Scavenging system developed for the Magill anesthetic circuit for use in the dental office.

Numerous potential problems have been associated with long term or occupational exposure to both nitrous oxide and halothane. Despite the lack of firmly established cause-and-effect relationships, particularly in humans, it would seem prudent to use techniques that minimize operator exposure. With this in mind, a scavenging system for use in both conscious sedation and general anesthetic techniques was developed which fulfills the requirements of both general dentists as well as those administering general anesthesia. This paper describes this system and its adaptation to the commonly used Magill circuit. It also briefly reviews the factors involved in potential toxicity caused by long term exposure to nitrous oxide and halothane.

[1]  L. Kaufman.  Complications of Sedation and Anaesthesia in Dentistry , 1989 .

[2]  A. Aynsley-Green,et al.  Does halothane anaesthesia decrease the metabolic and endocrine stress responses of newborn infants undergoing operation? , 1988, British medical journal.

[3]  W. Dick,et al.  Prolonged anaesthesia with isoflurane and halothane , 1988, Anaesthesia.

[4]  E. R. Young,et al.  Potential problems associated with occupational exposure to nitrous oxide. , 1988, Journal.

[5]  T. Pallasch.  Principles of pharmacotherapy: III. Drug allergy. , 1988, Anesthesia progress.

[6]  J. Neuberger,et al.  Specific antibodies to halothane-induced liver antigens in halothane-associated hepatitis. , 1987, British journal of anaesthesia.

[7]  B. Husum.  Mutagenicity of inhalation anaesthetics studied by the sister chromatid exchange test in lymphocytes of patients and operating room personnel. , 1987, Danish medical bulletin.

[8]  G. Cooper,et al.  Comparison of isoflurane and halothane in outpatient paediatric dental anaesthesia. , 1987, British journal of anaesthesia.

[9]  M. O'Halloran,et al.  Prospective study of liver function in children following multiple halothane anaesthetics at short intervals. , 1986, British journal of anaesthesia.

[10]  S. Loft,et al.  Increased hepatic microsomal activity after halothane anaesthesia in children , 1986, Anaesthesia.

[11]  G. Woods,et al.  Reversible inhibition of natural killer cell activity by volatile anaesthetic agents in vitro. , 1986, British journal of anaesthesia.

[12]  H. Kingston.  Halothane and Isoflurane Anesthesia in Pediatric Outpatients , 1986, Anesthesia and analgesia.

[13]  M. Murray,et al.  Halothane hepatitis. Detection of a constitutional susceptibility factor. , 1985, The New England journal of medicine.

[14]  R. Varma,et al.  Halothane hepatitis without halothane: Role of inapparent circuit contamination and its prevention , 1985, Hepatology.

[15]  J. Nunn,et al.  Nitrous oxide decreases motility of human neutrophils in vitro. , 1982, Anesthesiology.

[16]  B. Brown,et al.  Exposure to Nitrous Oxide and Neurologic Disease among Dental Professionals , 1981, Anesthesia and analgesia.

[17]  B. Brown,et al.  Occupational disease in dentistry and chronic exposure to trace anesthetic gases. , 1981, Journal of the American Dental Association.

[18]  A Baraka,et al.  Functional Classification of Anaesthesia Circuits , 1977, Middle East journal of anaesthesiology.

[19]  E. Eger,et al.  Comparative toxicities of enflurane, fluroxene and nitrous oxide at subanaesthetic concentrations in laboratory animals , 1977, Canadian Anaesthetists' Society journal.

[20]  K. Balogh,et al.  Testicular Reaction to Prolonged Exposure to Nitrous Oxide , 1976, Anesthesiology.

[21]  J. Nunn,et al.  Fresh gas economics of the Magill circuit. , 1968, Anesthesiology.

[22]  W W MAPLESON,et al.  The elimination of rebreathing in various semi-closed anaesthetic systems. , 1954, British journal of anaesthesia.