

[1]  S. Schwartz.  Faculty Opinions recommendation of Corrigendum: KLF4-dependent phenotypic modulation of smooth muscle cells has a key role in atherosclerotic plaque pathogenesis. , 2018, Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature.

[2]  M. Bennett,et al.  Vascular Smooth Muscle Cells in Atherosclerosis. , 2016, Circulation research.

[3]  N. Wettschureck,et al.  S1P2/G12/13 Signaling Negatively Regulates Macrophage Activation and Indirectly Shapes the Atheroprotective B1-Cell Population , 2016, Arteriosclerosis, thrombosis, and vascular biology.

[4]  Laura S. Shankman,et al.  Correction: Corrigendum: KLF4-dependent phenotypic modulation of smooth muscle cells has a key role in atherosclerotic plaque pathogenesis , 2015, Nature Medicine.

[5]  Laura S. Shankman,et al.  KLF4 Dependent Phenotypic Modulation of SMCs Plays a Key Role in Atherosclerotic Plaque Pathogenesis , 2015, Nature Medicine.

[6]  Ira Tabas,et al.  Recent insights into the cellular biology of atherosclerosis , 2015, The Journal of cell biology.

[7]  M. Nahrendorf,et al.  Do vascular smooth muscle cells differentiate to macrophages in atherosclerotic lesions? , 2014, Circulation research.

[8]  R. Feil,et al.  Transdifferentiation of Vascular Smooth Muscle Cells to Macrophage-Like Cells During Atherogenesis , 2014, Circulation research.

[9]  S. Allahverdian,et al.  Contribution of Intimal Smooth Muscle Cells to Cholesterol Accumulation and Macrophage-Like Cells in Human Atherosclerosis , 2014, Circulation.

[10]  M. Nussenzweig,et al.  Intestinal monocytes and macrophages are required for T cell polarization in response to Citrobacter rodentium , 2013, The Journal of experimental medicine.

[11]  K. Moore,et al.  Macrophages in atherosclerosis: a dynamic balance , 2013, Nature Reviews Immunology.

[12]  G. Ramadori,et al.  Identification of CD68(+) neutrophil granulocytes in in vitro model of acute inflammation and inflammatory bowel disease. , 2013, International journal of clinical and experimental pathology.

[13]  Quin F. Wills,et al.  Methods for qPCR gene expression profiling applied to 1440 lymphoblastoid single cells , 2013, Methods.

[14]  G. Owens,et al.  Smooth muscle cell phenotypic switching in atherosclerosis. , 2012, Cardiovascular research.

[15]  Johannes E. Schindelin,et al.  Fiji: an open-source platform for biological-image analysis , 2012, Nature Methods.

[16]  Klaus Ley,et al.  Monocyte and macrophage dynamics during atherogenesis. , 2011, Arteriosclerosis, thrombosis, and vascular biology.

[17]  K. Moore,et al.  Macrophages in the Pathogenesis of Atherosclerosis , 2011, Cell.

[18]  L. Luo,et al.  A global double‐fluorescent Cre reporter mouse , 2007, Genesis.

[19]  O. McDonald,et al.  A G/C Element Mediates Repression of the SM22&agr; Promoter Within Phenotypically Modulated Smooth Muscle Cells in Experimental Atherosclerosis , 2004, Circulation research.

[20]  E. Fisher,et al.  Transdifferentiation of mouse aortic smooth muscle cells to a macrophage-like state after cholesterol loading , 2003, Proceedings of the National Academy of Sciences of the United States of America.

[21]  W. Edwards,et al.  Smooth muscle cells in human coronary atherosclerosis can originate from cells administered at marrow transplantation , 2003, Proceedings of the National Academy of Sciences of the United States of America.

[22]  R. Virmani,et al.  Lessons from sudden coronary death: a comprehensive morphological classification scheme for atherosclerotic lesions. , 2000, Arteriosclerosis, thrombosis, and vascular biology.

[23]  M. Bureau,et al.  Protective role of interleukin-10 in atherosclerosis. , 1999, Circulation research.

[24]  W. Reith,et al.  Conditional gene targeting in macrophages and granulocytes using LysMcre mice , 1999, Transgenic Research.

[25]  P. Libby,et al.  The unstable atheroma. , 1997, Arteriosclerosis, thrombosis, and vascular biology.

[26]  N. Maeda,et al.  Generation of mice carrying a mutant apolipoprotein E gene inactivated by gene targeting in embryonic stem cells. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[27]  R. Renkawitz,et al.  Mouse lysozyme M gene: isolation, characterization, and expression studies. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[28]  Stefan Offermanns,et al.  G12-G13–LARG–mediated signaling in vascular smooth muscle is required for salt-induced hypertension , 2008, Nature Medicine.