Molecular diversity of structure and function of the voltage-gated Na+ channels.
暂无分享,去创建一个
[1] M. Meisler,et al. Evolution and diversity of mammalian sodium channel genes. , 1999, Genomics.
[2] H Bostock,et al. Low-threshold, persistent sodium current in rat large dorsal root ganglion neurons in culture. , 1997, Journal of neurophysiology.
[3] P. Grafe,et al. Capsaicin blocks tetrodotoxin-resistant sodium potentials and calcium potentials in unmyelinated C fibres of biopsied human sural nerve in vitro , 1996, Neuroscience Letters.
[4] S. Dib-Hajj,et al. Spinal sensory neurons express multiple sodium channel α-subunit mRNAs , 1996 .
[5] S. Dib-Hajj,et al. Differential role of GDNF and NGF in the maintenance of two TTX-resistant sodium channels in adult DRG neurons. , 1999, Brain research. Molecular brain research.
[6] L. Sivilotti,et al. A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons , 1996, Nature.
[7] H. Lerche,et al. Voltage-sensor sodium channel mutations cause hypokalemic periodic paralysis type 2 by enhanced inactivation and reduced current. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[8] J. Levine,et al. Hyperalgesic agents increase a tetrodotoxin-resistant Na+ current in nociceptors. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[9] R M Eglen,et al. A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of chronic pain. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[10] A. L. Goldin,et al. Resurgence of sodium channel research. , 2001, Annual review of physiology.
[11] Samuel F. Berkovic,et al. Febrile seizures and generalized epilepsy associated with a mutation in the Na+-channel ß1 subunit gene SCN1B , 1998, Nature Genetics.
[12] Stéphanie Baulac,et al. Mutations of SCN1A, encoding a neuronal sodium channel, in two families with GEFS+2 , 2000, Nature Genetics.
[13] M. Noda,et al. Nav2/NaG Channel Is Involved in Control of Salt-Intake Behavior in the CNS , 2000, The Journal of Neuroscience.
[14] A. George,et al. Molecular cloning of an atypical voltage-gated sodium channel expressed in human heart and uterus: evidence for a distinct gene family. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[15] E. Kinoshita,et al. Novel wasp toxin discriminates between neuronal and cardiac sodium channels. , 2001, Molecular pharmacology.
[16] F. Conti,et al. Structural parts involved in activation and inactivation of the sodium channel , 1989, Nature.
[17] P. Schwindt,et al. Modal gating of Na+ channels as a mechanism of persistent Na+ current in pyramidal neurons from rat and cat sensorimotor cortex , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[18] W. Crill,et al. Persistent sodium current in mammalian central neurons. , 1996, Annual review of physiology.
[19] K Mizuguchi,et al. beta 3: an additional auxiliary subunit of the voltage-sensitive sodium channel that modulates channel gating with distinct kinetics. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[20] A L Goldin,et al. Primary structure and functional expression of the beta 1 subunit of the rat brain sodium channel. , 1992, Science.
[21] K. Bley,et al. Tetrodotoxin inhibits neuropathic ectopic activity in neuromas, dorsal root ganglia and dorsal horn neurons , 1997, PAIN.
[22] S. A. Raymond,et al. Electrophysiological evidence for tetrodotoxin-resistant sodium channels in slowly conducting dural sensory fibers. , 1999, Journal of neurophysiology.
[23] Y Ueno,et al. The voltage-sensitive sodium channel is a bell-shaped molecule with several cavities , 2001, Nature.
[24] Clare,et al. Voltage-gated sodium channels as therapeutic targets. , 2000, Drug discovery today.
[25] R. Llinás,et al. Molecular characterization of the sodium channel subunits expressed in mammalian cerebellar Purkinje cells. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[26] Andrew G. Engel,et al. Dihydropyridine receptor mutations cause hypokalemic periodic paralysis , 1994, Cell.
[27] J. Hunter,et al. Blockade of neuropathic pain by antisense targeting of tetrodotoxin-resistant sodium channels in sensory neurons. , 2000, Methods in enzymology.
