EGF inhibits muscarinic receptor-mediated calcium signaling in a human salivary cell line.
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M. S. Katz | M. Lifschitz | M S Katz | C. Yeh | B X Zhang | C K Yeh | T K Hymer | M D Lifschitz | B. X. Zhang | T. Hymer
[1] M. S. Katz,et al. Modulation by food restriction of intracellular calcium signaling in parotid acinar cells of aging Fischer 344 rats. , 1997, The journals of gerontology. Series A, Biological sciences and medical sciences.
[2] M. Berridge,et al. The regulation of capacitative calcium entry by calcium and protein kinase C in Xenopus oocytes. , 1994, The Journal of biological chemistry.
[3] T. Hunter,et al. Phospholipase C-γ is a substrate for the PDGF and EGF receptor protein-tyrosine kinases in vivo and in vitro , 1989, Cell.
[4] R. Tsien,et al. A new generation of Ca2+ indicators with greatly improved fluorescence properties. , 1985, The Journal of biological chemistry.
[5] J. Putney,et al. A model for receptor-regulated calcium entry. , 1986, Cell calcium.
[6] S. Miller,et al. Increased distal nephron EGF content and altered distribution of peptide in compensatory renal hypertrophy. , 1992, The American journal of physiology.
[7] D T Denhardt,et al. Signal-transducing protein phosphorylation cascades mediated by Ras/Rho proteins in the mammalian cell: the potential for multiplex signalling. , 1996, The Biochemical journal.
[8] A. Marks,et al. Regulation of the Inositol 1,4,5-Trisphosphate Receptor by Tyrosine Phosphorylation , 1996, Science.
[9] E. Nexo. [The epidermal growth factor family]. , 1994, Ugeskrift for laeger.
[10] B. Baum,et al. Protein kinase C differentially inhibits muscarinic receptor operated Ca2+ release and entry in human salivary cells. , 1988, Biochemical and biophysical research communications.
[11] J. Ware,et al. Selective Inhibition of Thrombin Receptor-mediated Ca2+ Entry by Protein Kinase C β (*) , 1995, The Journal of Biological Chemistry.
[12] Hyosang Lee,et al. Feedback Regulation of ATP-induced Ca2+ Signaling in HL-60 Cells Is Mediated by Protein Kinase A- and C-mediated Changes in Capacitative Ca2+ Entry* , 1997, The Journal of Biological Chemistry.
[13] R. Derynck,et al. Inhibition of parathyroid hormone-responsive adenylate cyclase in clonal osteoblast-like cells by transforming growth factor alpha and epidermal growth factor. , 1987, The Journal of biological chemistry.
[14] G. Scatchard,et al. THE ATTRACTIONS OF PROTEINS FOR SMALL MOLECULES AND IONS , 1949 .
[15] T. Hunter,et al. Phospholipase C-gamma is a substrate for the PDGF and EGF receptor protein-tyrosine kinases in vivo and in vitro. , 1989, Cell.
[16] P. Barnes,et al. Transcriptional Down-regulation of m2 Muscarinic Receptor Gene Expression in Human Embryonic Lung (HEL 299) Cells by Protein Kinase C (*) , 1995, The Journal of Biological Chemistry.
[17] M. Foote. Ecological Controls on the Evolutionary Recovery of Post-Paleozoic Crinoids , 1996, Science.
[18] U. McGovern,et al. Intracellular calcium as a second messenger following growth stimulation of human keratinocytes , 1995, The British journal of dermatology.
[19] G. Carpenter,et al. The Epidermal Growth Factor Family , 1991 .
[20] B. Zhang,et al. Activation of purinergic receptors triggers oscillatory contractions in adult rat ventricular myocytes. , 1996, Circulation research.
[21] E. Peralta,et al. The m1 muscarinic acetylcholine receptor transactivates the EGF receptor to modulate ion channel activity , 1997, The EMBO journal.
[22] J. Putney,et al. Differential Effects of Protein Kinase C Activation on Calcium Storage and Capacitative Calcium Entry in NIH 3T3 Cells* , 1996, The Journal of Biological Chemistry.
