Cilnidipine suppresses podocyte injury and proteinuria in metabolic syndrome rats: possible involvement of N-type calcium channel in podocyte
暂无分享,去创建一个
H. Kobori | A. Nishiyama | Y. Tomino | K. Asanuma | M. Kohno | T. Fujita | T. Noma | N. Ohashi | H. Hitomi | D. Nakano | Yu-Yan Fan | H. Ohsaki | Diah Suwarni
[1] P. Bie,et al. Renin secretion and total body sodium: Pathways of integrative control , 2010, Clinical and experimental pharmacology & physiology.
[2] T. Fujita,et al. Comparison of antioxidant activity of cilnidipine and amlodipine. , 2009, Kidney International.
[3] K. Nitta,et al. Different Effects of L/N-Type and L-Type Calcium Channel Blockers on the Renin-Angiotensin-Aldosterone System in SHR/Izm , 2009, American Journal of Nephrology.
[4] J. Ingelfinger,et al. Apocynin attenuates tubular apoptosis and tubulointerstitial fibrosis in transgenic mice independent of hypertension. , 2009, Kidney international.
[5] W. Comper,et al. Disease-dependent mechanisms of albuminuria. , 2008, American journal of physiology. Renal physiology.
[6] K. Kimura,et al. Vasodilatory effect of cilnidipine, an L-type and N-type calcium channel blocker, on rat kidney glomerular arterioles. , 2008, International heart journal.
[7] Takashi Suzuki,et al. Modified Sympathetic Nerve System Activity with Overexpression of the Voltage-dependent Calcium Channel β3 Subunit* , 2008, Journal of Biological Chemistry.
[8] H. Pavenstädt,et al. Mechanisms of angiotensin II signaling on cytoskeleton of podocytes , 2008, Journal of Molecular Medicine.
[9] S. Ito,et al. Strict angiotensin blockade prevents the augmentation of intrarenal angiotensin II and podocyte abnormalities in type 2 diabetic rats with microalbuminuria , 2008, Journal of hypertension.
[10] H. Yokoi,et al. Overexpression of connective tissue growth factor in podocytes worsens diabetic nephropathy in mice. , 2008, Kidney international.
[11] V. Demarco,et al. Attenuation of NADPH Oxidase Activation and Glomerular Filtration Barrier Remodeling With Statin Treatment , 2008, Hypertension.
[12] J. Ingelfinger,et al. Overexpression of angiotensinogen increases tubular apoptosis in diabetes. , 2008, Journal of the American Society of Nephrology : JASN.
[13] G. Yasuda,et al. Antiproteinuric effect of the calcium channel blocker cilnidipine added to renin-angiotensin inhibition in hypertensive patients with chronic renal disease. , 2007, Kidney international.
[14] N. Vaziri,et al. Intra-Renal Angiotensin II/AT1 Receptor, Oxidative Stress, Inflammation, and Progressive Injury in Renal Mass Reduction , 2007, Journal of Pharmacology and Experimental Therapeutics.
[15] T. Iwasaka,et al. Renal and vascular protective effects of cilnidipine in patients with essential hypertension , 2007, Journal of hypertension.
[16] J. Ingelfinger,et al. Catalase overexpression attenuates angiotensinogen expression and apoptosis in diabetic mice. , 2007, Kidney international.
[17] K. Kario,et al. The differential effects of angiotensin II type 1 receptor blockers on microalbuminuria in relation to low-grade inflammation in metabolic hypertensive patients. , 2007, American journal of hypertension.
[18] K. Homma,et al. Ca2+ channel subtypes and pharmacology in the kidney. , 2007, Circulation research.
[19] V. Cachofeiro,et al. Insulin resistance, inflammatory biomarkers, and adipokines in patients with chronic kidney disease: effects of angiotensin II blockade. , 2006, Journal of the American Society of Nephrology : JASN.
[20] W. Atchison,et al. Acetylcholine Release at Neuromuscular Junctions of Adult Tottering Mice Is Controlled by N-(Cav2.2) and R-Type (Cav2.3) but Not L-Type (Cav1.2) Ca2+ Channels , 2006, Journal of Pharmacology and Experimental Therapeutics.
[21] A. Nishiyama,et al. Effects of Adrenomedullin on Cardiac Oxidative Stress and Collagen Accumulation in Aldosterone-Dependent Malignant Hypertensive Rats , 2006, Journal of Pharmacology and Experimental Therapeutics.
