Histone deacetylase-2 is a key regulator of diabetes- and transforming growth factor-beta1-induced renal injury.
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Ji Yeon Seo | Hyunjin Noh | H. Ha | Hi-Bahl Lee | M. Yu | J. Seo | H. Noh | Eun Young Oh | Mi Ra Yu | Young Ok Kim | Hunjoo Ha | Hi Bahl Lee | Y. O. Kim | Eunseo Oh
[1] J. H. Oh,et al. Sequential effects of high glucose on mesangial cell transforming growth factor-beta 1 and fibronectin synthesis. , 1998, Kidney international.
[2] Chun Li Zhang,et al. Class II Histone Deacetylases Act as Signal-Responsive Repressors of Cardiac Hypertrophy , 2002, Cell.
[3] K. Sharma,et al. The renal TGF-beta system in the db/db mouse model of diabetic nephropathy. , 1998, Experimental nephrology.
[4] L. Gaudreau,et al. High levels of oxidative stress globally inhibit gene transcription and histone acetylation. , 2006, DNA and cell biology.
[5] B. Groner,et al. The histone deacetylase inhibitor valproic acid selectively induces proteasomal degradation of HDAC2 , 2003, The EMBO journal.
[6] D. Schuppan,et al. Trichostatin A, a Histone Deacetylase Inhibitor, Suppresses Collagen Synthesis and Prevents TGF-β1-Induced Fibrogenesis in Skin Fibroblasts , 2002 .
[7] C. Nast,et al. Expression of transforming growth factor-β isoforms in human glomerular diseases , 1996 .
[8] J. Davie,et al. The Transcriptional Repressor Sp3 Is Associated with CK2-phosphorylated Histone Deacetylase 2* , 2002, The Journal of Biological Chemistry.
[9] I. G. Fantus,et al. Hyperglycemia-induced mitochondrial superoxide overproduction activates the hexosamine pathway and induces plasminogen activator inhibitor-1 expression by increasing Sp1 glycosylation. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[10] Y. Akai,et al. Quantification of glomerular TGF-beta 1 mRNA in patients with diabetes mellitus. , 1996, Kidney international.
[11] M. Bitzer,et al. TGF-beta signaling in renal disease. , 2002, Journal of the American Society of Nephrology : JASN.
[12] J. B. Kim,et al. Hypoxia inhibits adipocyte differentiation in a HDAC-independent manner. , 2005, Biochemical and biophysical research communications.
[13] Ping Zhu,et al. The histone deacetylase inhibitor valproic acid selectively induces proteasomal degradation of HDAC2 , 2003, The EMBO journal.
[14] D. Schuppan,et al. A histone deacetylase inhibitor, trichostatin A, suppresses myofibroblastic differentiation of rat hepatic stellate cells in primary culture , 1999, Hepatology.
[15] K. Sharma,et al. Long-term prevention of renal insufficiency, excess matrix gene expression, and glomerular mesangial matrix expansion by treatment with monoclonal antitransforming growth factor-beta antibody in db/db diabetic mice. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[16] C. Nast,et al. Expression of transforming growth factor-beta isoforms in human glomerular diseases. , 1996, Kidney international.
[17] Hong-Jian Zhu,et al. Smad7 inhibits fibrotic effect of TGF-Beta on renal tubular epithelial cells by blocking Smad2 activation. , 2002, Journal of the American Society of Nephrology : JASN.
[18] Ivan V. Gregoretti,et al. Molecular evolution of the histone deacetylase family: functional implications of phylogenetic analysis. , 2004, Journal of molecular biology.
[19] C. Allis,et al. Histone methylation versus histone acetylation: new insights into epigenetic regulation. , 2001, Current opinion in cell biology.
[20] C. Heldin,et al. TGF-(beta) type I receptor/ALK-5 and Smad proteins mediate epithelial to mesenchymal transdifferentiation in NMuMG breast epithelial cells. , 1999, Journal of cell science.
[21] E. Bottinger,et al. TGF-β signaling in renal disease , 2002 .
[22] K. H. Kim,et al. Melatonin and taurine reduce early glomerulopathy in diabetic rats. , 1999, Free radical biology & medicine.
[23] M. Steffes,et al. Structural-functional relationships in diabetic nephropathy. , 1984, The Journal of clinical investigation.
[24] K. Thai,et al. Expression of transforming growth factor-beta 1 during diabetic renal hypertrophy. , 1994, Kidney international.
