Phenotyping of peripheral blood mononuclear cells during acute dengue illness demonstrates infection and increased activation of monocytes in severe cases compared to classic dengue fever.

[1]  J. Schlesinger,et al.  Monocytes, but not T or B cells, are the principal target cells for dengue virus (DV) infection among human peripheral blood mononuclear cells , 2008, Journal of medical virology.

[2]  E. Harris,et al.  Dengue virus infects macrophages and dendritic cells in a mouse model of infection. , 2007, The Journal of infectious diseases.

[3]  M. St. Claire,et al.  Monoclonal antibody-mediated enhancement of dengue virus infection in vitro and in vivo and strategies for prevention , 2007, Proceedings of the National Academy of Sciences.

[4]  Charles M. Rice,et al.  Flaviviridae :T he Viruses and Their Replication , 2007 .

[5]  E. Harris,et al.  Murine Model for Dengue Virus-Induced Lethal Disease with IncreasedVascular Permeability , 2006, Journal of Virology.

[6]  E. Harris,et al.  High seroprevalence of antibodies against dengue virus in a prospective study of schoolchildren in Managua, Nicaragua , 2006, Tropical medicine & international health : TM & IH.

[7]  S. Almo,et al.  B7-1 and B7-2: similar costimulatory ligands with different biochemical, oligomeric and signaling properties. , 2006, Immunology letters.

[8]  E. Harris,et al.  Differences in dengue severity in infants, children, and adults in a 3-year hospital-based study in Nicaragua. , 2005, The American journal of tropical medicine and hygiene.

[9]  E. Harris,et al.  Dendritic Cell Precursors Are Permissive to Dengue Virus and Human Immunodeficiency Virus Infection , 2005, Journal of Virology.

[10]  I. Kurane,et al.  In vitro assessment of human endothelial cell permeability: effects of inflammatory cytokines and dengue virus infection. , 2004, Journal of virological methods.

[11]  K. Wong,et al.  Localization of dengue virus in naturally infected human tissues, by immunohistochemistry and in situ hybridization. , 2004, The Journal of infectious diseases.

[12]  S. Halstead,et al.  Dengue hemorrhagic fever in infants: a study of clinical and cytokine profiles. , 2004, The Journal of infectious diseases.

[13]  R. Steinman,et al.  DC-SIGN (CD209) Mediates Dengue Virus Infection of Human Dendritic Cells , 2003, The Journal of experimental medicine.

[14]  A. Atrasheuskaya,et al.  Anti-TNF antibody treatment reduces mortality in experimental dengue virus infection. , 2003, FEMS immunology and medical microbiology.

[15]  D. Vaughn,et al.  Dengue: an escalating problem , 2002, BMJ : British Medical Journal.

[16]  J. Mascola,et al.  Human dendritic cells as targets of dengue virus infection. , 2001, The journal of investigative dermatology. Symposium proceedings.

[17]  E. Azeredo,et al.  Characterisation of lymphocyte response and cytokine patterns in patients with dengue fever. , 2001, Immunobiology.

[18]  E. Harris,et al.  Clinical, epidemiologic, and virologic features of dengue in the 1998 epidemic in Nicaragua. , 2000, The American journal of tropical medicine and hygiene.

[19]  J. Mascola,et al.  Human skin Langerhans cells are targets of dengue virus infection , 2000, Nature Medicine.

[20]  E. Chungue,et al.  Prospective study of the duration and magnitude of viraemia in children hospitalised during the 1996–1997 dengue‐2 outbreak in French Polynesia , 2000, Journal of medical virology.

[21]  A. Nisalak,et al.  Dengue viremia titer, antibody response pattern, and virus serotype correlate with disease severity. , 2000, The Journal of infectious diseases.

[22]  A. Nisalak,et al.  B cells are the principal circulating mononuclear cells infected by dengue virus. , 1999, The Southeast Asian journal of tropical medicine and public health.

[23]  A. Pacsa,et al.  Sequential production of cytokines by dengue virus‐infected human peripheral blood leukocyte cultures , 1999, Journal of medical virology.

[24]  A. Nisalak,et al.  Early CD69 expression on peripheral blood lymphocytes from children with dengue hemorrhagic fever. , 1999, The Journal of infectious diseases.

