Cardiac contractility modulation by non-excitatory electrical currents. The new frontier for electrical therapy of heart failure.
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V. Santinelli | C. Pappone | G. Vicedomini | G. Augello | G. Chierchia | P. Mazzone | S. Gulletta | Y. Mika | Francesco Maggi
[1] M. Böhm,et al. Sarcoplasmic reticulum Ca2+ATPase and phospholamban mRNA and protein levels in end-stage heart failure due to ischemic or dilated cardiomyopathy , 1996, Journal of Molecular Medicine.
[2] S. Ben‐Haim,et al. Global Improvement in Left Ventricular Performance Observed with Cardiac Contractility Modulation is the Result of Changes in Regional Contractility , 2004, Heart Failure Reviews.
[3] D. Burkhoff,et al. Electric Currents Applied During the Refractory Period Can Modulate Cardiac Contractility In Vitro and In Vivo , 2004, Heart Failure Reviews.
[4] S. Goodman,et al. Cardiac resynchronization and death from progressive heart failure: a meta-analysis of randomized controlled trials. , 2003, JAMA.
[5] S. Ben‐Haim,et al. Cardiac contractility modulation with nonexcitatory electric signals improves left ventricular function in dogs with chronic heart failure. , 2003, Journal of cardiac failure.
[6] O. Alfieri,et al. Cardiac contractility modulation by electric currents applied during the refractory period in patients with heart failure secondary to ischemic or idiopathic dilated cardiomyopathy. , 2002, The American journal of cardiology.
[7] Milton Packer,et al. Cardiac resynchronization in chronic heart failure. , 2002, The New England journal of medicine.
[8] M. Redfield,et al. Heart failure--an epidemic of uncertain proportions. , 2002, The New England journal of medicine.
[9] H. Drexler,et al. Carvedilol prospective randomized cumulative survival (COPERNICUS) trial: carvedilol as the sun and center of the beta-blocker world? , 2002, Circulation.
[10] D. DeMets,et al. Effect of Carvedilol on the Morbidity of Patients With Severe Chronic Heart Failure: Results of the Carvedilol Prospective Randomized Cumulative Survival (COPERNICUS) Study , 2002, Circulation.
[11] M. Pfeffer,et al. New Therapeutic Options in Congestive Heart Failure: Part II , 2002, Circulation.
[12] D. Burkhoff,et al. Cardiac contractility modulation by electric currents applied during the refractory period. , 2002, American journal of physiology. Heart and circulatory physiology.
[13] Marc A Pfeffer,et al. New therapeutic options in congestive heart failure: Part I. , 2002, Circulation.
[14] B. Swynghedauw,et al. What is wrong with positive inotropic drugs? Lessons from basic science and clinical trials , 2002 .
[15] D. DeMets,et al. Effect of carvedilol on survival in severe chronic heart failure. , 2001, The New England journal of medicine.
[16] J. Daubert,et al. Effects of multisite biventricular pacing in patients with heart failure and intraventricular conduction delay. , 2001, The New England journal of medicine.
[17] Arma,et al. EFFECTS OF MULTISITE BIVENTRICULAR PACING IN PATIENTS WITH HEART FAILURE AND INTRAVENTRICULAR CONDUCTION DELAY , 2001 .
[18] G. Isenberg. How can overexpression of Na(+),Ca(2+)-exchanger compensate the negative inotropic effects of downregulated SERCA? , 2001, Cardiovascular research.
[19] S. Houser,et al. The sarcoplasmic reticulum and the Na+/Ca2+ exchanger both contribute to the Ca2+ transient of failing human ventricular myocytes. , 1999, Circulation research.
[20] S. Lehnart,et al. Relationship between Na+-Ca2+-exchanger protein levels and diastolic function of failing human myocardium. , 1999, Circulation.
[21] L. Stevenson. Inotropic therapy for heart failure. , 1998, The New England journal of medicine.
[22] S. Houser,et al. Contribution of reverse-mode sodium-calcium exchange to contractions in failing human left ventricular myocytes. , 1998, Cardiovascular research.
[23] G Arnold,et al. Evidence for functional relevance of an enhanced expression of the Na(+)-Ca2+ exchanger in failing human myocardium. , 1996, Circulation.
[24] W. Giles,et al. Effects of action potential duration on excitation-contraction coupling in rat ventricular myocytes. Action potential voltage-clamp measurements. , 1995, Circulation research.
[25] H. Drexler,et al. Gene expression of the cardiac Na(+)-Ca2+ exchanger in end-stage human heart failure. , 1994, Circulation research.
[26] M. Movsesian,et al. Ca2+‐Transporting ATPase, Phospholamban, and Calsequestrin Levels in Nonfailing and Failing Human Myocardium , 1994 .
[27] M. Movsesian,et al. Ca(2+)-transporting ATPase, phospholamban, and calsequestrin levels in nonfailing and failing human myocardium. , 1994, Circulation.
[28] N. Alpert,et al. Alterations in sarcoplasmic reticulum gene expression in human heart failure. A possible mechanism for alterations in systolic and diastolic properties of the failing myocardium. , 1993, Circulation research.
[29] E H Wood,et al. Inotropic Effects of Electric Currents , 1969, Circulation research.