[23] B. Baum,et al. Activation and regulation of calcium entry in rat parotid gland acinar cells. , 1993, Critical reviews in oral biology and medicine : an official publication of the American Association of Oral Biologists.
[24] S. Muallem,et al. Ca(2+)-dependent kinase and phosphatase control inositol 1,4,5-trisphosphate-mediated Ca2+ release. Modification by agonist stimulation. , 1993, The Journal of biological chemistry.
[25] R. Huganir,et al. Inositol trisphosphate receptor: phosphorylation by protein kinase C and calcium calmodulin-dependent protein kinases in reconstituted lipid vesicles. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[26] S. Snyder,et al. Cyclic AMP-dependent phosphorylation of a brain inositol trisphosphate receptor decreases its release of calcium. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[27] K. Nakamura,et al. Multiple actions of epidermal growth factor and TGF-alpha on rabbit gastric parietal cell function. , 1994, The American journal of physiology.
[28] L. Birnbaumer,et al. Cloning of Trp1β isoform from rat brain: immunodetection and localization of the endogenous Trp1 protein. , 1999, American journal of physiology. Cell physiology.
[29] J. Putney,et al. Role of inositol (1,4,5)trisphosphate in epidermal growth factor-induced Ca2+ signaling in A431 cells. , 1991, Molecular pharmacology.
[30] L. Birnbaumer,et al. Calcium Channels Formed by Mammalian Trp Homologues. , 1998, News in physiological sciences : an international journal of physiology produced jointly by the International Union of Physiological Sciences and the American Physiological Society.
[31] H. Yoshida,et al. Generation of cells with phenotypes of both intercalated duct-type and myoepithelial cells in human parotid gland adenocarcinoma clonal cells grown in athymic nude mice , 1986, Virchows Archiv. B, Cell pathology including molecular pathology.
[32] Philip R. Cohen,et al. PD 098059 Is a Specific Inhibitor of the Activation of Mitogen-activated Protein Kinase Kinase in Vitro and in Vivo(*) , 1995, The Journal of Biological Chemistry.
[33] J. Putney,et al. Sinusoidal oscillations in intracellular calcium requiring negative feedback by protein kinase C. , 1993, The Journal of biological chemistry.
[34] J. Putney,et al. Identification of cellular activation mechanisms associated with salivary secretion. , 1986, Annual review of physiology.
[35] L. Wang,et al. Epidermal growth factor inhibits carbachol-stimulated canine parietal cell function via protein kinase C. , 1996, Gastroenterology.
[36] M. S. Katz,et al. Bidirectional modulation of parathyroid hormone-responsive adenylyl cyclase by protein kinase C. , 1994, The American journal of physiology.
[37] P. Cullen,et al. Thapsigargin, a tumor promoter, discharges intracellular Ca2+ stores by specific inhibition of the endoplasmic reticulum Ca2(+)-ATPase. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[38] M. Humphreys-Beher,et al. Effect of EGF on rat parotid gland secretory function. , 1995, Comparative biochemistry and physiology. Part C, Pharmacology, toxicology & endocrinology.
[39] K. Malarkey,et al. The regulation of tyrosine kinase signalling pathways by growth factor and G-protein-coupled receptors. , 1995, The Biochemical journal.
[40] K. Barrett,et al. Carbachol stimulates transactivation of epidermal growth factor receptor and mitogen-activated protein kinase in T84 cells. Implications for carbachol-stimulated chloride secretion. , 1998, The Journal of biological chemistry.
[41] L. Kao,et al. Nerve Growth Factor, Epidermal Growth Factor, and Insulin Differentially Potentiate ATP‐Induced [Ca2+]i Rise and Dopamine Secretion in PC12 Cells , 1996, Journal of neurochemistry.
[42] K. Barrett,et al. Epidermal growth factor inhibits Ca(2+)-dependent Cl- transport in T84 human colonic epithelial cells. , 1996, The American journal of physiology.
[43] R. Hurst,et al. Cloning and Expression of a Novel Mammalian Homolog ofDrosophila Transient Receptor Potential (Trp) Involved in Calcium Entry Secondary to Activation of Receptors Coupled by the Gq Class of G Protein* , 1997, The Journal of Biological Chemistry.