p16 gene transfer increases cell killing with abnormal nucleation after ionising radiation in glioma cells
暂无分享,去创建一个
F. Yamasaki | Y. Kajiwara | K. Kurisu | H. Tauchi | S. Matsuura | S. Hama | K. Arita | H. Yoshioka | Y. Heike | K. Mandai
[1] F. Yamasaki,et al. Staurosporine-induced apoptosis is independent of p16 and p21 and achieved via arrest at G2/M and at G1 in U251MG human glioma cell line , 2003, Cancer Chemotherapy and Pharmacology.
[2] G. Bernardi,et al. Curcumin Inhibits Activation of Vγ9Vδ2 T Cells by Phosphoantigens and Induces Apoptosis Involving Apoptosis-Inducing Factor and Large Scale DNA Fragmentation1 , 2001, The Journal of Immunology.
[3] V. N. Bhattathiri. Amitotic cell divisions and tumour growth: an alternative model for cell kinetic compartments in solid tumours. , 2001, Oral oncology.
[4] K. Yamato,et al. Osteoclast differentiation is associated with transient upregulation of cyclin‐dependent kinase inhibitors p21WAF1/CIP1 and p27KIP1 , 2001 .
[5] D. Delia,et al. The Forkhead-associated Domain of NBS1 Is Essential for Nuclear Foci Formation after Irradiation but Not Essential for hRAD50·hMRE11·NBS1 Complex DNA Repair Activity* , 2000, The Journal of Biological Chemistry.
[6] K. Yamato,et al. Osteoclast differentiation is associated with transient upregulation of cyclin-dependent kinase inhibitors p21(WAF1/CIP1) and p27(KIP1). , 2001, Journal of cellular biochemistry.
[7] J. Roth,et al. Adenovirus-mediated p16INK4a gene expression radiosensitizes non-small cell lung cancer cells in a p53-dependent manner , 2000, Oncogene.
[8] D. Shewach,et al. The role of cell cycle progression in radiosensitization by 2',2'-difluoro-2'-deoxycytidine. , 2000, Cancer research.
[9] H. Yokozaki,et al. Molecular characteristics of eight gastric cancer cell lines established in Japan , 2000, Pathology international.
[10] T. Nakamura,et al. Intercellular Adhesion Molecule 1 Discriminates Functionally Different Populations of Human Osteoblasts: Characteristic Involvement of Cell Cycle Regulators , 2000, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[11] H. Hwang,et al. Radiosensitivity of thymidylate synthase-deficient human tumor cells is affected by progression through the G1 restriction point into S-phase: implications for fluoropyrimidine radiosensitization. , 2000, Cancer research.
[12] B. Li,et al. Expression of p16 and CDK4 in oral premalignant lesions and oral squamous cell carcinomas: a semi-quantitative immunohistochemical study. , 2007, Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology.
[13] Fengzhi Li,et al. Pleiotropic cell-division defects and apoptosis induced by interference with survivin function , 1999, Nature Cell Biology.
[14] A. Beaudet,et al. Administration of helper-dependent adenoviral vectors and sequential delivery of different vector serotype for long-term liver-directed gene transfer in baboons. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[15] S. Pavey,et al. Ultraviolet radiation induces p16CDKN2A expression in human skin. , 1999, Cancer research.
[16] S. Sakaki,et al. Restoration of wild-type p16 down-regulates vascular endothelial growth factor expression and inhibits angiogenesis in human gliomas. , 1999, Cancer research.
[17] Peter Lichter,et al. Amplification and Expression of Cyclin D Genes (CCND1 CCND2 and CCND3) in Human Malignant Gliomas , 1999, Brain pathology.
[18] N. Khodarev,et al. Mechanisms of induction of apoptotic DNA fragmentation. , 2009, International journal of radiation biology.
[19] C. Miller,et al. Differential susceptibility of primary and established human glioma cells to adenovirus infection: targeting via the epidermal growth factor receptor achieves fiber receptor-independent gene transfer. , 1998, Cancer Research.
[20] N. Hayward,et al. Involvement of p16CDKN2A in cell cycle delays after low dose UV irradiation. , 1998, Mutation research.
[21] R. Prathapan,et al. Prediction of radiosensitivity of oral cancers by serial cytological assay of nuclear changes. , 1998, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.
[22] T. Nakajima,et al. Immunohistochemical overexpression of p16 protein associated with intact retinoblastoma protein expression in cervical cancer and cervical intraepithelial neoplasia , 1998, Pathology international.
