Analysis of antigen-driven positive and negative selection of phosphocholine-specific bone marrow B-cells.

[1]  A. Rolink,et al.  Surrogate light chain in B cell development. , 1993, Advances in immunology.

[2]  D. Longo,et al.  B cells from M167 mu kappa transgenic mice fail to proliferate after stimulation with soluble anti-Ig antibodies. A model for antigen-induced B cell anergy. , 1994, Journal of immunology.

[3]  S. Clarke,et al.  Development of B-1 cells: segregation of phosphatidyl choline-specific B cells to the B-1 population occurs after immunoglobulin gene expression , 1994, The Journal of experimental medicine.

[4]  F. Alt,et al.  The expression of Vpre-B/λ5 surrogate light chain in early bone marrow precursor B cells of normal and B cell-deficient mutant mice , 1994, Cell.

[5]  F. Melchers,et al.  Influence of surrogate L chain on DHJH-reading frame 2 suppression in mouse precursor B cells. , 1994, International immunology.

[6]  S. Clarke,et al.  VH CDR3‐dependent positive selection of murine VH12‐expressing B cells in the neonate , 1993, European journal of immunology.

[7]  D. Spencer,et al.  Mechanisms that limit the diversity of antibody: three sequentially acting mechanisms that favor the spontaneous production of germline encoded anti-phosphatidyl choline. , 1993, International immunology.

[8]  T. Honjo,et al.  The bcl-2 gene product inhibits clonal deletion of self-reactive B lymphocytes in the periphery but not in the bone marrow , 1993, The Journal of experimental medicine.

[9]  R. Hardy,et al.  The regulated expression of B lineage associated genes during B cell differentiation in bone marrow and fetal liver , 1993, The Journal of experimental medicine.

[10]  C. O'Connell,et al.  Antigen binding and idiotype analysis of antibodies obtained after electroporation of heavy and light chain genes encoding phosphocholine- specific antibodies: a model for T15-idiotype dominance , 1992, The Journal of experimental medicine.

[11]  J C Reed,et al.  Differential effects of Bcl-2 on T and B cells in transgenic mice. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[12]  G. Bishop,et al.  Differential structure-function requirements of the transmembranal domain of the B cell antigen receptor , 1992, The Journal of experimental medicine.

[13]  John Calvin Reed,et al.  Inhibition of thymocyte apoptosis and negative antigenic selection in bcl-2 transgenic mice. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[14]  T. Honjo,et al.  Antigen-induced apoptotic death of Ly-1 B cells responsible for autoimmune disease in transgenic mice , 1992, Nature.

[15]  S. Shinton,et al.  Selection of Autoantibody Specificities in the Ly‐1 B Subset a , 1992, Annals of the New York Academy of Sciences.

[16]  T. Honjo,et al.  A transgenic model of autoimmune hemolytic anemia , 1992, The Journal of experimental medicine.

[17]  D. Longo,et al.  Modulation of Signal Transduction in Phosphocholine-Specific B Cells from μκ Transgenic Mice , 1992 .

[18]  S. Korsmeyer.  Bcl-2: a repressor of lymphocyte death. , 1992, Immunology today.

[19]  Suzanne Cory,et al.  bcl-2 transgene inhibits T cell death and perturbs thymic self-censorship , 1991, Cell.

[20]  S. Korsmeyer,et al.  bcl-2 inhibits multiple forms of apoptosis but not negative selection in thymocytes , 1991, Cell.

[21]  P. Tucker,et al.  COOH terminus of membrane IgM is essential for an antigen-specific induction of some but not all early activation events in mature B cells , 1991, The Journal of experimental medicine.

[22]  D. Longo,et al.  Selection of antigen-specific, idiotype-positive B cells in transgenic mice expressing a rearranged M167-mu heavy chain gene , 1991, The Journal of experimental medicine.

[23]  D. Vaux,et al.  Enforced BCL2 expression in B-lymphoid cells prolongs antibody responses and elicits autoimmune disease. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[24]  A. Coutinho,et al.  Biased VH gene expression in murine CD5 B cells results from age‐dependent cellular selection , 1991, European journal of immunology.

[25]  A. Coutinho,et al.  Normal serum immunoglobulins participate in the selection of peripheral B-cell repertoires. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[26]  K. Rajewsky,et al.  Most peripheral B cells in mice are ligand selected , 1991, The Journal of experimental medicine.

[27]  D. Longo,et al.  Receptor-mediated elimination of phosphocholine-specific B cells in x-linked immune-deficient mice. , 1991, Journal of immunology.

[28]  K. Rajewsky,et al.  B cell development regulated by gene rearrangement: Arrest of maturation by membrane-bound Dμ protein and selection of DH element reading frames , 1991, Cell.

[29]  Klaus Rajewsky,et al.  A B cell-deficient mouse by targeted disruption of the membrane exon of the immunoglobulin μ chain gene , 1991, Nature.

[30]  A. Coutinho,et al.  All T15 Id-positive antibodies (but not the majority of VHT15+ antibodies) are produced by peritoneal CD5+ B lymphocytes. , 1990, International immunology.

[31]  K. Rajewsky,et al.  The bulk of the peripheral B-cell pool in mice is stable and not rapidly renewed from the bone marrow. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[32]  A. Coutinho,et al.  Expression of antibody V-regions is genetically and developmentally controlled and modulated by the B lymphocyte environment. , 1989, International immunology.

[33]  S. Korsmeyer,et al.  bcl-2-Immunoglobulin transgenic mice demonstrate extended B cell survival and follicular lymphoproliferation , 1989, Cell.

[34]  C. Webb,et al.  Immunoglobulin receptor signaling depends on the carboxyl terminus but not the heavy-chain class. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[35]  D. G. Osmond,et al.  Population Dynamics of Bone Marrow B Lymphocytes , 1986, Immunological reviews.

[36]  C. Pinkert,et al.  Transgenic mice with mu and kappa genes encoding antiphosphorylcholine antibodies , 1986, The Journal of experimental medicine.

[37]  C. V. Oss,et al.  Progress in immunology V , 1985 .