Class II MHC Expression Inhibition of Class II Transactivator and β The Smad 3 Protein Is Involved in TGF-Benveniste
暂无分享,去创建一个
[1] M. Gimbrone,et al. Inhibition of E-Selectin Gene Expression by Transforming Growth Factor β in Endothelial Cells Involves Coactivator Integration of Smad and Nuclear Factor κB–Mediated Signals , 2000, The Journal of experimental medicine.
[2] J. Harton,et al. Class II Transactivator: Mastering the Art of Major Histocompatibility Complex Expression , 2000, Molecular and Cellular Biology.
[3] V. Nguyen,et al. Involvement of STAT-1 and Ets Family Members in Interferon-γ Induction of CD40 Transcription in Microglia/Macrophages* , 2000, The Journal of Biological Chemistry.
[4] H. Lodish,et al. Role of transforming growth factor beta in human disease. , 2000, The New England journal of medicine.
[5] W. Reith,et al. CIITA is a transcriptional coactivator that is recruited to MHC class II promoters by multiple synergistic interactions with an enhanceosome complex. , 2000, Genes & development.
[6] J. Massagué,et al. Transcriptional control by the TGF‐β/Smad signaling system , 2000 .
[7] K. Miyazono,et al. c-Ski Acts as a Transcriptional Co-repressor in Transforming Growth Factor-β Signaling through Interaction with Smads* , 1999, The Journal of Biological Chemistry.
[8] A. Roberts. TGF-β signaling from receptors to the nucleus , 1999 .
[9] S. Wahl,et al. Manipulation of TGF-β to control autoimmune and chronic inflammatory diseases , 1999 .
[10] J. Ting,et al. Differential selectivity of CIITA promoter activation by IFN-γ and IRF-1 in astrocytes and macrophages: CIITA promoter activation is not affected by TNF-α , 1999, Journal of Neuroimmunology.
[11] B. Peterlin,et al. The class II transactivator CIITA is a transcriptional integrator. , 1999, Microbes and infection.
[12] J. Ting,et al. Class II MHC genes: a model gene regulatory system with great biologic consequences. , 1999, Microbes and infection.
[13] Qiang Zhou,et al. The Ski oncoprotein interacts with the Smad proteins to repress TGFbeta signaling. , 1999, Genes & development.
[14] E. Benveniste,et al. IFN-gamma regulation of the type IV class II transactivator promoter in astrocytes. , 1999, Journal of immunology.
[15] J. Massagué,et al. A Smad Transcriptional Corepressor , 1999, Cell.
[16] A. Roberts,et al. Targeted disruption of SMAD3 results in impaired mucosal immunity and diminished T cell responsiveness to TGF‐β , 1999, The EMBO journal.
[17] J. Ting,et al. Two Distinct Gamma Interferon-Inducible Promoters of the Major Histocompatibility Complex Class II Transactivator Gene Are Differentially Regulated by STAT1, Interferon Regulatory Factor 1, and Transforming Growth Factor β , 1999, Molecular and Cellular Biology.
[18] M. Merika,et al. Involvement of CREB Binding Protein in Expression of Major Histocompatibility Complex Class II Genes via Interaction with the Class II Transactivator , 1998, Molecular and Cellular Biology.
[19] C. Heldin,et al. Identification and Functional Characterization of a Smad Binding Element (SBE) in the JunB Promoter That Acts as a Transforming Growth Factor-β, Activin, and Bone Morphogenetic Protein-inducible Enhancer* , 1998, The Journal of Biological Chemistry.
[20] C. Deng,et al. Failure of egg cylinder elongation and mesoderm induction in mouse embryos lacking the tumor suppressor smad2. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[21] E. Li,et al. Smad2 role in mesoderm formation, left–right patterning and craniofacial development , 1998, Nature.
[22] B. Mach,et al. Quantitative control of MHC class II expression by the transactivator CIITA , 1998, European journal of immunology.
[23] B. Mach,et al. Activation of the MHC class II transactivator CIITA by interferon-gamma requires cooperative interaction between Stat1 and USF-1. , 1998, Immunity.
