Bcl-2 up-regulates ha-ras mRNA expression and induces c-Jun phosphorylation at Ser73 via an ERK-dependent pathway in PC 12 cells
暂无分享,去创建一个
[1] E. Wagner,et al. Transgenic Activation of Ras in Neurons Promotes Hypertrophy and Protects from Lesion-Induced Degeneration , 2000, The Journal of cell biology.
[2] W. Ansorge,et al. Complex Functions of AP-1 Transcription Factors in Differentiation and Survival of PC12 Cells , 2001, Molecular and Cellular Biology.
[3] Aaron P. Campbell,et al. Suppression subtractive hybridization: a method for generating differentially regulated or tissue-specific cDNA probes and libraries. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[4] A. Suzuki,et al. Bcl-2 accelerates the neuronal differentiation: new evidence approaching to the biofunction of Bcl-2 in the neuronal system , 1998, Brain Research.
[5] D. Green,et al. The Release of Cytochrome c from Mitochondria: A Primary Site for Bcl-2 Regulation of Apoptosis , 1997, Science.
[6] E. Wagner,et al. Amino-terminal phosphorylation of c-Jun regulates stress-induced apoptosis and cellular proliferation , 1999, Nature Genetics.
[7] J. Frost,et al. A requirement for extracellular signal-regulated kinase (ERK) function in the activation of AP-1 by Ha-Ras, phorbol 12-myristate 13-acetate, and serum. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[8] R. Treisman,et al. Regulation of transcription by MAP kinase cascades. , 1996, Current opinion in cell biology.
[9] M. Cobb,et al. Mitogen-activated protein kinase pathways. , 1997, Current opinion in cell biology.
[10] S. R. Datta,et al. Cell survival promoted by the Ras-MAPK signaling pathway by transcription-dependent and -independent mechanisms. , 1999, Science.
[11] Y. Oh,et al. Activation of c-Jun N-terminal Kinase Antagonizes an Anti-apoptotic Action of Bcl-2* , 1997, The Journal of Biological Chemistry.
[12] S. Cory,et al. The Bcl-2 protein family: arbiters of cell survival. , 1998, Science.
[13] E. Bossy‐Wetzel,et al. Induction of apoptosis by the transcription factor c‐Jun , 1997, The EMBO journal.
[14] C. Marshall,et al. Activation of MAP kinase kinase is necessary and sufficient for PC12 differentiation and for transformation of NIH 3T3 cells , 1994, Cell.
[15] Dirk Bohmann,et al. Reduced Ubiquitin-Dependent Degradation of c-Jun After Phosphorylation by MAP Kinases , 1997, Science.
[16] E. Van Obberghen,et al. Cyclic AMP activates the mitogen-activated protein kinase cascade in PC12 cells. , 1994, The Journal of biological chemistry.
[17] D. Bar-Sagi,et al. Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway. , 2000, Science.
[18] Michael E. Greenberg,et al. Opposing Effects of ERK and JNK-p38 MAP Kinases on Apoptosis , 1995, Science.
[19] D. Green,et al. Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD‐specific caspase activation and independently of mitochondrial transmembrane depolarization , 1998, The EMBO journal.
[20] B. Pettmann,et al. Neuronal Cell Death , 1998, Neuron.
[21] J Downward,et al. Ras signalling and apoptosis. , 1998, Current opinion in genetics & development.
[22] O. Bernard,et al. Dominant-Negative c-Jun Promotes Neuronal Survival by Reducing BIM Expression and Inhibiting Mitochondrial Cytochrome c Release , 2001, Neuron.
[23] D. Kaplan,et al. Signal transduction by the neutrophin receptors , 1997 .
[24] T. Klockgether,et al. Overexpression of bcl-2 results in reduction of cytochrome c content and inhibition of complex I activity. , 2001, Biochemical and biophysical research communications.
[25] E. Zandi,et al. AP-1 function and regulation. , 1997, Current opinion in cell biology.