[23] P. Remani,et al. Radiation-Induced Acute Immediate Nuclear Abnormalities in Oral Cancer Cells , 1998, Acta Cytologica.
[24] T. Tsuruo,et al. Up‐regulation of p27Kip1 Correlates Inversely with Anchorage‐independent Growth of Human Cancer Cell Lines , 1998, Japanese journal of cancer research : Gann.
[25] R. Naviaux,et al. Restoration of growth arrest by p16INK4, p21WAF1, pRB, and p53 is dependent on the integrity of the endogenous cell-cycle control pathways in human glioblastoma cell lines. , 1998, Experimental cell research.
[26] K. Kurisu,et al. Adenovirus-mediated p16 gene transfer prevents drug-induced cell death through G1 arrest in human glioma cells. , 1998, International journal of cancer.
[27] V. Baldin,et al. Involvement of p21 in mitotic exit after paclitaxel treatment in MCF-7 breast adenocarcinoma cell line , 1997, Oncogene.
[28] D. Tang,et al. Overexpression of adenovirus-encoded transgenes from the cytomegalovirus immediate early promoter in irradiated tumor cells. , 1997, Human gene therapy.
[29] G. Reifenberger,et al. Molecular genetic analysis of giant cell glioblastomas. , 1997, The American journal of pathology.
[30] T. McDonnell,et al. Characterization of p53 and p21 functional interactions in glioma cells en route to apoptosis. , 1997, Journal of the National Cancer Institute.
[31] K. Kurisu,et al. Transformation of human glioma cell lines with the p16 gene inhibits cell proliferation. , 1997, Anticancer research.
[32] J. Bartek,et al. Adenovirally transferred p16INK4/CDKN2 and p53 genes cooperate to induce apoptotic tumor cell death , 1997, Nature Medicine.
[33] C. Sherr. Cancer Cell Cycles , 1996, Science.
[34] J. Dickinson,et al. Accumulation of p16CDKN2A in response to ultraviolet irradiation correlates with late S-G(2)-phase cell cycle delay. , 1996, Cancer research.
[35] W. Yung,et al. Adenovirus-mediated transfer of the p53 gene produces rapid and generalized death of human glioma cells via apoptosis. , 1996, Cancer research.
[36] Y. Kanegae,et al. Efficient generation of recombinant adenoviruses using adenovirus DNA-terminal protein complex and a cosmid bearing the full-length virus genome. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[37] J. Bruner,et al. Adenovirus-mediated p16/CDKN2 gene transfer induces growth arrest and modifies the transformed phenotype of glioma cells. , 1996, Oncogene.
[38] D. Louis,et al. CDKN2/p16 or RB alterations occur in the majority of glioblastomas and are inversely correlated. , 1996, Cancer research.
[39] S. Teramoto,et al. Effect of adenoviral vector infection on cell proliferation in cultured primary human airway epithelial cells. , 1995, Human gene therapy.
[40] J. Bartek,et al. Retinoblastoma-protein-dependent cell-cycle inhibition by the tumour suppressor p16 , 1995, Nature.
[41] G. Reifenberger,et al. CDKN2 (p16/MTS1) gene deletion or CDK4 amplification occurs in the majority of glioblastomas. , 1994, Cancer research.
[42] C. James,et al. CDK4 amplification is an alternative mechanism to p16 gene homozygous deletion in glioma cell lines. , 1994, Cancer research.
[43] D. Carson,et al. Deletions of the cyclin-dependent kinase-4 inhibitor gene in multiple human cancers , 1994, Nature.
[44] J. R. Smith,et al. Cloning of senescent cell-derived inhibitors of DNA synthesis using an expression screen. , 1994, Experimental cell research.
[45] David Beach,et al. p21 is a universal inhibitor of cyclin kinases , 1993, Nature.
[46] J. Trent,et al. WAF1, a potential mediator of p53 tumor suppression , 1993, Cell.
[47] Yamamura Ken-ichi,et al. Efficient selection for high-expression transfectants with a novel eukaryotic vector , 1991 .
[48] H. Niwa,et al. Efficient selection for high-expression transfectants with a novel eukaryotic vector. , 1991, Gene.
[49] P. Burger,et al. Correlations between cytologic composition and biologic behavior in the glioblastoma multiforme. A postmortem study of 50 cases , 1983, Cancer.
[50] L. Tolmach,et al. Variations in several responses of HeLa cells to x-irradiation during the division cycle. , 1963, Biophysical journal.