ZNF281 contributes to the DNA damage response by controlling the expression of XRCC2 and XRCC4

[1]  J. Sáez-Vásquez,et al.  Nucleolin: dual roles in rDNA chromatin transcription. , 2015, Gene.

[2]  R A Knight,et al.  PPISURV: a novel bioinformatics tool for uncovering the hidden role of specific genes in cancer survival outcome , 2014, Oncogene.

[3]  R. Sears,et al.  Endogenous c-Myc is essential for p53-induced apoptosis in response to DNA damage in vivo , 2014, Cell Death and Differentiation.

[4]  Bruno Amati,et al.  Genome recognition by MYC. , 2014, Cold Spring Harbor perspectives in medicine.

[5]  R A Knight,et al.  DRUGSURV: a resource for repositioning of approved and experimental drugs in oncology based on patient survival information , 2014, Cell Death and Disease.

[6]  P. Jeggo,et al.  DNA DSB repair pathway choice: an orchestrated handover mechanism. , 2014, The British journal of radiology.

[7]  D. Adhikari,et al.  The regulation of maturation promoting factor during prophase I arrest and meiotic entry in mammalian oocytes , 2014, Molecular and Cellular Endocrinology.

[8]  H. Hermeking,et al.  SNAIL and miR‐34a feed‐forward regulation of ZNF281/ZBP99 promotes epithelial–mesenchymal transition , 2013, The EMBO journal.

[9]  I Celardo,et al.  Senescence and aging: the critical roles of p53 , 2013, Oncogene.

[10]  M. Kastan,et al.  Nucleolin mediates nucleosome disruption critical for DNA double-strand break repair , 2013, Proceedings of the National Academy of Sciences.

[11]  M. Christmann,et al.  Transcriptional regulation of human DNA repair genes following genotoxic stress: trigger mechanisms, inducible responses and genotoxic adaptation , 2013, Nucleic acids research.

[12]  Gerry Melino,et al.  Activation of miR200 by c-Myb depends on ZEB1 expression and miR200 promoter methylation , 2013, Cell cycle.

[13]  M. Crescenzi,et al.  The WRN and MUS81 proteins limit cell death and genome instability following oncogene activation , 2013, Oncogene.

[14]  M. Ikura,et al.  Transcriptional/epigenetic regulator CBP/p300 in tumorigenesis: structural and functional versatility in target recognition , 2013, Cellular and Molecular Life Sciences.

[15]  W. Curran,et al.  c-Myc suppression of DNA double-strand break repair. , 2012, Neoplasia.

[16]  R. Knight,et al.  MIRUMIR: an online tool to test microRNAs as biomarkers to predict survival in cancer using multiple clinical data sets , 2012, Cell Death and Differentiation.

[17]  S. Powell,et al.  Rad51 Paralog Complexes BCDX2 and CX3 Act at Different Stages in the BRCA1-BRCA2-Dependent Homologous Recombination Pathway , 2012, Molecular and Cellular Biology.

[18]  Charles Y. Lin,et al.  Transcriptional Amplification in Tumor Cells with Elevated c-Myc , 2012, Cell.

[19]  Julian Gingold,et al.  Zfp281 mediates Nanog autorepression through recruitment of the NuRD complex and inhibits somatic cell reprogramming , 2012, Proceedings of the National Academy of Sciences.

[20]  G. Blandino,et al.  Direct and delayed X‐ray‐induced DNA damage in male mouse germ cells , 2012, Environmental and molecular mutagenesis.

[21]  F. Markowetz,et al.  The genomic and transcriptomic architecture of 2,000 breast tumours reveals novel subgroups , 2012, Nature.

[22]  M. Paulsson,et al.  Epidermal Transglutaminase (TGase 3) Is Required for Proper Hair Development, but Not the Formation of the Epidermal Barrier , 2012, PloS one.

[23]  J. Piette,et al.  Importance of PIKKs in NF-κB activation by genotoxic stress. , 2011, Biochemical pharmacology.

[24]  Yen-Sin Ang,et al.  Zfp281 Functions as a Transcriptional Repressor for Pluripotency of Mouse Embryonic Stem Cells , 2011, Stem cells.

[25]  A. Trumpp,et al.  Genome-wide mapping of Myc binding and gene regulation in serum-stimulated fibroblasts , 2011, Oncogene.

[26]  R. Bristow,et al.  es that Control DNA Double-Strand Break Repair , 2010 .

[27]  J. Bartek,et al.  The DNA-damage response in human biology and disease , 2009, Nature.

[28]  Colleen D. McCabe,et al.  Genome-wide promoter analysis of the SOX4 transcriptional network in prostate cancer cells. , 2009, Cancer research.

[29]  David J. Chen,et al.  Live cell imaging of XLF and XRCC4 reveals a novel view of protein assembly in the non-homologous end-joining pathway , 2008, Cell cycle.

[30]  B. A. Ballif,et al.  ATM and ATR Substrate Analysis Reveals Extensive Protein Networks Responsive to DNA Damage , 2007, Science.

[31]  J. Bartek,et al.  DNA damage checkpoints: from initiation to recovery or adaptation. , 2007, Current opinion in cell biology.

[32]  J. Yates,et al.  Large-Scale Identification of c-MYC-Associated Proteins Using a Combined TAP/MudPIT Approach , 2007, Cell cycle.

[33]  D. Angelov,et al.  Nucleolin is a histone chaperone with FACT‐like activity and assists remodeling of nucleosomes , 2006, The EMBO journal.

[34]  E. Prochownik.  c-Myc as a therapeutic target in cancer , 2004, Expert review of anticancer therapy.

[35]  Chi V. Dang,et al.  A strategy for identifying transcription factor binding sites reveals two classes of genomic c-Myc target sites , 2003, Proceedings of the National Academy of Sciences of the United States of America.

[36]  G. Evan,et al.  Myc-Is this the oncogene from Hell? , 2002, Cancer cell.

[37]  C. Dang,et al.  Translocations involving c-myc and c-myc function , 2001, Oncogene.

[38]  E. Prochownik,et al.  Inverse regulation of cyclin B1 by c-Myc and p53 and induction of tetraploidy by cyclin B1 overexpression. , 2001, Cancer research.

[39]  C. Wilson,et al.  XRCC2 Is a Nuclear RAD51-like Protein Required for Damage-dependent RAD51 Focus Formation without the Need for ATP Binding* , 2001, The Journal of Biological Chemistry.

[40]  J. Bartek,et al.  Pathways governing G1/S transition and their response to DNA damage , 2001, FEBS letters.

[41]  J. Merchant,et al.  ZBP-99 defines a conserved family of transcription factors and regulates ornithine decarboxylase gene expression. , 1999, Biochemical and biophysical research communications.

[42]  B. Calabretta,et al.  Targeting c-myc in leukemia. , 1997, Anti-cancer drug design.

[43]  M. Reivich,et al.  ACTIVATION , 1980, The Social Value of Zoos.

[44]  David G Johnson,et al.  Transcriptional and nontranscriptional functions of E2F1 in response to DNA damage. , 2012, Cancer research.

[45]  R. Eisenman,et al.  The Max transcription factor network: involvement of Mad in differentiation and an approach to identification of target genes. , 1994, Cold Spring Harbor symposia on quantitative biology.