[24] J. Ting,et al. Identification of Distinct Regions of 5′ Flanking DNA That Mediate Constitutive, IFN-γ, STAT1, and TGF-β-Regulated Expression of the Class II Transactivator Gene , 1998, The Journal of Immunology.
[25] J. Strominger,et al. Transactivation by CIITA, the type II bare lymphocyte syndrome-associated factor, requires participation of multiple regions of the TATA box binding protein. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[26] B. Peterlin,et al. The class II trans-activator CIITA interacts with the TBP-associated factor TAFII32. , 1997, Nucleic acids research.
[27] B. Mach,et al. Expression of MHC class II molecules in different cellular and functional compartments is controlled by differential usage of multiple promoters of the transactivator CIITA , 1997, The EMBO journal.
[28] R. Ransohoff,et al. TGF-beta suppresses IFN-gamma induction of class II MHC gene expression by inhibiting class II transactivator messenger RNA expression. , 1997, Journal of immunology.
[29] A. Kulkarni,et al. Autoimmunity associated with TGF-beta1-deficiency in mice is dependent on MHC class II antigen expression. , 1996, The Journal of clinical investigation.
[30] R. Flavell,et al. Mice lacking the MHC class II transactivator (CIITA) show tissue-specific impairment of MHC class II expression. , 1996, Immunity.
[31] J. Darnell,et al. Maximal activation of transcription by statl and stat3 requires both tyrosine and serine phosphorylation , 1995, Cell.
[32] Y. Lee,et al. TGF-beta suppression of IFN-gamma-induced class II MHC gene expression does not involve inhibition of phosphorylation of JAK1, JAK2, or signal transducers and activators of transcription, or modification of IFN-gamma enhanced factor X expression. , 1995, Journal of immunology.
[33] Y. Lee,et al. Characterization of astrocyte nuclear proteins involved in IFN-gamma- and TNF-alpha-mediated class II MHC gene expression. , 1994, Journal of immunology.
[34] R. Flavell,et al. Class II transactivator (CIITA) is sufficient for the inducible expression of major histocompatibility complex class II genes , 1994, The Journal of experimental medicine.
[35] B. Mach,et al. Regulation of MHC class II expression by interferon-gamma mediated by the transactivator gene CIITA. , 1994, Science.
[36] J. W. Rooney,et al. Transforming growth factor beta 1 repression of the HLA-DR alpha gene is mediated by conserved proximal promoter elements. , 1993, Journal of immunology.
[37] F. Ruscetti,et al. Pleiotropic Effects of Transforming Growth Factor‐β on Cells of the Immune System , 1993, Annals of the New York Academy of Sciences.
[38] G. Proetzel,et al. Targeted disruption of the mouse transforming growth factor-β1 gene results in multifocal inflammatory disease , 1992, Nature.
[39] L. Matrisian,et al. TGF-β1 inhibition of transin/stromelysin gene expression is mediated through a fos binding sequence , 1990, Cell.
[40] G. Wong,et al. Transforming growth factor-beta 1 modulates the expression of class II histocompatibility antigens on human cells. , 1988, Journal of immunology.
[41] M. Sporn,et al. Production of transforming growth factor beta by human T lymphocytes and its potential role in the regulation of T cell growth , 1986, The Journal of experimental medicine.
[42] L. Eng,et al. Localization of the glial fibrillary acidic protein in astrocytes by immunofluorescence. , 1972, Brain research.
[43] D. Singer,et al. Transcriptional coactivator, CIITA, is an acetyltransferase that bypasses a promoter requirement for TAF(II)250. , 2001, Molecular cell.
[44] J. Massagué,et al. Inhibition of transforming growth factor-beta/SMAD signalling by the interferon-gamma/STAT pathway. , 1999, Nature.
[45] A. Roberts,et al. Regulation of immune responses by TGF-beta. , 1998, Annual review of immunology.
[46] J. Massagué,et al. TGF- SIGNAL TRANSDUCTION , 1998 .
[47] Y. Lee,et al. Regulation of class II MHC expression. , 1996, Critical reviews in immunology.
[48] L. Glimcher,et al. Immune responses in MHC class II-deficient mice. , 1995, Annual Review of Immunology.