[22] D. Ekuni,et al. Regulation of epithelial cell growth factor receptor protein and gene expression using a rat periodontitis model. , 2006, Journal of periodontal research.
[23] Hiroshi Yamamoto,et al. Effects of L/N-type calcium channel antagonist, cilnidipine on progressive renal injuries in Dahl salt-sensitive rats. , 2006, Biological & pharmaceutical bulletin.
[24] A. Nishiyama,et al. D-allose, an all-cis aldo-hexose, suppresses development of salt-induced hypertension in Dahl rats , 2005, Journal of hypertension.
[25] T. Moriyama,et al. The N- and L-Type Calcium Channel Blocker Cilnidipine Suppresses Renal Injury in Dahl Rats Fed a High-Sucrose Diet, an Experimental Model of Metabolic Syndrome , 2005, Nephron Physiology.
[26] H. Kobori,et al. Enhanced intrarenal angiotensinogen contributes to early renal injury in spontaneously hypertensive rats. , 2005, Journal of the American Society of Nephrology : JASN.
[27] G. Chertow,et al. Metabolic syndrome and the risk for chronic kidney disease among nondiabetic adults. , 2005, Journal of the American Society of Nephrology : JASN.
[28] H. Kobori,et al. Temporary angiotensin II blockade at the prediabetic stage attenuates the development of renal injury in type 2 diabetic rats. , 2005, Journal of the American Society of Nephrology : JASN.
[29] I. G. Fantus,et al. Reactive oxygen species blockade and action of insulin on expression of angiotensinogen gene in proximal tubular cells. , 2004, The Journal of endocrinology.
[30] P. Whelton,et al. Prevalence of non-traditional cardiovascular disease risk factors among persons with impaired fasting glucose, impaired glucose tolerance, diabetes, and the metabolic syndrome: analysis of the Third National Health and Nutrition Examination Survey (NHANES III). , 2004, Annals of epidemiology.
[31] H. Yokoyama,et al. Comparison between cilnidipine and amlodipine besilate with respect to proteinuria in hypertensive patients with renal diseases. , 2004, Hypertension research : official journal of the Japanese Society of Hypertension.
[32] Katrin Rittinger,et al. Molecular Basis of Phosphorylation-Induced Activation of the NADPH Oxidase , 2003, Cell.
[33] A. Shah,et al. Intracellular Localization and Preassembly of the NADPH Oxidase Complex in Cultured Endothelial Cells* , 2002, The Journal of Biological Chemistry.
[34] E. Frohlich,et al. N- and L-type calcium channel antagonist improves glomerular dynamics, reverses severe nephrosclerosis, and inhibits apoptosis and proliferation in an l-NAME/SHR model , 2002, Journal of hypertension.
[35] L. Cebotaru,et al. Tubular and cellular localization of the cardiac L-type calcium channel in rat kidney. , 2002, Kidney international.
[36] E. Frohlich,et al. Cilnidipine improves spontaneously hypertensive rat coronary hemodynamics without altering cardiovascular mass and collagen , 2002, Journal of hypertension.
[37] H. Suzuki,et al. Cilnidipine is as effective as benazepril for control of blood pressure and proteinuria in hypertensive patients with benign nephrosclerosis. , 2001, Hypertension research : official journal of the Japanese Society of Hypertension.
[38] H. Pavenstädt,et al. Acetylcholine increases the free intracellular calcium concentration in podocytes in intact rat glomeruli via muscarinic M(5) receptors. , 2001, Journal of the American Society of Nephrology : JASN.
[39] S. Nielsen,et al. The alpha(1G)-subunit of a voltage-dependent Ca(2+) channel is localized in rat distal nephron and collecting duct. , 2000, American journal of physiology. Renal physiology.
[40] J. Gloy,et al. Catecholamines modulate podocyte function. , 1998, Journal of the American Society of Nephrology : JASN.
[41] R. Zeller,et al. Rearrangements of the cytoskeleton and cell contacts induce process formation during differentiation of conditionally immortalized mouse podocyte cell lines. , 1997, Experimental cell research.
[42] Maristela L Onozato,et al. Oxidative stress and nitric oxide synthase in rat diabetic nephropathy: effects of ACEI and ARB. , 2002, Kidney international.
[43] G. Shearer,et al. Hypoalbuminemia and proteinuria contribute separately to reduced lipoprotein catabolism in the nephrotic syndrome. , 2001, Kidney international.
[44] J. Gloy,et al. Angiotensin II increases the intracellular calcium activity in podocytes of the intact glomerulus. , 2000, Kidney international.