TLN-58, an Additional hCAP18 Processing Form, Found in the Lesion Vesicle of Palmoplantar Pustulosis in the Skin.

[1]  A. Ishida-Yamamoto,et al.  Vesicular LL-37 Contributes to Inflammation of the Lesional Skin of Palmoplantar Pustulosis , 2014, PloS one.

[2]  K. Sayama,et al.  Eccrine Sweat Contains IL-1α, IL-1β and IL-31 and Activates Epidermal Keratinocytes as a Danger Signal , 2013, PloS one.

[3]  Hideoki Ogawa,et al.  Human antimicrobial peptide LL-37 modulates proinflammatory responses induced by cytokine milieus and double-stranded RNA in human keratinocytes. , 2013, Biochemical and biophysical research communications.

[4]  T. Bíró,et al.  Endocannabinoids regulate growth and survival of human eccrine sweat gland-derived epithelial cells. , 2012, The Journal of investigative dermatology.

[5]  Dong-Youn Lee,et al.  An antimicrobial protein, lactoferrin exists in the sweat: proteomic analysis of sweat , 2011, Experimental dermatology.

[6]  K. Sayama,et al.  Mite allergen is a danger signal for the skin via activation of inflammasome in keratinocytes. , 2011, The Journal of allergy and clinical immunology.

[7]  E. Diamandis,et al.  Kallikrein-related Peptidase-8 (KLK8) Is an Active Serine Protease in Human Epidermis and Sweat and Is Involved in a Skin Barrier Proteolytic Cascade , 2010, The Journal of Biological Chemistry.

[8]  H. Hedstrand,et al.  Novel findings of Langerhans cells and interleukin‐17 expression in relation to the acrosyringium and pustule in palmoplantar pustulosis , 2010, The British journal of dermatology.

[9]  A. Ishida-Yamamoto,et al.  Acrosyringium is the main site of the vesicle/pustule formation in palmoplantar pustulosis. , 2010, Journal of Investigative Dermatology.

[10]  K. Sayama,et al.  Living skin equivalents constructed using human amnions as a matrix. , 2009, Journal of dermatological science.

[11]  P. Parren,et al.  IL-8 as Antibody Therapeutic Target in Inflammatory Diseases: Reduction of Clinical Activity in Palmoplantar Pustulosis1 , 2008, The Journal of Immunology.

[12]  A. Hovnanian,et al.  Increased serine protease activity and cathelicidin promotes skin inflammation in rosacea , 2007, Nature Medicine.

[13]  Henry Lin,et al.  The FASEB Journal • Research Communication Kallikrein-mediated proteolysis regulates the antimicrobial effects of cathelicidins in skin , 2022 .

[14]  K. Rabe,et al.  The antimicrobial peptide LL-37 enhances IL-8 release by human airway smooth muscle cells. , 2006, The Journal of allergy and clinical immunology.

[15]  H. Tagami,et al.  Localization of IL-8 and Complement Components in Lesional Skin of Psoriasis vulgaris and Pustulosis palmaris et plantaris , 2005, Dermatology.

[16]  R. Gallo,et al.  Structure-Function Relationships among Human Cathelicidin Peptides: Dissociation of Antimicrobial Properties from Host Immunostimulatory Activities , 2005, The Journal of Immunology.

[17]  R. Gallo,et al.  Postsecretory Processing Generates Multiple Cathelicidins for Enhanced Topical Antimicrobial Defense1 , 2004, The Journal of Immunology.

[18]  R. Gallo,et al.  Neonatal Skin in Mice and Humans Expresses Increased Levels of Antimicrobial Peptides: Innate Immunity During Development of the Adaptive Response , 2003, Pediatric Research.

[19]  Takaaki Ohtake,et al.  Biology and clinical relevance of naturally occurring antimicrobial peptides. , 2002, The Journal of allergy and clinical immunology.

[20]  C. Garbe,et al.  Cathelicidin anti-microbial peptide expression in sweat, an innate defense system for the skin. , 2002, The Journal of investigative dermatology.

[21]  Tomio Ogasawara,et al.  A cell-free protein synthesis system for high-throughput proteomics , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[22]  Nikolaus Blin,et al.  Dermcidin: a novel human antibiotic peptide secreted by sweat glands , 2001, Nature Immunology.

[23]  J. Calafat,et al.  Human cathelicidin, hCAP-18, is processed to the antimicrobial peptide LL-37 by extracellular cleavage with proteinase 3. , 2001, Blood.

[24]  H. Jörnvall,et al.  The human antimicrobial and chemotactic peptides LL-37 and alpha-defensins are expressed by specific lymphocyte and monocyte populations. , 2000, Blood.

[25]  Ji Ming Wang,et al.  Ll-37, the Neutrophil Granule–And Epithelial Cell–Derived Cathelicidin, Utilizes Formyl Peptide Receptor–Like 1 (Fprl1) as a Receptor to Chemoattract Human Peripheral Blood Neutrophils, Monocytes, and T Cells , 2000, The Journal of experimental medicine.

[26]  Y Endo,et al.  A highly efficient and robust cell-free protein synthesis system prepared from wheat embryos: plants apparently contain a suicide system directed at ribosomes. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[27]  G. Michaëlsson,et al.  Palmoplantar pustulosis: a clinical and immunohistological study , 1998, The British journal of dermatology.

[28]  H. Wigzell,et al.  The Expression of the Gene Coding for the Antibacterial Peptide LL-37 Is Induced in Human Keratinocytes during Inflammatory Disorders* , 1997, The Journal of Biological Chemistry.

[29]  E. Valore,et al.  Broad-spectrum antimicrobial activity of human intestinal defensin 5 , 1997, Infection and immunity.

[30]  E. Bröcker,et al.  Differential Expression of GRO-α and IL-8 mRNA Psoriasis: A Model for Neutrophil Migration and Accumulation In Vivo , 1996 .

[31]  C. Suschek,et al.  A proinflammatory activity of interleukin 8 in human skin: expression of the inducible nitric oxide synthase in psoriatic lesions and cultured keratinocytes , 1996, The Journal of experimental medicine.

[32]  J. Schröder,et al.  Co-localized overexpression of GRO-alpha and IL-8 mRNA is restricted to the suprapapillary layers of psoriatic lesions. , 1996, The Journal of investigative dermatology.

[33]  J. Odeberg,et al.  FALL-39, a putative human peptide antibiotic, is cysteine-free and expressed in bone marrow and testis. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[34]  R. Crystal,et al.  Myelomonocytic cell lineage expression of the neutrophil elastase gene. , 1988, The Journal of biological chemistry.

[35]  C. Jones,et al.  Biochemical and ultrastructural studies of human eccrine sweat glands isolated by shearing and maintained for seven days. , 1984, Journal of cell science.

[36]  M. Uehara,et al.  The morphogenesis of pustulosis palmaris et plantaris. , 1974, Archives of dermatology.

[37]  G. Passalacqua,et al.  Journal of Allergy and clinical immunology , 2013 .

[38]  P. Altmeyer,et al.  The pustule in palmoplantar psoriasis: transformed vesicle or mature microabscess? A three-dimensional study. , 1992, Dermatology.