[28] K. Beam,et al. The Sodium Channel Scn8a Is the Major Contributor to the Postnatal Developmental Increase of Sodium Current Density in Spinal Motoneurons , 1998, The Journal of Neuroscience.
[29] S. Tonegawa,et al. Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma. , 1997, Science.
[30] W. Catterall,et al. From Ionic Currents to Molecular Mechanisms The Structure and Function of Voltage-Gated Sodium Channels , 2000, Neuron.
[31] S. H. Chandler,et al. Electrophysiological properties of guinea pig trigeminal motoneurons recorded in vitro. , 1994, Journal of neurophysiology.
[32] Y. Berwald‐Netter,et al. The glial voltage-gated sodium channel: cell- and tissue-specific mRNA expression. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[33] L. Urbán,et al. Mechanisms of inflammatory pain. , 2001, British journal of anaesthesia.
[34] J. Elliott,et al. Characterization of TTX‐sensitive and TTX‐resistant sodium currents in small cells from adult rat dorsal root ganglia. , 1993, The Journal of physiology.
[35] S. Halegoua,et al. Identification of PN1, a predominant voltage-dependent sodium channel expressed principally in peripheral neurons. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[36] S. McMahon,et al. Neurotrophic factors and neuropathic pain. , 2001, Current opinion in pharmacology.
[37] J. Fitzsimons. Angiotensin, thirst, and sodium appetite. , 1998, Physiological reviews.
[38] J. Dubois,et al. Late sodium current in the node of Ranvier , 2004, Pflügers Archiv.
[39] R. Rogart,et al. A Mutant of TTX-Resistant Cardiac Sodium Channels with TTX-Sensitive Properties , 1992, Science.
[40] P. Mantyh,et al. Inhibition of hyperalgesia by ablation of lamina I spinal neurons expressing the substance P receptor. , 1997, Science.
[41] A. Akopian,et al. Structure and distribution of a broadly expressed atypical sodium channel , 1997, FEBS letters.
[42] P. Schwindt,et al. A persistent negative resistance in cat lumbar motoneurons , 1977, Brain Research.
[43] G. Ebers,et al. A novel sodium channel mutation in a family with hypokalemic periodic paralysis , 1999, Neurology.
[44] N. Tashiro,et al. Single-channel analysis of two types of Na+currents in rat dorsal root ganglia , 1995, Pflügers Archiv.
[45] S G Waxman,et al. Ionic mechanisms of anoxic injury in mammalian CNS white matter: role of Na+ channels and Na(+)-Ca2+ exchanger , 1992, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[46] I. Raman,et al. Resurgent Sodium Current and Action Potential Formation in Dissociated Cerebellar Purkinje Neurons , 1997, The Journal of Neuroscience.
[47] J. Harris,et al. Studies on tetrodotoxin resistant action potentials in denervated skeletal muscle. , 1971, Acta physiologica Scandinavica.
[48] I. Raman,et al. Inactivation and recovery of sodium currents in cerebellar Purkinje neurons: evidence for two mechanisms. , 2001, Biophysical journal.
[49] N. Grigorieff,et al. Three-dimensional structure of a voltage-gated potassium channel at 2.5 nm resolution. , 2001, Structure.
[50] R. Llinás,et al. Electrophysiological properties of in vitro Purkinje cell dendrites in mammalian cerebellar slices. , 1980, The Journal of physiology.
[51] S. Oliet,et al. Osmoreceptors in the central nervous system. , 1997, Annual review of physiology.
[52] Ming Zhou,et al. Sodium channel mutations in paramyotonia congenita uncouple inactivation from activation , 1994, Neuron.
[53] H. Tatebayashi,et al. Kinetic analysis of two types of Na+ channels in rat dorsal root ganglia. , 1993, The Journal of physiology.
[54] J. Chung,et al. Signs of neuropathic pain depend on signals from injured nerve fibers in a rat model , 1993, Brain Research.
[55] R Llinás,et al. Kinetic and stochastic properties of a persistent sodium current in mature guinea pig cerebellar Purkinje cells. , 1998, Journal of neurophysiology.
[56] C. Bountra,et al. Two sodium channels contribute to the TTX-R sodium current in primary sensory neurons , 1998, Nature Neuroscience.
