Patients with IgA nephropathy have increased serum galactose-deficient IgA1 levels.

[1]  E. Haddad,et al.  Glycosylation and size of IgA1 are essential for interaction with mesangial transferrin receptor in IgA nephropathy. , 2004, Journal of the American Society of Nephrology : JASN.

[2]  J. Barratt,et al.  Mesangial IgA1 in IgA nephropathy exhibits aberrant O-glycosylation: observations in three patients. , 2001, Kidney international.

[3]  B. Julian,et al.  Galactose-deficient IgA1 in sera of IgA nephropathy patients is present in complexes with IgG. , 1997, Kidney international.

[4]  G. Carpenter,et al.  O-glycosylation of salivary IgA as determined by lectin analysis. , 1997, Biochemical Society transactions.

[5]  B. Julian,et al.  Interactions of human mesangial cells with IgA and IgA-containing immune complexes. , 2002, Kidney international.

[6]  W. Haynes,et al.  IgA nephropathy: a syndrome of uniform morphology, diverse clinical features and uncertain prognosis. , 1977, Clinical nephrology.

[7]  B. Julian,et al.  IgA nephropathy, the most common glomerulonephritis worldwide. A neglected disease in the United States? , 1988, The American journal of medicine.

[8]  B. Julian,et al.  Circulating immune complexes and immunoglobulin A rheumatoid factor in patients with mesangial immunoglobulin A nephropathies. , 1986, The Journal of clinical investigation.

[9]  Y. Hiki,et al.  Underglycosylation of IgA1 hinge plays a certain role for its glomerular deposition in IgA nephropathy. , 1999, Journal of the American Society of Nephrology : JASN.

[10]  Y. Hiki,et al.  O-Linked Oligosaccharide on IgA1 Hinge Region in IgA Nephropathy1 , 1995 .

[11]  B. Julian,et al.  IgA1-containing immune complexes in IgA nephropathy differentially affect proliferation of mesangial cells. , 2005, Kidney international.

[12]  R. Sinniah.  Occurrence of mesangial IgA and IgM deposits in a control necropsy population. , 1983, Journal of clinical pathology.

[13]  S. Harper,et al.  Galactosylation of N‐ and O‐linked carbohydrate moieties of IgA1 and IgG in IgA nephropathy , 1995, Clinical and experimental immunology.

[14]  J. Mestecky,et al.  The human IgA system: a reassessment. , 1986, Clinical immunology and immunopathology.

[15]  B. Julian,et al.  Complement activation in IgA nephropathy. , 1987, Kidney international.

[16]  D. Droz.  Natural History of Primary Glomerulonephritis with Mesangial Deposits of IgA , 1976 .

[17]  J. Feehally,et al.  Abnormal IgA glycosylation in Henoch-Schönlein purpura restricted to patients with clinical nephritis. , 1998, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[18]  A. Michael,et al.  Selective deposition of immunoglobulin A1 in immunoglobulin A nephropathy, anaphylactoid purpura nephritis, and systemic lupus erythematosus. , 1980, The Journal of clinical investigation.

[19]  Ming-hui Zhao,et al.  Aberrantly glycosylated serum IgA1 are closely associated with pathologic phenotypes of IgA nephropathy. , 2005, Kidney international.

[20]  F. Schena,et al.  Increased serum levels of IgA1-IgG immune complexes and anti-F(ab')2 antibodies in patients with primary IgA nephropathy. , 1989, Clinical and experimental immunology.

[21]  G. Mazzucco,et al.  IgA1 and IgA2 immune complexes in primary IgA nephropathy and Henoch-Schönlein nephritis. , 1984, Clinical and experimental immunology.

[22]  R. Coppo,et al.  Aberrant glycosylation in IgA nephropathy (IgAN). , 2004, Kidney international.

[23]  G. Mazzucco,et al.  Circulating immune complexes containing IgA, IgG and IgM in patients with primary IgA nephropathy and with Henoch-Schoenlein nephritis. Correlation with clinical and histologic signs of activity. , 1982, Clinical nephrology.

[24]  D. Chevet,et al.  Impairment of jacalin binding to serum IgA in IgA nephropathy , 1990, Journal of clinical laboratory analysis.

[25]  Y. Hiki,et al.  O-linked oligosaccharide on IgA1 hinge region in IgA nephropathy. Fundamental study for precise structure and possible role. , 1995, Contributions to nephrology.

