Caspofungin suppresses brain cell necroptosis in the ischemic stroke rat via up-regulation of Pellino3

[1]  Zhenhua Zhou,et al.  TGR5 activation attenuates neuroinflammation via Pellino3 inhibition of caspase-8/NLRP3 after middle cerebral artery occlusion in rats , 2020, Journal of Neuroinflammation.

[2]  I. Khan,et al.  A decade of cell death studies: Breathing new life into necroptosis. , 2020, Pharmacology & therapeutics.

[3]  H. Iwasaki,et al.  Caspofungin suppresses zymosan-induced cytokine and chemokine release in THP-1 cells: Possible involvement of the spleen tyrosine kinase pathway. , 2020, Translational research : the journal of laboratory and clinical medicine.

[4]  Haiwei Zhang,et al.  Catalytically inactive RIP1 and RIP3 deficiency protect against acute ischemic stroke by inhibiting necroptosis and neuroinflammation , 2020, Cell Death & Disease.

[5]  Xiu-ju Luo,et al.  Targeting the pathways of regulated necrosis: a potential strategy for alleviation of cardio-cerebrovascular injury , 2020, Cellular and Molecular Life Sciences.

[6]  D. Yavagal,et al.  Cell Death Pathways in Ischemic Stroke and Targeted Pharmacotherapy , 2020, Translational Stroke Research.

[7]  Junying Yuan,et al.  Sequential activation of necroptosis and apoptosis cooperates to mediate vascular and neural pathology in stroke , 2020, Proceedings of the National Academy of Sciences.

[8]  Jie Yang,et al.  Mitochondrial E3 ubiquitin ligase 1 promotes brain injury by disturbing mitochondrial dynamics in a rat model of ischemic stroke. , 2019, European journal of pharmacology.

[9]  F. Sun,et al.  Necrostatin-1 Prevents Necroptosis in Brains after Ischemic Stroke via Inhibition of RIPK1-Mediated RIPK3/MLKL Signaling , 2019, Aging and disease.

[10]  Jie Yang,et al.  Ligustroflavone reduces necroptosis in rat brain after ischemic stroke through targeting RIPK1/RIPK3/MLKL pathway , 2019, Naunyn-Schmiedeberg's Archives of Pharmacology.

[11]  Jie Yang,et al.  Ligustroflavone reduces necroptosis in rat brain after ischemic stroke through targeting RIPK1/RIPK3/MLKL pathway , 2019, Naunyn-Schmiedeberg's Archives of Pharmacology.

[12]  Joshua A. Salomon,et al.  GLOBAL, REGIONAL, AND COUNTRY-SPECIFIC LIFETIME RISK OF STROKE, 1990–2016 , 2018, The New England journal of medicine.

[13]  Junying Yuan,et al.  Necroptosis and RIPK1-mediated neuroinflammation in CNS diseases , 2018, Nature Reviews Neuroscience.

[14]  Jennifer Martinez,et al.  Programmed Necrosis and Disease:We interrupt your regular programming to bring you necroinflammation , 2018, Cell Death & Differentiation.

[15]  Liu Wen-wu,et al.  Necroptosis Signaling Pathways in Stroke: From Mechanisms to Therapies , 2018, Current neuropharmacology.

[16]  Giou-Teng Yiang,et al.  Current Mechanistic Concepts in Ischemia and Reperfusion Injury , 2018, Cellular Physiology and Biochemistry.

[17]  Jie Yang,et al.  Combination of Emricasan with Ponatinib Synergistically Reduces Ischemia/Reperfusion Injury in Rat Brain Through Simultaneous Prevention of Apoptosis and Necroptosis , 2018, Translational Stroke Research.

[18]  Meng Li,et al.  PTPN21 protects PC12 cell against oxygen‐glucose deprivation by activating cdk5 through ERK1/2 signaling pathway , 2017, European journal of pharmacology.

[19]  Sai Zhang,et al.  Hypoxia-inducible factor-1 alpha is involved in RIP-induced necroptosis caused by in vitro and in vivo ischemic brain injury , 2017, Scientific Reports.

[20]  Daniel S. Chertow,et al.  Necroptosis: Mechanisms and Relevance to Disease , 2017 .

[21]  D. Stevens,et al.  Caspofungin: Pharmacodynamics, pharmacokinetics, clinical uses and treatment outcomes , 2016, Critical reviews in microbiology.

[22]  J. Callanan,et al.  Pellino3 targets RIP1 and regulates the pro-apoptotic effects of TNF-α , 2013, Nature Communications.

[23]  A. Hickey,et al.  Stroke rehabilitation: recent advances and future therapies. , 2013, QJM : monthly journal of the Association of Physicians.