[57] S. Waxman,et al. Na+ channel beta 1 subunit mRNA: differential expression in rat spinal sensory neurons. , 1995, Brain research. Molecular brain research.
[58] S. McMahon,et al. Potent analgesic effects of GDNF in neuropathic pain states. , 2000, Science.
[59] S G Waxman,et al. A Novel Persistent Tetrodotoxin-Resistant Sodium Current In SNS-Null And Wild-Type Small Primary Sensory Neurons , 1999, The Journal of Neuroscience.
[60] E. Stevens,et al. β3, a novel auxiliary subunit for the voltage‐gated sodium channel, is expressed preferentially in sensory neurons and is upregulated in the chronic constriction injury model of neuropathic pain , 2000 .
[61] Gail Mandel,et al. Nomenclature of Voltage-Gated Sodium Channels , 2000, Neuron.
[62] S. McMahon,et al. A Distinct Subgroup of Small DRG Cells Express GDNF Receptor Components and GDNF Is Protective for These Neurons after Nerve Injury , 1998, The Journal of Neuroscience.
[63] S. Dib-Hajj,et al. Sodium channels and pain. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[64] S. Cannon,et al. The Human Skeletal Muscle Na Channel Mutation R669H Associated with Hypokalemic Periodic Paralysis Enhances Slow Inactivation , 2000, The Journal of Neuroscience.
[65] S G Waxman,et al. Noninactivating, tetrodotoxin-sensitive Na+ conductance in rat optic nerve axons. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[66] C. Belmonte,et al. Tetrodotoxin‐resistant impulses in single nociceptor nerve terminals in guinea‐pig cornea , 1998, The Journal of physiology.
[67] J. Caldwell,et al. Sodium channel Na(v)1.6 is localized at nodes of ranvier, dendrites, and synapses. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[68] R. Llinás,et al. Electrophysiological properties of in vitro Purkinje cell somata in mammalian cerebellar slices. , 1980, The Journal of physiology.
[69] S. Boyce,et al. The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways , 1999, Nature Neuroscience.
[70] A. L. Goldin,et al. Amino acid residues required for fast Na(+)-channel inactivation: charge neutralizations and deletions in the III-IV linker. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[71] F. W. Kerr. Neuroanatomical substrates of nociception in the spinal cord , 1975, PAIN.
[72] C. Woolf,et al. Diversity of Expression of the Sensory Neuron-Specific TTX-Resistant Voltage-Gated Sodium Ion Channels SNS and SNS2 , 2000, Molecular and Cellular Neuroscience.
[73] H. Naoki,et al. Isolation and structure of pompilidotoxins, novel peptide neurotoxins in solitary wasp venoms. , 1998, Biochemical and biophysical research communications.
[74] P. Gage,et al. A persistent sodium current in rat ventricular myocytes. , 1992, The Journal of physiology.
[75] A. Akopian,et al. Trans‐splicing of a voltage‐gated sodium channel is regulated by nerve growth factor , 1999, FEBS letters.
[76] I. Raman,et al. Altered Subthreshold Sodium Currents and Disrupted Firing Patterns in Purkinje Neurons of Scn8a Mutant Mice , 1997, Neuron.
[77] K. Beam,et al. The absence of resurgent sodium current in mouse spinal neurons , 1999, Brain Research.
[78] P W Gage,et al. A voltage-dependent persistent sodium current in mammalian hippocampal neurons , 1990, The Journal of general physiology.
[79] M. Tamkun,et al. Primary structure and differential expression during development and pregnancy of a novel voltage-gated sodium channel in the mouse. , 1994, The Journal of biological chemistry.
[80] S. Dib-Hajj,et al. SNS Na+ channel expression increases in dorsal root ganglion neurons in the carrageenan inflammatory pain model , 1998, Neuroreport.
[81] R. Eglen,et al. Distribution of the Tetrodotoxin-Resistant Sodium Channel PN3 in Rat Sensory Neurons in Normal and Neuropathic Conditions , 1998, The Journal of Neuroscience.
[82] Nicholas W. Plummer,et al. Functional Analysis of the Mouse Scn8a Sodium Channel , 1998, The Journal of Neuroscience.