[25]  S. A. Oliveira,et al.  Increased pro-inflammatory cytokines (TNF-alpha and IL-6) and anti-inflammatory compounds (sTNFRp55 and sTNFRp75) in Brazilian patients during exanthematic dengue fever. , 1999, Memorias do Instituto Oswaldo Cruz.

[26]  A. Rothman,et al.  Immunopathogenesis of Dengue hemorrhagic fever. , 1999, Virology.

[27]  A. Nisalak,et al.  Early immune activation in acute dengue illness is related to development of plasma leakage and disease severity. , 1999, The Journal of infectious diseases.

[28]  G. Crane Dengue haemorrhagic fever: diagnosis, treatment, prevention and control , 1999 .

[29]  D. Kwiatkowski,et al.  Pathophysiologic and prognostic role of cytokines in dengue hemorrhagic fever. , 1998, The Journal of infectious diseases.

[30]  H. Reiser,et al.  Costimulatory B7 molecules in the pathogenesis of infectious and autoimmune diseases. , 1996, The New England journal of medicine.

[31]  D. Hober,et al.  High Levels of sTNFR p75 and TNFα in Dengue‐Infected Patients , 1996 .

[32]  Arunandan Kumar,et al.  Differential modulation of B7‐1 and B7‐2 isoform expression on human monocytes by cytokines which influence the development of T helper cell phenotype , 1996, European journal of immunology.

[33]  F. Ennis,et al.  T cell activation in vivo by dengue virus infection. , 1995, Journal of clinical & laboratory immunology.

[34]  F. Ennis,et al.  Cytokines in dengue virus infections: role of cytokines in the pathogenesis of dengue hemorrhagic fever , 1994 .

[35]  I. Kurane,et al.  Immunity and immunopathology in dengue virus infections. , 1992, Seminars in immunology.

[36]  S. Halstead,et al.  Growth of dengue type 2 virus isolates in human peripheral blood leukocytes correlates with severe and mild dengue disease. , 1991, The American journal of tropical medicine and hygiene.

[37]  A. Nisalak,et al.  Activation of T lymphocytes in dengue virus infections. High levels of soluble interleukin 2 receptor, soluble CD4, soluble CD8, interleukin 2, and interferon-gamma in sera of children with dengue. , 1991, The Journal of clinical investigation.

[38]  S. Vázquez,et al.  Serological diagnosis of dengue by an ELISA inhibition method (EIM). , 1990, Memorias do Instituto Oswaldo Cruz.

[39]  F. Ennis,et al.  HUMAN IgG FC RECEPTOR I 1 MEDIATES ANTIBODY-DEPENDENT ENHANCEMENT OF DENGUE VIRUS INFECTION ' , 2001 .

[40]  A. Nisalak,et al.  Antibody-dependent enhancement of dengue virus growth in human monocytes as a risk factor for dengue hemorrhagic fever. , 1989, The American journal of tropical medicine and hygiene.

[41]  F. Ennis,et al.  Gamma interferon augments Fc gamma receptor-mediated dengue virus infection of human monocytic cells , 1988, Journal of virology.

[42]  A. Nisalak,et al.  A prospective study of dengue infections in Bangkok. , 1988, The American journal of tropical medicine and hygiene.

[43]  G. Kuno,et al.  Low rates of antigen detection and virus isolation from the peripheral blood leukocytes of dengue fever patients. , 1985, The American journal of tropical medicine and hygiene.

[44]  A. Nisalak,et al.  Isolation of dengue viruses from peripheral blood leukocytes of patients with hemorrhagic fever. , 1980, The Journal of infectious diseases.

[45]  S. Halstead In vivo enhancement of dengue virus infection in rhesus monkeys by passively transferred antibody. , 1979, The Journal of infectious diseases.

[46]  SB Halstead,et al.  Dengue viruses and mononuclear phagocytes. I. Infection enhancement by non-neutralizing antibody , 1977, The Journal of experimental medicine.

[47]  A. Allison,et al.  Dengue viruses and mononuclear phagocytes. II. Identity of blood and tissue leukocytes supporting in vitro infection , 1977, The Journal of experimental medicine.

[48]  S. Halstead,et al.  Immunological enhancement of dengue virus replication. , 1973, Nature: New biology.

[49]  S. Halstead,et al.  RECENT EPIDEMICS OF HEMORRHAGIC FEVER IN THAILAND. OBSERVATIONS RELATED TO PATHOGENESIS OF A "NEW" DENGUE DISEASE. , 1965, American journal of public health and the nation's health.