[25] T. Fujita,et al. Inhibition of histone deacetylase activity suppresses epithelial-to-mesenchymal transition induced by TGF-beta1 in human renal epithelial cells. , 2007, Journal of the American Society of Nephrology : JASN.
[26] Jia Guo,et al. Neutralization of TGF-β by Anti-TGF-β Antibody Attenuates Kidney Hypertrophy and the Enhanced Extracellular Matrix Gene Expression in STZ-Induced Diabetic Mice , 1996, Diabetes.
[27] Jia Guo,et al. The Renal TGF-β System in the db/db Mouse Model of Diabetic Nephropathy , 1998, Nephron Experimental Nephrology.
[28] S. Berger,et al. Histone modifications in transcriptional regulation. , 2002, Current opinion in genetics & development.
[29] M. Jeong,et al. Inhibition of Histone Deacetylation Blocks Cardiac Hypertrophy Induced by Angiotensin II Infusion and Aortic Banding , 2005, Circulation.
[30] I. Adcock,et al. Decreased histone deacetylase activity in chronic obstructive pulmonary disease. , 2005, The New England journal of medicine.
[31] Y. Akai,et al. Quantification of glomerular TGF-β1 mRNA in patients with diabetes mellitus , 1996 .
[32] Sang Gyun Kim,et al. Class I Histone Deacetylase-Selective Novel Synthetic Inhibitors Potently Inhibit Human Tumor Proliferation , 2004, Clinical Cancer Research.
[33] H. Ha,et al. Reactive oxygen species amplify protein kinase C signaling in high glucose-induced fibronectin expression by human peritoneal mesothelial cells. , 2004, Kidney international.
[34] Junwei Yang,et al. Dissection of key events in tubular epithelial to myofibroblast transition and its implications in renal interstitial fibrosis. , 2001, The American journal of pathology.
[35] S. Uh,et al. Role of Reactive Oxygen Species in TGF-β1-Induced Mitogen-Activated Protein Kinase Activation and Epithelial-Mesenchymal Transition in Renal Tubular Epithelial Cells , 2005 .
[36] P. London. Injury , 1969, Definitions.
[37] E. Ballestar,et al. Snail Mediates E-Cadherin Repression by the Recruitment of the Sin3A/Histone Deacetylase 1 (HDAC1)/HDAC2 Complex , 2004, Molecular and Cellular Biology.
[38] I. Morita,et al. Protein kinase CK2 is a key activator of histone deacetylase in hypoxia‐associated tumors , 2008, International journal of cancer.
[39] K. Sharma,et al. Renal hypertrophy is associated with upregulation of TGF-beta 1 gene expression in diabetic BB rat and NOD mouse. , 1994, The American journal of physiology.
[40] E Ruoslahti,et al. Expression of transforming growth factor beta is elevated in human and experimental diabetic nephropathy. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[41] E. Olson,et al. Suppression of Class I and II Histone Deacetylases Blunts Pressure-Overload Cardiac Hypertrophy , 2006, Circulation.
[42] Jessica E. Bolden,et al. Anticancer activities of histone deacetylase inhibitors , 2006, Nature Reviews Drug Discovery.
[43] P. Wade. Transcriptional control at regulatory checkpoints by histone deacetylases: molecular connections between cancer and chromatin. , 2001, Human molecular genetics.
[44] Y. Tomino,et al. mRNA Expression of Growth Factors in Glomeruli From Diabetic Rats , 1993, Diabetes.
[45] E. Lobenhofer,et al. Short-chain fatty acids enhance nuclear receptor activity through mitogen-activated protein kinase activation and histone deacetylase inhibition. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[46] D. Brenner,et al. Hepatitis C virus–induced oxidative stress suppresses hepcidin expression through increased histone deacetylase activity , 2008, Hepatology.
[47] K. Thai,et al. Expression of transforming growth factor-β1 during diabetic renal hypertrophy , 1994 .
[48] J. Meinkoth,et al. Histone Deacetylase Is a Target of Valproic Acid-Mediated Cellular Differentiation , 2004, Cancer Research.
[49] J. Y. Lee,et al. Synthesis and biological evaluation of 3-(4-substituted-phenyl)-N-hydroxy-2-propenamides, a new class of histone deacetylase inhibitors. , 2003, Journal of medicinal chemistry.
[50] Y. Kaneda,et al. Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage , 2000, Nature.
[51] E. Seto,et al. Regulation of Histone Deacetylase 2 by Protein Kinase CK2* , 2002, The Journal of Biological Chemistry.