Toll-like receptors in wound healing: location, accessibility, and timing.

[1]  Xiaoping Yang,et al.  Toll-like receptor 3 ligand polyinosinic:polycytidylic acid promotes wound healing in human and murine skin. , 2012, The Journal of investigative dermatology.

[2]  M. Yamashita,et al.  The authors state no conflict of interest. , 2012 .

[3]  K. Mills,et al.  TLR based therapeutics. , 2011, Current opinion in pharmacology.

[4]  B. Holst,et al.  Characterization of murine melanocortin receptors mediating adipocyte lipolysis and examination of signalling pathways involved , 2011, Molecular and Cellular Endocrinology.

[5]  Sangdun Choi,et al.  Similar Structures but Different Roles – An Updated Perspective on TLR Structures , 2011, Front. Physio..

[6]  E. Tredget,et al.  Toll‐like receptors expressed by dermal fibroblasts contribute to hypertrophic scarring , 2011, Journal of cellular physiology.

[7]  J. Daly Accelerated wound healing mediated by activation of Toll-like receptor 9 , 2011 .

[8]  A. Di Nardo,et al.  Plasmacytoid dendritic cells sense skin injury and promote wound healing through type I interferons , 2010, The Journal of experimental medicine.

[9]  L. Lavery,et al.  Erratum: the costs of diabetic foot: The economic case for the limb salvage team (Journal of Vascular Surgery (2010) 52:3 SUPPL. (17S-22S)) , 2010 .

[10]  Takashi Sato,et al.  Accelerated wound healing mediated by activation of Toll‐like receptor 9 , 2010, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.

[11]  L. Lavery,et al.  The costs of diabetic foot: the economic case for the limb salvage team. , 2010, Journal of vascular surgery.

[12]  I. Jialal,et al.  TLR2 expression and signaling-dependent inflammation impair wound healing in diabetic mice , 2010, Laboratory Investigation.

[13]  K. Harding,et al.  Non-healing is associated with persistent stimulation of the innate immune response in chronic venous leg ulcers. , 2010, Journal of dermatological science.

[14]  Philippe Froguel,et al.  Variants in ADCY5 and near CCNL1 are associated with fetal growth and birth weight , 2010, Nature Genetics.

[15]  C. Bertolotto,et al.  Fifteen‐year quest for microphthalmia‐associated transcription factor target genes , 2010, Pigment cell & melanoma research.

[16]  Jorge Amador Berlanga Acosta,et al.  The pro‐inflammatory environment in recalcitrant diabetic foot wounds , 2008, International wound journal.

[17]  L. O’Neill,et al.  Signalling adaptors used by Toll-like receptors: an update. , 2008, Cytokine.

[18]  T. Luger,et al.  -Melanocyte-Stimulating Hormone and Related Tripeptides: Biochemistry, Antiinflammatory and Protective Effects in Vitro and in Vivo, and Future Perspectives for the Treatment of Immune-Mediated Inflammatory Diseases , 2008 .

[19]  S. Yuan,et al.  TOLL-LIKE RECEPTOR 4 CONTRIBUTES TO MICROVASCULAR INFLAMMATION AND BARRIER DYSFUNCTION IN THERMAL INJURY , 2007, Shock.

[20]  A. Sher,et al.  Cooperation of Toll-like receptor signals in innate immune defence , 2007, Nature Reviews Immunology.

[21]  Y. Helfrich,et al.  Injury enhances TLR2 function and antimicrobial peptide expression through a vitamin D-dependent mechanism. , 2007, The Journal of clinical investigation.

[22]  L. Miller,et al.  Toll-like receptors in the skin , 2007, Seminars in Immunopathology.

[23]  J. Nadel,et al.  Pseudomonas Lipopolysaccharide Accelerates Wound Repair via Activation of a Novel Epithelial Cell Signaling Cascade1 , 2006, The Journal of Immunology.

[24]  T. Billiar,et al.  EMERGING PARADIGM: TOLL-LIKE RECEPTOR 4-SENTINEL FOR THE DETECTION OF TISSUE DAMAGE , 2006, Shock.

[25]  J. Roth,et al.  Fever and circulating cytokines induced by double‐stranded RNA in guinea pigs: dependence on the route of administration and effects of repeated injections , 2006, Acta physiologica.

[26]  J. Barsig,et al.  The macrophage‐activating lipopeptide‐2 accelerates wound healing in diabetic mice , 2004, Experimental dermatology.

[27]  Zhenghong Yuan,et al.  Toll‐like receptor‐9 induced by physical trauma mediates release of cytokines following exposure to CpG motif in mouse skin , 2003, Immunology.

[28]  R. Sturm Skin colour and skin cancer - MC1R, the genetic link. , 2002, Melanoma research.

[29]  J. Nadeau,et al.  Pigmentation phenotypes of variant extension locus alleles result from point mutations that alter MSH receptor function , 1993, Cell.