Pathology of the Vulnerable Plaque
暂无分享,去创建一个
R. Virmani | F. Kolodgie | A. Farb | A. Burke
[1] R. Virmani,et al. Phenotypic Modulation of Intima and Media Smooth Muscle Cells in Fatal Cases of Coronary Artery Lesion , 2005 .
[2] F. Cipollone,et al. Factors Associated with Apoptosis in Symptomatic and Asymptomatic Carotid Atherosclerotic Plaques , 2005, International journal of immunopathology and pharmacology.
[3] F. Cipollone,et al. Balance Between PGD Synthase and PGE Synthase Is a Major Determinant of Atherosclerotic Plaque Instability in Humans , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[4] Renu Virmani,et al. Intraplaque hemorrhage and progression of coronary atheroma. , 2003, The New England journal of medicine.
[5] M. Daemen,et al. Atherosclerotic plaque rupture: local or systemic process? , 2003, Arteriosclerosis, thrombosis, and vascular biology.
[6] A. Dyer,et al. Major Risk Factors as Antecedents of Fatal and Nonfatal Coronary Heart Disease Events , 2003 .
[7] R. Virmani,et al. 34th Bethesda Conference: Task force #2--What is the pathologic basis for new atherosclerosis imaging techniques? , 2003, Journal of the American College of Cardiology.
[8] Renu Virmani,et al. Pathology of the thin-cap fibroatheroma: a type of vulnerable plaque. , 2003, Journal of interventional cardiology.
[9] D. Kass,et al. Wall Tension Is a Potent Negative Regulator of In Vivo Thrombomodulin Expression , 2003, Circulation Research.
[10] Renu Virmani,et al. Vulnerable plaque: the pathology of unstable coronary lesions. , 2002, Journal of interventional cardiology.
[11] Y. Nemerson. A simple experiment and a weakening paradigm: the contribution of blood to propensity for thrombus formation. , 2002, Arteriosclerosis, thrombosis, and vascular biology.
[12] Paul M. Ridker,et al. Prospective Study of C-Reactive Protein, Homocysteine, and Plasma Lipid Levels as Predictors of Sudden Cardiac Death , 2002, Circulation.
[13] R. Virmani,et al. Elevated C-Reactive Protein Values and Atherosclerosis in Sudden Coronary Death: Association With Different Pathologies , 2002, Circulation.
[14] R. Virmani,et al. Role of circulating myeloperoxidase positive monocytes and neutrophils in occlusive coronary thrombi , 2002 .
[15] R. Virmani,et al. The thin-cap fibroatheroma: a type of vulnerable plaque: The major precursor lesion to acute coronary syndromes , 2001, Current opinion in cardiology.
[16] Renu Virmani,et al. Pathophysiology of Calcium Deposition in Coronary Arteries , 2001, Herz.
[17] P. Libby,et al. Macrophage myeloperoxidase regulation by granulocyte macrophage colony-stimulating factor in human atherosclerosis and implications in acute coronary syndromes. , 2001, The American journal of pathology.
[18] P. Libby. Coronary artery injury and the biology of atherosclerosis: inflammation, thrombosis, and stabilization. , 2000, The American journal of cardiology.
[19] R. Virmani,et al. Platelet-rich intramyocardial thromboemboli are frequent in acute coronary thrombosis, especially plaque erosions , 2000 .
[20] R. Virmani,et al. Lessons from sudden coronary death: a comprehensive morphological classification scheme for atherosclerotic lesions. , 2000, Arteriosclerosis, thrombosis, and vascular biology.
[21] J. Badimón,et al. The role of plaque rupture and thrombosis in coronary artery disease. , 2000, Atherosclerosis.
[22] R. Virmani,et al. Coronary calcification: insights from sudden coronary death victims , 2000, Zeitschrift für Kardiologie.
[23] R. Virmani,et al. Plaque erosion is a major substrate for coronary thrombosis in acute myocardial infarction , 1998, Heart.
[24] R. Virmani,et al. Effect of risk factors on the mechanism of acute thrombosis and sudden coronary death in women. , 1998, Circulation.
[25] R. Virmani,et al. Coronary risk factors and plaque morphology in men with coronary disease who died suddenly. , 1997, The New England journal of medicine.
[26] C. Depré,et al. Neovascularization in human coronary atherosclerotic lesions. , 1996, Catheterization and cardiovascular diagnosis.
[27] R. Virmani,et al. Coronary plaque erosion without rupture into a lipid core. A frequent cause of coronary thrombosis in sudden coronary death. , 1996, Circulation.
[28] R. Virmani,et al. Sudden coronary death. Frequency of active coronary lesions, inactive coronary lesions, and myocardial infarction. , 1995, Circulation.
[29] V. Fuster,et al. Coronary plaque disruption. , 1995, Circulation.
[30] P. Fitzgerald,et al. Detection of intralesional calcium by intracoronary ultrasound depends on the histologic pattern. , 1994, American heart journal.
[31] Robertag . Williams,et al. 25th Bethesda Conference: Future Personnel Needs for Cardiovascular Health Care. November 15-16, 1993. , 1994, Journal of the American College of Cardiology.
[32] W D Wagner,et al. A definition of initial, fatty streak, and intermediate lesions of atherosclerosis. A report from the Committee on Vascular Lesions of the Council on Arteriosclerosis, American Heart Association. , 1994, Circulation.
[33] W. Roberts,et al. Morphologic comparison of frequency and types of acute lesions in the major epicardial coronary arteries in unstable angina pectoris, sudden coronary death and acute myocardial infarction. , 1991, Journal of the American College of Cardiology.
[34] W. Roberts,et al. Morphometric analysis of the composition of coronary arterial plaques in isolated unstable angina pectoris with pain at rest. , 1990, The American journal of cardiology.
[35] E. Falk. Morphologic features of unstable atherothrombotic plaques underlying acute coronary syndromes. , 1989, The American journal of cardiology.
[36] M J Davies,et al. Plaque fissuring--the cause of acute myocardial infarction, sudden ischaemic death, and crescendo angina. , 1985, British heart journal.
[37] M J Davies,et al. Pathological view of sudden cardiac death. , 1981, British heart journal.
[38] W. Roberts,et al. Quantitation of coronary arterial narrowing at necropsy in sudden coronary death: analysis of 31 patients and comparison with 25 control subjects. , 1979, The American journal of cardiology.