[26]  A. Levey,et al.  A More Accurate Method To Estimate Glomerular Filtration Rate from Serum Creatinine: A New Prediction Equation , 1999, Annals of Internal Medicine.

[27]  J. Wieslander,et al.  Serum IgA-Fibronectin Aggregates in Patients With IgA Nephropathy and Henoch-Schiinlein Purpura: Diagnostic Value and Pathogenic Implications , 1991 .

[28]  J. Feehally,et al.  New insights into the pathogenesis of IgA nephropathy , 2003, Springer Seminars in Immunopathology.

[29]  B. Julian,et al.  Defective galactosylation and clearance of IgA1 molecules as a possible etiopathogenic factor in IgA nephropathy. , 1993, Contributions to nephrology.

[30]  D. Gómez-Garre,et al.  Potential factors governing extracellular matrix production by mesangial cells: their relevance for the pathogenesis of IgA nephropathy. , 1995, Contributions to nephrology.

[31]  Ann Chen,et al.  Pathogenesis of IgA nephropathy: In vitro activation of human mesangial cells by IgA immune complex leads to cytokine secretion , 1994, The Journal of pathology.

[32]  F. Berthoux,et al.  Different glycosylation profile of serum IgA1 in IgA nephropathy according to the glomerular basement membrane thickness: normal versus thin. , 2003, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[33]  B. Julian,et al.  Reactivities of N-acetylgalactosamine-specific lectins with human IgA1 proteins. , 2007, Molecular immunology.

[34]  L. Peruzzi,et al.  Glycosylation of circulating IgA in patients with IgA nephropathy modulates proliferation and apoptosis of mesangial cells. , 2001, Journal of the American Society of Nephrology : JASN.

[35]  O. Hashim,et al.  THE INTERACTION OF SELECTIVE PLANT LECTINS WITH NEURAMINIDASE-TREATED AND UNTREATED IgA1 FROM THE SERA OF IgA NEPHROPATHY PATIENTS , 2001, Immunological investigations.

[36]  P. V. D. van der Boog,et al.  An increased polymeric IgA level is not a prognostic marker for progressive IgA nephropathy. , 2004, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[37]  B. Julian,et al.  Progress in Molecular and Genetic Studies of IgA Nephropathy , 2001, Journal of Clinical Immunology.

[38]  J. Egido,et al.  Receptors for immune complexes activate gene expression and synthesis of matrix proteins in cultured rat and human mesangial cells: role of TGF-beta. , 1996, Journal of immunology.

[39]  B. Julian,et al.  Circulating immune complexes in IgA nephropathy consist of IgA1 with galactose-deficient hinge region and antiglycan antibodies. , 1999, The Journal of clinical investigation.

[40]  J. Floege,et al.  IgA nephropathy: recent developments. , 2000, Journal of the American Society of Nephrology : JASN.

[41]  B. Julian,et al.  Immunoglobulin A-fibronectin aggregate levels in children and adults with immunoglobulin A nephropathy. , 1993, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[42]  H. Oksa,et al.  Immunoglobulin and complement deposition in glomeruli of 756 subjects who had committed suicide or met with a violent death. , 1993, Journal of clinical pathology.

[43]  B. Julian,et al.  IgA nephropathy: an update , 2004, Current opinion in nephrology and hypertension.

[44]  Y. Hiki,et al.  Mass spectrometry proves under-O-glycosylation of glomerular IgA1 in IgA nephropathy. , 2001, Kidney international.

[45]  M. Rambausek,et al.  Frequency of mesangial IgA deposits in a non-selected autopsy series. , 1989, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[46]  Y. Yamaguchi,et al.  Incidence of latent mesangial IgA deposition in renal allograft donors in Japan. , 2003, Kidney international.

[47]  H. Rosenberg,et al.  Morphological findings in 70 kidneys of living donors for renal transplant. , 1990, Pathology, research and practice.

[48]  J. Floege,et al.  Activated complement C3: a potentially novel predictor of progressive IgA nephropathy. , 1997, Kidney international.

[49]  M. Haas,et al.  Histologic subclassification of IgA nephropathy: a clinicopathologic study of 244 cases. , 1997, American journal of kidney diseases : the official journal of the National Kidney Foundation.