Exposure to receptor-activator of NFκB ligand renders pre-osteoclasts resistant to IFN-γ by inducing terminal differentiation
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[1] Sandrine Bongiovanni,et al. Transcriptional Program of Mouse Osteoclast Differentiation Governed by the Macrophage Colony-stimulating Factor and the Ligand for the Receptor Activator of NFκB* , 2002, The Journal of Biological Chemistry.
[2] Shi Wei,et al. Interleukin-4 Reversibly Inhibits Osteoclastogenesis via Inhibition of NF-κB and Mitogen-activated Protein Kinase Signaling* , 2002, The Journal of Biological Chemistry.
[3] C. Nelson,et al. Crystal structure of the TRANCE/RANKL cytokine reveals determinants of receptor-ligand specificity. , 2001, The Journal of clinical investigation.
[4] G. Bain,et al. Osteoprotegerin and receptor activator of nuclear factor kappaB ligand (RANKL) regulate osteoclast formation by cells in the human rheumatoid arthritic joint. , 2001, Rheumatology.
[5] Y. Abu-Amer. IL-4 abrogates osteoclastogenesis through STAT6-dependent inhibition of NF-κB , 2001 .
[6] N. Kamatani,et al. Activated human T cells directly induce osteoclastogenesis from human monocytes: possible role of T cells in bone destruction in rheumatoid arthritis patients. , 2001, Arthritis and rheumatism.
[7] T. Martin,et al. IL-12 Alone and in Synergy with IL-18 Inhibits Osteoclast Formation In Vitro1 , 2001, The Journal of Immunology.
[8] Y. Kadono,et al. Segregation of TRAF6‐mediated signaling pathways clarifies its role in osteoclastogenesis , 2001, The EMBO journal.
[9] Sung-Hwan Park,et al. Shift toward T helper 1 cytokines by type II collagen-reactive T cells in patients with rheumatoid arthritis. , 2001, Arthritis and rheumatism.
[10] E. Schwarz,et al. Efficacy of Etanercept for Wear Debris‐Induced Osteolysis , 2001, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[11] J. Haug,et al. T Cell Activation Induces Human Osteoclast Formation via Receptor Activator of Nuclear Factor κB Ligand‐Dependent and ‐Independent Mechanisms , 2001 .
[12] S. Takeshita,et al. TNF-alpha induces osteoclastogenesis by direct stimulation of macrophages exposed to permissive levels of RANK ligand. , 2000, The Journal of clinical investigation.
[13] M. Mezzina,et al. Adeno-associated virus-mediated delivery of IL-4 prevents collagen-induced arthritis , 2000, Gene Therapy.
[14] T. Chambers,et al. Interferon-gamma directly inhibits TRANCE-induced osteoclastogenesis. , 2000, Biochemical and biophysical research communications.
[15] S. Teitelbaum,et al. Bone resorption by osteoclasts. , 2000, Science.
[16] G. Boivin,et al. Adeno-associated virus mediates long-term gene transfer and delivery of chondroprotective IL-4 to murine synovium. , 2000, Molecular therapy : the journal of the American Society of Gene Therapy.
[17] T. Martin,et al. Fibroblastic Stromal Cells Express Receptor Activator of NF‐κB Ligand and Support Osteoclast Differentiation , 2000, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[18] L. Joosten,et al. IL-4 gene therapy for collagen arthritis suppresses synovial IL-17 and osteoprotegerin ligand and prevents bone erosion. , 2000, The Journal of clinical investigation.
[19] J. Pike,et al. The Inhibitory Effect of Interleukin‐10 on Mouse Osteoclast Formation Involves Novel Tyrosine‐Phosphorylated Proteins , 2000, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[20] J. Farrés,et al. Differential Th1/Th2 cytokine patterns in chronic arthritis: interferon γ is highly expressed in synovium of rheumatoid arthritis compared with seronegative spondyloarthropathies , 2000, Annals of the rheumatic diseases.
[21] Sundeep Khosla,et al. The Roles of Osteoprotegerin and Osteoprotegerin Ligand in the Paracrine Regulation of Bone Resorption , 2000, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[22] G. Tau,et al. Biologic functions of the IFN‐γ receptors , 1999, Allergy.
[23] T. Martin,et al. Activated T lymphocytes support osteoclast formation in vitro. , 1999, Biochemical and biophysical research communications.
[24] T. Martin,et al. Osteoblasts/stromal cells stimulate osteoclast activation through expression of osteoclast differentiation factor/RANKL but not macrophage colony-stimulating factor: receptor activator of NF-kappa B ligand. , 1999, Bone.
[25] S. Morony,et al. TRAF6 deficiency results in osteopetrosis and defective interleukin-1, CD40, and LPS signaling. , 1999, Genes & development.
[26] S. Morony,et al. Tumor necrosis factor receptor family member RANK mediates osteoclast differentiation and activation induced by osteoprotegerin ligand. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[27] N. Udagawa,et al. A new member of tumor necrosis factor ligand family, ODF/OPGL/TRANCE/RANKL, regulates osteoclast differentiation and function. , 1999, Biochemical and biophysical research communications.
[28] G. Rodan,et al. Interleukin 1 induces multinucleation and bone-resorbing activity of osteoclasts in the absence of osteoblasts/stromal cells. , 1999, Experimental cell research.
[29] S. Morony,et al. OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis , 1999, Nature.
[30] C. Ritchlin,et al. Patterns of cytokine production in psoriatic synovium. , 1998, The Journal of rheumatology.
[31] D. Lacey,et al. Osteoprotegerin Ligand Is a Cytokine that Regulates Osteoclast Differentiation and Activation , 1998, Cell.
[32] K Yano,et al. Osteoclast differentiation factor is a ligand for osteoprotegerin/osteoclastogenesis-inhibitory factor and is identical to TRANCE/RANKL. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[33] R. Dubose,et al. A homologue of the TNF receptor and its ligand enhance T-cell growth and dendritic-cell function , 1997, Nature.
[34] S. Huang,et al. High susceptibility to collagen-induced arthritis in mice lacking IFN-gamma receptors. , 1997, Journal of immunology.
[35] A. Billiau,et al. Accelerated collagen-induced arthritis in IFN-gamma receptor-deficient mice. , 1997, Journal of immunology.
[36] R. Schreiber,et al. Targeted Disruption of the Stat1 Gene in Mice Reveals Unexpected Physiologic Specificity in the JAK–STAT Signaling Pathway , 1996, Cell.
[37] R. Rodriguiz,et al. Long-term treatment of osteopetrosis with recombinant human interferon gamma. , 1995, The New England journal of medicine.
[38] J. Smolen,et al. Recombinant human interferon gamma in the treatment of rheumatoid arthritis: double blind placebo controlled study. , 1992, Annals of the rheumatic diseases.
[39] Elizabeth,et al. Selection of a human T helper type 1-like T cell subset by mycobacteria , 1991, The Journal of experimental medicine.
[40] J. Gerdes,et al. Immunohistologic assessment of cytokine production of infiltrating cells in various forms of leprosy. , 1990, The American journal of pathology.
[41] R. Maki,et al. Quantitative analysis of cytokine gene expression in rheumatoid arthritis. , 1990, Journal of immunology.
[42] F. Wolfe,et al. Double‐blind trial of recombinant γ‐interferon versus placebo in the treatment of rheumatoid arthritis , 1989 .
[43] T. Birdi,et al. Correlation between macrophage activation and bactericidal function and Mycobacterium leprae antigen presentation in macrophages of leprosy patients and normal individuals , 1989, Infection and immunity.
[44] J. Grosclaude,et al. Interferon gamma in rheumatoid arthritis--a double blind study comparing human recombinant interferon gamma with placebo. , 1988, The Journal of rheumatology.
[45] G. Roodman,et al. Recombinant human interferon-gamma inhibits formation of human osteoclast-like cells. , 1986, Journal of immunology.
[46] W. Ries,et al. Characterization of interferon gamma receptors on osteoclasts: Effect of interferon gamma on osteoclastic superoxide generation , 2002, Journal of cellular biochemistry.
[47] J. Haug,et al. T cell activation induces human osteoclast formation via receptor activator of nuclear factor kappaB ligand-dependent and -independent mechanisms. , 2001, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[48] E. Jimi,et al. The molecular basis of osteoclast differentiation and activation. , 2001, Novartis Foundation symposium.
[49] E. Shevach,et al. Susceptibility to collagen-induced arthritis: cytokine-mediated regulation. , 2001, Clinical immunology.
[50] Y. Abu-Amer. IL-4 abrogates osteoclastogenesis through STAT6-dependent inhibition of NF-kappaB. , 2001, The Journal of clinical investigation.
[51] T. Taniguchi,et al. T-cell-mediated regulation of osteoclastogenesis by signalling cross-talk between RANKL and IFN-gamma. , 2000, Nature.
[52] Josef M. Penninger,et al. Activated T cells regulate bone loss and joint destruction in adjuvant arthritis through osteoprotegerin ligand , 1999, Nature.
[53] M Aguet,et al. The IFN gamma receptor: a paradigm for cytokine receptor signaling. , 1997, Annual review of immunology.
[54] P. Emery,et al. A randomized, double-blind study comparing twenty-four-week treatment with recombinant interferon-gamma versus placebo in the treatment of rheumatoid arthritis. , 1997, Arthritis and rheumatism.
[55] P. Emery,et al. A randomized, double-blind study comparing twenty-four-week treatment with recombinant interferon-gamma versus placebo in the treatment of rheumatoid arthritis. , 1997, Arthritis and rheumatism.
[56] M. Farrar,et al. The molecular cell biology of interferon-gamma and its receptor. , 1993, Annual review of immunology.
[57] L. Hayman,et al. Correspondence , 1992, Neuroradiology.
[58] M. C. Googe,et al. The Arthritis Foundation. , 1984, Orthopedic nursing.